Диссертация (1174350), страница 12
Текст из файла (страница 12)
(Lausanne). – 2017. – Vol. 4. – P. 83. –doi: 10.3389/fmed.2017.00083.9775. Current understanding of molecular and cellular mechanisms in fibroplasiaand angiogenesis during acute wound healing / N. S. Greaves, K. J. Ashcroft,M. Baguneid, A. Bayat // J. Dermatol. Sci. – 2013. – Vol. 72 (3). – P. 206–217.76. Cutaneous scarring: basic science, current treatments, and future directions/ C.
D. Marshall, M. S. Hu, T. Leavitt et al. // Adv. Wound Care (New Rochelle). – 2018.– Vol. 7 (2). – P. 29–45. – doi: 10.1089/wound.2016.0696.77. De Boer, W. I. Perspectives for cytokine antagonist therapy in COPD/ W. I. De Boer // Drug Discov. Today. – 2005. – Vol. 10 (2). – P. 93–106.78. Deep dermal fibroblasts contribute to hypertrophic scarring / J. F. Wang,C. Dodd, H. A.
Shankowsky et al. // Lab. Invest. – 2008. – Vol. 88 (12). – P. 1278–1290.– doi: 10.1038/labinvest.2008.101.79. Defective thymocyte maturation in p44 MAP kinase (Erk 1) knockout mice/ G. Pagès, S. Guérin, D. Grall et al. // Science. – 1999. – Vol. 286 (5443). – P. 1374–1377.80. Delayed topical p38 MAPK inhibition attenuates full-thickness burn woundinflammatory signaling / D.
Carter, A Warsen, K. Mandell et al. // J. Burn Care. Res. –2014. – Vol. 35 (2). – P. 83–92. – doi:10.1097/BCR.0b013e31828a8d6e.81. Diegelmann, R. F. Wound healing: an overview of acute, fibrotic and delayedhealing / R. F. Diegelmann, M. C. Evans // Front Biosci.
– 2004. – Vol. 9. – P. 283–289.82. Doersch, K. M. The contribution of interleukin-2 to effective wound healing/ K.M. Doersch,D.J. Dello Stritto,M. K. Newell-Rogers// Exp.Biol.Med.(Maywood). – 2017. – Vol. 242 (4). – P. 384–396. – doi: 10.1177/1535370216675773.83. Does hyperbaric oxygen therapy work in facilitating acute wound healing:a systematic review / P.
B. Dauwe, B. J. Pulikkottil, L. Lavery et al. // Plast. Reconstr.Surg. – 2014. – Vol. 133 (2). – P. 208–215.84. Dong, S. MicroRNA-22 may promote apoptosis and inhibit the proliferationof hypertrophic scar fibroblasts by regulating the mitogen-activated protein kinasekinase/extracellular signal-regulated kinase/p21 pathway / S. Dong, Y. Sun // Exp. Ther.Med. – 2017.
– Vol. 14 (4). – P. 3841–3845. – doi: 10.3892/etm.2017.4942.9885. Dovi, J. V. Accelerated wound closure in neutrophil-depleted mice/ J. V. Dovi, L. K. He, L. A. DiPietro // J. Leukoc. Biol. – 2003. – Vol. 73. – P. 448–455.86. Edwards, R. Bacteria and wound healing / R. Edwards, K. G. Harding // Curr.Opin. Infect. Dis. – 2004. – Vol. 17 (2). – P.
91–96.87. Effect of equal daily doses achieved by different power densities of low-levellaser therapy at 635 nm on open skin wound healing in normal and corticosteroid-treatedrats / P. Gál, M. Mokrý, B. Vidinský et al. // Lasers Med. Sci. – 2009. – Vol. 24 (4). –P. 539–547. – doi: 10.1007/s10103-008-0604-9.88. Effect of ibuprofen on the inflammatory response to surgical wounds/ Y. L.
Dong, R. Y. Fleming, T. Z. Yan et al. // J. Trauma. – 1993. – Vol. 35 (3). –P. 340–343.89. Elsaie, M. L. Lasers for scars: A review and evidence-based appraisal/ M. L. Elsaie, S. Choudhary // J. Drugs Dermatol. – 2010. – Vol. 9. – P. 1355–1362.90. Endoglin overexpression modulates cellular morphology, migration, andadhesion of mouse fibroblasts / M. Guerrero-Esteo, P. Lastres, A.
Letamendia et al.// Eur. J. Cell Biol. – 1999. – Vol. 78 (9). – P. 614–623. – doi: 10.1016/S01719335(99)80046-6.91. Energy metabolism of experimental wounds at various oxygen environments./ J. Kivisaari, T. Vihersaari, S. Renvall, J. Niinikoski // Ann. Surg. – 1975. – Vol.
181. –P. 823–828.92. Extracellularsignal-regulatedkinase 2isnecessary formesodermdifferentiation / Y. Yao, W. Li, J. Wu et al. // Proc. Natl. Acad. Sci. USA. – 2003. –Vol. 100 (22). – P. 12759–12764.93. Five-year safety data for more than 55,000 subjects following breastimplantation: comparison of rare adverse event rates with silicone implants versus nationalnorms and saline implants / N. Singh, G. J. Picha, B. Hardas et al. // Plast. Reconstr. Surg. –2017. – Vol.
140 (4). – P. 666–679. – doi: 10.1097/PRS.0000000000003711.94. Follistatin and the breast implant capsule / B. A. Frenkiel, P. Temple-Smith,D. de Kretser, G. J. Southwick // Plast. Reconstr. Surg. Glob. Open. – 2017. –Vol. 5 (3). – P. e1258. – doi: 10.1097/GOX.0000000000001258.9995. Franz, M. G. Optimizing healing of the acute wound by minimizingcomplications / M. G. Franz, D. L. Steed, M.
C. Robson // Curr. Probl. Surg. – 2007. –Vol. 44 (11). – P. 691–763.96. Fulton, J. E. Silicone gel sheeting for the prevention and managementof evolving hypertrophic and keloid scars / J. E. Fulton // Dermatol. Surg. – 1995. –Vol. 21. – P. 947–951.97. Garrington, T. P. Organization and regulation of mitogen-activated proteinkinase signaling pathways / T. P. Garrington, G. L. Johnson // Curr.
Opin. Cell Biol. –1999. – Vol. 11 (2). – P. 211–218.98.healing:Gompelman, M. Update on the role of infection and biofilms in woundpathophysiologyandtreatment/ M. Gompelman,S. A. V. van Asten,E. J. G. Peters // Plast. Reconst. Surg. – 2016. – Vol. 138 (3S). – P. 61S–70S.99.Grabowska, A. I. FOXN1 transcription factor in epithelial growth andwound healing / A. I. Grabowska, T. Wilanowski // Mol. Cell Biol.
– 2017. –Vol. 37 (17). – P. e00110–e00117. – doi: 10.1128/MCB.00110-17.100. Guo, S. Factors affecting wound healing / S. Guo, L. A. DiPietro // J. Dent.Res. – 2010. – Vol. 89 (3). – P. 219–229.101. Har-Shai, Y. Intralesional cryotherapy for enhancing the involutionof hypertrophic scars and keloids / Y. Har-Shai, M. Amar, E. Sabo // Plast. Reconstr.Surg.
– 2003. – Vol. 111 (6). – P. 1841–1852.102. Hart, C. E. Dermagraft: use in the treatment of chronic wounds / C. E. Hart,A. Loewen-Rodriguez, J. Lessem // Adv. Wound Care (New Rochelle). – 2012. –Vol. 1 (3). – P. 138–141.103. Hidalgo, D. A. Breast augmentation / D. A. Hidalgo, J. A. Spector// Plast.Reconst.Surg.–2014.–Vol. 133 (4).–P. 567e–583e.–doi: 10.1097/PRS.0000000000000033.104. Holmström, K. M. Cellular mechanisms and physiological consequencesof redox-dependent signaling / K. M. Holmström, T. Finkel // Nat. Rev. Mol. Cell.Biol.
– 2014. – Vol. 15 (6). – P. 411–421. – doi: 10.1038/nrm3801.100105. Human precision-cut liver slices as a model to test antifibrotic drugsin the early onset of liver fibrosis / I. M. Westra, H. A. Mutsaers, T. Luangmonkong et al.// Toxicol. In Vitro. – 2016. – Vol. 35. – P. 77–85. – doi: 10.1016/j.tiv.2016.05.012.106. Identification and analysis of mitogen-activated protein kinase (MAPK)cascades in Fragaria vesca / H. Zhou, S. Ren, Y. Han et al. // Int. J. Mol. Sci.
– 2017. –Vol. 18 (8). – P. 1766. – doi: 10.3390/ijms18081766.107. Impaired wound healing / N. B. Menke, K. R. Ward, T. M. Witten et al.// Clin. Dermatol. – 2007. – Vol. 25 (1). – P. 19–25. – doi: https://doi.org/10.1016/j.clindermatol.2006.12.005.108. Inamed silicone breast implant core study results at 6 years / S.
L. Spear,D. K. Murphy, A. Slicton, P. S. Walker // Plast. Reconstr. Surg. – 2007. – Vol. 120 (7). –P. 8S–16S; Discussion 17S–18S.109. Induction of collagenase-3 (MMP-13) expression in human skin fibroblastsby three-dimensional collagen is mediated by p38 mitogen-activated protein kinase/ L. Ravanti, J. Heino, C. López-Otín, V.
M. Kähäri // J. Biol. Chem. – 1999. –Vol. 274 (4). – P. 2446–2455.110. Inhibition of lipid peroxidation restores impaired vascular endothelialgrowth factor expression and stimulates wound healing and angiogenesis in thegenetically diabetic mouse / D.
Altavilla, A. Saitta, D. Cucinotta et al. // Diabetes. –2001. – Vol. 50 (3). – P. 667–674.111. Internationalclinicalrecommendationsonscarmanagement/ T. A. Mustoe, R. D. Cooter, M. H. Gold et al. // Plast. Reconstr. Surg. – 2002. –Vol. 110 (2). – P. 560–571.112. Involvement of p38 MAPK in reactive astrogliosis induced by ischemicstroke / R. G. Choudhury, M. G. Ryou, E. Poteet et al. // Brain Res. – 2014. –Vol.
1551. – P. 45–58. – doi: 10.1016/j.brainres.2014.01.013.113. Ito, R. Influence of MAPK Signaling on ischemic wound healingin the elderly / R. Ito, Q. Zhang // Plast. Reconstr. Surg. – 2014. – March Suppl. – P. 158.114. Jalali, M. Current use of steroids in management of abnormal raised skinscars / M. Jalali, A. Bayat // Surgeon. – 2007. – Vol. 5 (3). – P. 175–180.101115.
Janis, J. E. Introduction to “Current Concepts in Wound Healing: Update2016” / J. E. Janis, C. E. Attinger, L. Lavery // Plast. Reconstr. Surg. – 2016. –Vol. 138 (3S). – P. 7–8. – doi: 10.1097/PRS.0000000000002697.116. Janis, J. E. Wound healing: Part II. Clinical applications / J. E.
Janis,B. Harrison // Plast. Reconstr. Surg. – 2014. – Vol. 133 (3). – P. 383–392.117. Janis, J. E. Wound healing: Part 1. Basic science / J. E. Janis, B. Harrison// Plast.Reconstr.Surg.–2016.–Vol. 138 (3 Suppl.).–Р. 9S–17S.–doi: 10.1097/PRS.0000000000002773.118. JNK, p38, ERK, and SGK1 inhibitors in cancer / J.
Cicenas, E. Zalyte,A. Rimkus et al. // Cancers (Basel). – 2017. – Vol. 10 (1). – P. 1–12.119. Kavalukas, S. L. Nutrition and wound healing : an update / S. L. Kavalukas,A. Barbul// Plast.Reconstr.Surg.–2011.–Vol. 127.–P. 38S–43S. –doi: 10.1097/PRS.0b013e318201256c.120. Kragholm, K. Emerging treatment options to improve cardiovascularoutcomes in patients with acute coronary syndrome: focus on losmapimod / K. Kragholm,L. K. Newby, C.
Melloni // Drug Des. Devel. Ther. – 2015. – Vol. 9. – P. 4279–4286. –doi: 10.2147/DDDT.S69546.121. Kwan, P. O. Biological principles of scar and contracture / P. O. Kwan,E. E. Tredget// HandClin.–2017.–Vol. 33 (2).–P. 277–292.–doi: 10.1016/j.hcl.2016.12.004.122. Kyriakis, J.
M. Mammalian MAPK signal transduction pathways activatedby stress and inflammation: a 10-year update / J. M. Kyriakis, J. Avruch // Physiol. Rev. –2012. – Vol. 92 (2). – P. 689–737. – doi: 10.1152/physrev.00028.2011.123. Kyttaris, V. C. Kinase inhibitors: a new class of antirheumatic drugs/ V. C. Kyttaris // Drug Des. Devel. Ther. – 2012. – Vol. 6.