Диссертация (1154897), страница 11
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1061-9.28. Cho A. K., Sioutas C., Miguel A. H., Kumagai Y., Schmitz D. A., Singh M.,Eiguren-Fernandez A., Froines J. R. Redox activity of airborne particulate matter atdifferent sites in the Los Angeles Basin // Environ Res. ‒ 2005. ‒ T. 99, № 1. ‒ C. 40-7.29. Matthay M. A., Geiser T., Matalon S., Ischiropoulos H. Oxidant-mediated lunginjury in the acute respiratory distress syndrome // Crit Care Med. ‒ 1999. ‒ T. 27, № 9.69‒ C. 2028-30.30. Comhair S. A., Thomassen M. J., Erzurum S. C. Differential induction ofextracellular glutathione peroxidase and nitric oxide synthase 2 in airways of healthyindividuals exposed to 100% O(2) or cigarette smoke // Am J Respir Cell Mol Biol.
‒2000. ‒ T. 23, № 3. ‒ C. 350-4.31. Narayanan P. K., Goodwin E. H., Lehnert B. E. Alpha particles initiatebiological production of superoxide anions and hydrogen peroxide in human cells //Cancer Res. ‒ 1997. ‒ T. 57, № 18. ‒ C. 3963-71.32. Chiu S. M., Xue L. Y., Friedman L. R., Oleinick N. L. Copper ion-mediatedsensitization of nuclear matrix attachment sites to ionizing radiation // Biochemistry.
‒1993. ‒ T. 32, № 24. ‒ C. 6214-9.33. Kinnula V. L., Crapo J. D. Superoxide dismutases in the lung and human lungdiseases // Am J Respir Crit Care Med. ‒ 2003. ‒ T. 167, № 12. ‒ C. 1600-19.34. Kinnula V. L. Production and degradation of oxygen metabolites duringinflammatory states in the human lung // Curr Drug Targets Inflamm Allergy. ‒ 2005. ‒T. 4, № 4. ‒ C.
465-70.35. Arthur J. R. The glutathione peroxidases // Cell Mol Life Sci. ‒ 2000. ‒ T. 57,№ 13-14. ‒ C. 1825-35.36. Manevich Y., Feinstein S. I., Fisher A. B. Activation of the antioxidant enzyme1-CYS peroxiredoxin requires glutathionylation mediated by heterodimerization with piGST // Proc Natl Acad Sci U S A. ‒ 2004. ‒ T. 101, № 11. ‒ C. 3780-5.37. Bunker V. W. Free radicals, antioxidants and ageing // Med Lab Sci.
‒ 1992. ‒T. 49, № 4. ‒ C. 299-312.38. El-Agamey A., Lowe G. M., McGarvey D. J., Mortensen A., Phillip D. M.,Truscott T. G., Young A. J. Carotenoid radical chemistry and antioxidant/pro-oxidantproperties // Arch Biochem Biophys. ‒ 2004. ‒ T. 430, № 1. ‒ C. 37-48.39.
Галунска Б., Паскалев Д., Янкова Т., Чанкова П. Двуликий янусбиохимии: мочевая кислота-оксидант или антиоксидант? // Нефрология. ‒ 2004. ‒T. 8, № 4.7040. Kelly F. J., Mudway I. S. Protein oxidation at the air-lung interface // AminoAcids. ‒ 2003.
‒ T. 25, № 3-4. ‒ C. 375-96.41. Fuchs G. J. Antioxidants for children with kwashiorkor // BMJ. ‒ 2005. ‒ T.330, № 7500. ‒ C. 1095-6.42. Halliwell B. Reactive species and antioxidants. Redox biology is a fundamentaltheme of aerobic life // Plant Physiol. ‒ 2006. ‒ T. 141, № 2.
‒ C. 312-22.43. Системные некротизирующие васкулиты. / Семенкова Е. Н. ‒ М.: Русскийврач, 2001.44. Stegeman C. A., Tervaert J. W., Sluiter W. J., Manson W. L., de Jong P. E.,Kallenberg C. G. Association of chronic nasal carriage of Staphylococcus aureus andhigher relapse rates in Wegener granulomatosis // Ann Intern Med.
‒ 1994. ‒ T. 120, №1. ‒ C. 12-7.45. Popa E. R., Stegeman C. A., Abdulahad W. H., van der Meer B., Arends J.,Manson W. M., Bos N. A., Kallenberg C. G., Tervaert J. W. Staphylococcal toxicshock-syndrome-toxin-1 as a risk factor for disease relapse in Wegener's granulomatosis// Rheumatology (Oxford). ‒ 2007. ‒ T. 46, № 6. ‒ C. 1029-33.46. Wiik A. Clinical and pathophysiological significance of anti-neutrophilcytoplasmic autoantibodies in vasculitis syndromes // Mod Rheumatol. ‒ 2009.
‒ T. 19,№ 6. ‒ C. 590-9.47. Savage C. O., Winearls C. G., Evans D. J., Rees A. J., Lockwood C. M.Microscopic polyarteritis: presentation, pathology and prognosis // Q J Med. ‒ 1985. ‒T. 56, № 220. ‒ C. 467-83.48. Новиков П. И. Клиническая оценка вариантов течения и прогнозагранулематоза с полиангиитом (Вегенера): диссертация ... кандидата медицинскихнаук: 14.01.04. ‒ Москва, 2015.
‒ 113 c.49. Lanham J. G., Elkon K. B., Pusey C. D., Hughes G. R. Systemic vasculitis withasthma and eosinophilia: a clinical approach to the Churg-Strauss syndrome // Medicine(Baltimore). ‒ 1984. ‒ T. 63, № 2. ‒ C. 65-81.50. Моисеев С. В., Новиков П. И., Федоров К. Е., Жабина Е.
С.71Эозинофильный гранулематоз с полиангиитом (Черга-Страусс): клиническиеварианты, диагностика и лечение // Клиническая фармакология и терапия. ‒ 2013.‒ T. 22, № 5. ‒ C. 86-92.51. Мухин Н. А., Новиков П. И., Моисеев С. В., Семенкова Е. Н., КозловскаяЛ. В., Фомин В. В., Игнатова Т. М., Стрижаков Л. А., Гуляев С. В., Янушкевич Т.Н. Эффективность и безопасность генно-инженерных биологических препаратову пациентов с ревматоидным артритом и другими ревматическими заболеваниями(проспективное неконтролируемое исследование) // Клин фармакол тер. ‒ 2012.
‒T. 21, № 5. ‒ C. 25-32.52. Cartin-Ceba R., Peikert T., Specks U. Pathogenesis of ANCA-associatedvasculitis // Curr Rheumatol Rep. ‒ 2012. ‒ T. 14, № 6. ‒ C. 481-93.53. Jennette J. C., Falk R. J. Pathogenesis of antineutrophil cytoplasmicautoantibody-mediated disease // Nat Rev Rheumatol. ‒ 2014. ‒ T. 10, № 8. ‒ C. 46373.54. Griffiths H. R. ROS as signalling molecules in T cells--evidence for abnormalredox signalling in the autoimmune disease, rheumatoid arthritis // Redox Rep. ‒ 2005.‒ T.
10, № 6. ‒ C. 273-80.55. D'Autréaux B., Toledano M. B. ROS as signalling molecules: mechanisms thatgenerate specificity in ROS homeostasis // Nat Rev Mol Cell Biol. ‒ 2007. ‒ T. 8, № 10.‒ C. 813-24.56. Charles L. A., Jennette J. C., Xiao H., Hu P. Complement in ANCA-associatedvasculitis // Semin Nephrol. ‒ 2013. ‒ T. 33, № 6. ‒ C. 557-64.57. Falk R. J., Terrell R. S., Charles L. A., Jennette J. C. Anti-neutrophilcytoplasmic autoantibodies induce neutrophils to degranulate and produce oxygenradicals in vitro // Proc Natl Acad Sci U S A. ‒ 1990.
‒ T. 87, № 11. ‒ C. 4115-9.58. Millet A., Pederzoli-Ribeil M., Guillevin L., Witko-Sarsat V., Mouthon L.Antineutrophil cytoplasmic antibody-associated vasculitides: is it time to split up thegroup? // Ann Rheum Dis. ‒ 2013. ‒ T. 72, № 8. ‒ C. 1273-9.59. Salama A. D. Pathogenesis and treatment of ANCA-associated systemic72vasculitis // J R Soc Med. ‒ 1999. ‒ T. 92, № 9. ‒ C. 456-61.60. Radford D. J., Lord J. M., Savage C. O. The activation of the neutrophilrespiratory burst by anti-neutrophil cytoplasm autoantibody (ANCA) from patients withsystemic vasculitis requires tyrosine kinases and protein kinase C activation // Clin ExpImmunol.
‒ 1999. ‒ T. 118, № 1. ‒ C. 171-9.61. Bae Y. S., Lee J. H., Choi S. H., Kim S., Almazan F., Witztum J. L., Miller Y.I. Macrophages generate reactive oxygen species in response to minimally oxidizedlow-density lipoprotein: toll-like receptor 4- and spleen tyrosine kinase-dependentactivation of NADPH oxidase 2 // Circ Res. ‒ 2009.
‒ T. 104, № 2. ‒ C. 210-8, 21pfollowing 218.62. Ohta N., Fukase S., Aoyagi M. Study of peripheral neutrophil functions ofpatients with Wegener's granulomatosis // Arerugi. ‒ 1994. ‒ T. 43, № 9. ‒ C. 1156-62.63. Savage C. O., Pottinger B. E., Gaskin G., Pusey C.
D., Pearson J. D.Autoantibodies developing to myeloperoxidase and proteinase 3 in systemic vasculitisstimulate neutrophil cytotoxicity toward cultured endothelial cells // Am J Pathol. ‒1992. ‒ T. 141, № 2. ‒ C. 335-42.64. Lu X., Garfield A., Rainger G. E., Savage C. O., Nash G. B. Mediation ofendothelial cell damage by serine proteases, but not superoxide, released fromantineutrophil cytoplasmic antibody-stimulated neutrophils // Arthritis Rheum. ‒ 2006.‒ T. 54, № 5. ‒ C. 1619-28.65. Wai Y.
T., Williams J., Pall A., Wilkes M., Savage C. O., Adu D.Antineutrophil cytoplasm antibody-induced neutrophil nitric oxide production is nitricoxide synthase independent // Kidney international. ‒ 2001. ‒ T. 59, № 2. ‒ C. 593-600.66. Bruce I., McNally J., Bell A. Enhanced monocyte generation of reactiveoxygen species in primary systemic vasculitis // J Rheumatol. ‒ 1997.
‒ T. 24, № 12. ‒C. 2364-70.67. Weidner S., Neupert W., Goppelt-Struebe M., Rupprecht H. D. Antineutrophilcytoplasmic antibodies induce human monocytes to produce oxygen radicals in vitro //Arthritis Rheum. ‒ 2001. ‒ T. 44, № 7. ‒ C. 1698-706.7368.
Ames P. R., Alves J., Murat I., Isenberg D. A., Nourooz-Zadeh J. Oxidativestress in systemic lupus erythematosus and allied conditions with vascular involvement// Rheumatology (Oxford). ‒ 1999. ‒ T. 38, № 6. ‒ C. 529-34.69. Guilpain P., Chéreau C., Goulvestre C., Servettaz A., Montani D., Tamas N.,Pagnoux C., Hachulla E., Weill B., Guillevin L., Mouthon L., Batteux F. The oxidationinduced by antimyeloperoxidase antibodies triggers fibrosis in microscopic polyangiitis// Eur Respir J. ‒ 2011. ‒ T.
37, № 6. ‒ C. 1503-13.70. Ohlsson S. M., Ohlsson S., Söderberg D., Gunnarsson L., Pettersson Å.,Segelmark M., Hellmark T. Neutrophils from vasculitis patients exhibit an increasedpropensity for activation by anti-neutrophil cytoplasmic antibodies // Clin ExpImmunol. ‒ 2014. ‒ T. 176, № 3. ‒ C. 363-72.71. Barnado A., Crofford L. J., Oates J. C. At the Bedside: Neutrophil extracellulartraps (NETs) as targets for biomarkers and therapies in autoimmune diseases // J LeukocBiol. ‒ 2016.
‒ T. 99, № 2. ‒ C. 265-78.72. Zhong X. Y., von Mühlenen I., Li Y., Kang A., Gupta A. K., Tyndall A.,Holzgreve W., Hahn S., Hasler P. Increased concentrations of antibody-boundcirculatory cell-free DNA in rheumatoid arthritis // Clin Chem. ‒ 2007. ‒ T. 53, № 9. ‒C. 1609-14.73. Neeli I., Khan S. N., Radic M.
Histone deimination as a response toinflammatory stimuli in neutrophils // J Immunol. ‒ 2008. ‒ T. 180, № 3. ‒ C. 1895-902.74. Remijsen Q., Vanden Berghe T., Wirawan E., Asselbergh B., Parthoens E., DeRycke R., Noppen S., Delforge M., Willems J., Vandenabeele P. Neutrophilextracellular trap cell death requires both autophagy and superoxide generation // CellRes. ‒ 2011. ‒ T. 21, № 2. ‒ C. 290-304.75. Fuchs T. A., Abed U., Goosmann C., Hurwitz R., Schulze I., Wahn V.,Weinrauch Y., Brinkmann V., Zychlinsky A.
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