Диссертация (1146649), страница 23
Текст из файла (страница 23)
Vol. 133(2). P. 472–480.139. Craig, J.M. The pathological changes in the liver in cystic fibrosis ofthe pancreas / J.M. Craig, H. Haddad, H. Schwachman // A.M.A. journal of diseases of children. – 1957. – Vol. 93 (4). – P. 357–369.140. Cupfer, S.S. Pathophysiology of celiac disease / S.S. Cupfer, B. Jabry// Gastrointestinal endoscopy clinics of North America. – 2012. – Vol. 24 (4). –P. 639–660.141. Cystic fibrosis : book / ed. by M. Hodson, D.
Geddes, A. Bush. – 3rded. – London : Hodder Arnold, 2007. – P. 21–45.142. Cystic fibrosis mutation database [Электронный ресурс]. – URL:http://www.genet.sickkids.on.ca/cftr/143. Cystic fibrosis transmembrane conductance regulator: a chloridechannel with novel regulation / M.J. Welsh [et al.] // Neuron. – 1992. – Vol.
8(5). – P. 821–829.144. Davis, S.E. Identifying the origins of cystic fibrosis lung disease /S.E. Davis, T. Ferkol // The New England journal of medicine. – 2013. – Vol. 368,No. 21. – P. 2026–2028.145. Demonstration that CFTR is a chloride channel by alteration of itsanion selectivity / M.P. Anderson [et al.] // Science. – 1991. – Vol. 253, Issue5016. – P. 202–205.136146. Dewar, D.H. Clinical features and diagnosis of celiac disease /D.H. Dewar, P.J. Ciclitira // Gastroenterology.
– 2005. – Vol. 128 (4), Suppl. 1. –P. S19–S24.147. Di Magno I.P. Impaired cholecystokinin-pancreozymin secretion,intraluminal dilution, and maldigestion of fat in sprue / I.P. Di Magno, W.L. Go,W.H. Summerskil, // Gastroenterology. – 1972. – Vol. 63 (1). – P. 25–32.148. Di Sabatino, A. Coeliac disease / A. Di Sabatino, G.R. Corazza // TheLancet. – 2009. – Vol.
373, No. 9673. – P. 1480–1493.149. Dicke, W.K. Coeliac disease. The presence in wheat of a factor havinga deleterious effect in cases of coeliac disease / W.K. Dicke, H.A. Weijers,J.H. van de Kamer // Acta paediatrica Scandinavica. 1953. Vol. 42 (1). Р. 34–42.150. Differences in the risk of celiac disease associated with HLA-DQ2.5or HLA-DQ2.2 are related to sustained gluten antigen presentation / L.E.
Fallang[et al.] // Nature immunology. – 2009. – Vol. 10, Issue 10. – P. 1096–1101.151. Domenick, H.C. Ultrastructure of liver biopsies in cystic fibrosis (abstract) / H.C. Domenick, P. Arends, H.C. van Bassewitz // Cystic Fibrosis Quarterly Annotated References. – 1974. – P. 13.152. Durie, P.R. The pathophysiology of the pancreatic defect in cystic fibrosis / P.R. Durie // Acta paediatrica Scandinavica. – 1989. – Suppl. 363. – P. 41–44.153. Early diagnosis of cystic fibrosis through neonatal screening preventssevere malnutrition and improves long-term growth. Wisconsin Cystic FibrosisNeonatal Screening Study Group / P.M. Farrell [et al.] // Pediatrics.
– 2001. –Vol. 107 (1). – P. 1–13.154. Effectiveness of PTC124 treatment of cystic fibrosis caused by nonsense mutations: a prospective phase II trial / E. Kerem [et al.] // The Lancet. 2008. Vol. 372, No. 9640. P. 719–727.137155. Effects of topically delivered benzamil and amiloride on nasal potential difference in cystic fibrosis / T. Hofmann [et al.] // American journal of respiratory and critical care medicine. – 1998. – Vol.
157 (6). – P. 1844–1849.156. Efficient degradation of gluten by a prolylendoprotease in a gastrointestinal model: implications for coeliac disease / C. Mitea [et al.] // Gut. 2008. Vol. 57 (1). – P. 25–32.157. Elevated plasma neurotensin in adults with cystic fibrosis / J.M. Allen[et al.] // Clinical Science. – 1983. – Vol. 65, No. 1.
– P. 64.158. European Society for Pediatric Gastroenterology, Hepatology, andNutrition Guidelines for the Diagnosis of Coeliac Disease / S. Husby [et al.] //Journal of pediatric gastroenterology and nutrition. – 2012. – Vol. 54, Issue 1. –P. 136–160.159. Evidence for periciliary liquid layer depletion, not abnormal ion composition, in the pathogenesis of cystic fibrosis airways disease / H. Matsui [et al.] //Cell. – 1998. – Vol.
95, Issue 7. – P. 1005–1015.160. Evidence of intestinal inflammation in patients with cystic fibrosis /S.L. Werlin [et al.] // Journal of pediatric gastroenterology and nutrition. – 2010. –Vol. 51, Issue 3. – P. 304–308.161. Evidence that celiac disease is primarily associated with a DC locusallelic specificity / R. Tosi [et al.] // Clinical immunology and immunopathology. 1983. – Vol. 28, Issue 3. P. 395–404.162. Exocrine pancreatic function in children with coeliac disease beforeand after a gluten free diet / A.
Carroccio [et al.] // Gut. – 1991. – Vol. 32 (7). –P. 796–799.163. Farrell, R.J. Diagnosis of coeliac sprue / R.J. Farrell, C.P. Kelly // TheAmerican journal of gastroenterology. – 2001. – Vol. 96, Issue 12. – P. 3237–3246.164. Farrell, R.J. Celiac Sprue / R.J. Farrell, C.P. Kelly // The New England Journal of Medicine. – 2002. – Vol. 346, No.
3. – P. 180–188.138165. Freeman, H.J. Hepatobiliary and pancreatic disorders in celiac disease/ H.J. Freeman // World journal of gastroenterology. – 2006. – Vol. 12 (10). –P. 1503–1508.166. Gallbladder inertia and sluggish enterohepatic circulation of bile saltsin coeliac disease / T.S.
Low-Beer [et al.] // The Lancet. – 1971. – Vol. 297,No. 7707. – P. 991–994.167. Gastroesophageal reflux (symptomatic and silent): a potentially significant problem in patients with cystic fibrosis before and after lung transplantation /B.M. Button [et al.] // The Journal of heart and lung transplantation. – 2005. –Vol. 24, Issue 10. – P.
1522–1529.168. Gastrointestinal transit times and motility in patients with cystic fibrosis / C. Hedsund [et al.] // Scandinavian journal of gastroenterology. – 2012. –Vol. 47, No. 8–9. – P. 920–926.169. Genetic determination of exocrine pancreatic function in cystic fibrosis / P. Kristidis [et al.] // The American journal of human genetics. – 1992.
–Vol. 50 (6). – P. 1178–1184.170. Genetics in coeliac disease / D.A. van Heel [et al.] // Best Practice &Research: Clinical Gastroenterology. – 2005. – Vol. 19, Issue 3. – P. 323339.171. Gluten induces an intestinal cytokine response strongly dominated byinterferon gamma in patients with celiac disease / E.M. Nilsen [et al.] // Gastroenterology. – 1998. – Vol. 115 (3).
– P. 551–563.172. Goldberg, H.J. Advances in lung transplantation for patients who havecystic fibrosis / H.J. Goldberg, A. Deykin // Clinics in chest medicine. – 2007. –Vol. 28, Issue 2. – P. 445–457.173. Gut Microflora. Digestive physiology and pathology / edited by J.C.
Rambaud, J.-P. Buts, G. Corthier, B. Flourie. – Paris : John Libbey Eurotext,2006. – 247 p.174. Halstensen, T.S. Activated T lymphocytes in the celiac lesion: nonproliferative activation (CD25) of CD4+ alpha/beta cells in the lamina propria butproliferation (Ki-67) of alpha/beta and gamma/delta cells in the epithelium /139T.S.
Halstensen, P. Brandtzaeg // European journal of immunology. – 1993. –Vol. 23, Issue 2. – P. 505–510.175. Hepatic complication in cystic fibrosis / H.T. Psacharopoulos [et al.] //The Lancet. — 1981. – Vol. 318, No. 8237. – P. 78–80.176. Hepatic parenchymal cells in cystic fibrosis: ultrastructural evidencefor abnormal intracellular transport / W. Bradford [et al.] // Pediatric pathology. –1983.
– Vol. 1 (3). – P. 269–279.177. High prevalence of coeliac disease in apparently healthy Iranian blooddonors / B. Shahbazkhani [et al.] // European Journal of Gastroenterology and Hepatology. – 2003. – Vol. 15, Issue 5. – P. 475–478.178. HLA DQ gene dosage and risk and severity of celiac disease /J.A. Murray [et al.] // Clinical Gastroenterology and Hepatology. – 2007. – Vol. 5,Issue 12. – P. 1406–1412.179.
HLA-DQ2-restricted gluten-reactive T cells produce IL-21 but not IL17 or IL-22 / M. Bodd [et al.] // Mucosal immunology. – 2010. – Vol. 3, Issue 6. –P. 594–601.180. HLA-DQ2 and -DQ8 signatures of gluten T cell epitopes in celiac disease / S. Tollefsen [et al.] // The Journal of Clinical Investigation. – 2006. –Vol. 116, Issue 8. – P.
2226–2236.181. HLA-DQ typing in the diagnostic algorithm of celiac disease /B. Piccini [et al.] // Revistaespañola de enfermedadesdigestivas :organooficial de laSociedad Española de PatologíaDigestiva. – 2012. – Vol. 104, Issue 5. – P. 248–254.182. Hulcrantz, R. Morfological findings in the liver of children with cysticfibrosis: a light and electron microscopical study / R. Hulcrantz, S. Mengarelli,B. Strandvic // Hepatology. – 1986. – Vol. 6, Issue 5. – P. 881–889.183. Identification of the cystic fibrosis gene: cloning and characterizationof complementary DNA / J.R. Riordan [et al.] // Science. – 1989.
– Vol. 245, Issue 4922. – P. 1066–1073.140184. Identification of the cystic fibrosis gene: genetic analysis / B. Kerem[et al.] // Science. – 1989. – Vol. 245, Issue 4922. – P. 1073–1080.185. Immunohystochemical analysis of CFTR in normal and disruptedspermatogenesis / S. Teixeira [et al.] // Systems Biology in Reproductive Medicine. – 2013.
![](https://s.studizba.com/z.php?f=/templates/si/i/noavatar.png&w=100&h=100&t=1"){kind=avatar}
