Диссертация (1174348), страница 19
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Чулков, Н. К. Вереина, С.П. Синицын, В.Ф.Долгушина // Вестник Российской академии медицинских наук. – 2013. – Т.68, № 11. – С. 22-25.58. Шетикова, О.В. Определение роли ангиогенных факторов в прогнозированииразвития преэклампсии: дис. ... канд. мед. наук: 14.01.01/ Шетикова ОльгаВладимировна. – М., 2010.59. Щеголев, А.И. Диагностическая значимость гистологических характеристикплаценты зависит от вида и степени тяжести преэклампсии / А.И.
Щеголев,В.М. Ляпин // В сборнике: Материалы V съезда Российского обществапатологоанатомов Российское общество патологоанатомов. – 2017. – С. 369370.60. Щеголев, А.И. Морфометрическая характеристика терминальных ворсинплаценты при преэклампсии / Щеголев А.И., Дубова Е.А., Павлов К.А., ЛяпинВ.М., Куликова Г.В., Шмаков Р.Г. // Бюллетень экспериментальной биологии имедицины. – 2012. – No 7.
– С. 104-107.61. Ahmad, S. Elevated placental soluble vascular endothelial growth factor receptor-1inhibits angiogenesis in preeclampsia / Ahmad S., Ahmed A. // Circ Res. – 2004.Vol. 95. – P. 884-91.62. Ahmed, A. Colocalisation of vascular endothelial growth factor and its Flt-1receptor in human placenta / Ahmed A., Li X., Dunk C., Whittle MJ., Rushton DI.,Rollason T. // Growth Factors. – 1995. – Vol. 12. – P. 235- 43.63. Ahmed, A. Regulation of placental vascular endothelial growth factor (VEGF) andplacenta growth factor (PIGF) and soluble Flt-1 by oxygen – a review / Ahmed A., Dunk C., Ahmad S., Khaliq A. // Placenta. – 2000. – Vol. 21.
– P. 16-24.64. Albayrak, M. Atypical preeclampsia and eclampsia: report of four cases and revive122of the literature / Albayrak M. [et al.] // J Turk Ger Gynecol Assoc. – 2010. –Vol.11 . – P.115-117.65. Alpoim, P.N. Polymorphisms in endothelial nitric oxide synthase gene in early andlate severe preeclampsia / P.
N. Alpoim [et al.] // Nitric Oxide. – 2014. – Vol. 42. –Р. 19- 23.66. American College of Obstetricians and Gynecologists / Diagnosis andmanagement of preeclampsia and eclampsia: ACOG practice bulletin no. 33.(Level III) // Obstet Gynecol. – 2002. – Vol. 99. P. – 159–167.67. An analysis of the differences between early and late preeclampsia with severehypertension / X.
L. Li [et al.] // Pregnancy Hypertens. – 2016. – Vol. 6, № 1. – Р.47-52.68. Antonios, T.F. Microvascular remodelling in preeclampsia: quantifying capillaryrarefaction accurately and independently predicts preeclampsia / T. F. Antonios [etal.] // Am J Hypertens. – 2013. – Vol. 26, № 9. – P. 1162-1169.69.
Astudillo, R. Expectant management in pregnant women with early and severepreeclampsia and concomitant risk factors / Astudillo R. // PregnancyHypertension: An international Journal of Women’s Cardiovascular Health. –2013. – Vol. 3. – P. 235-241.70. Baergen, R.N. Manual of Benirschke and Kaufmann’s Pathology of the HumanPlacenta / Baergen R.N. // New York: Springer. – 2005.71. Bartynski, W.S. Distinct imaging patterns and lesion distribution in posteriorreversible encephalopathy syndrome / Bartynski W.S., Boardman J.F. // AJNR AmJ Neuroradiol.
– 2007. – Vol.28. – P.1320–1327.72. Bembalgi, S. A Study of Eclampsia Cases Associated with Posterior ReversibleEncephalopathy Syndrome / Bembalgi S., Kamate V., Shruthi K.R. //J Clin DiagnRes. – 2015. – Vol.9б№7. – QC05-QC07.73. Benedetto, C. Synergistic effect of renin-angiotensin system and nitric oxidesynthase genes polymorphisms in pre-eclampsia / C. Benedetto [et al.] // ActaObstet Gynecol Scand. – 2007.
– Vol. 86, № 6. – Р. 678-682.74. Benisrchke, K. Pathology of the Human Placenta, 5th ed. / Benisrchke K.,123Kaufmann P., Baergen R.N. // New York: Springer – 2006.75. Benyo, D.F. Expression of inflammatory cytokines in placentas from women withpreeclampsia / Benyo DF., Smarason A., Redman CW., Sims C., Conrad KP. // JClin Endocrinol Metab.
– 2001. -–Vol. 86(6). – P. 2505–1210.76. Blaauw, J. Abnormal endothelium-dependent microvascular reactivity in recentlypree- clamptic women / Blaauw J. [et al.] // Obstet Gynecol. – 2005. – Vol. 105, №3. – Р. 626-632.77. Boeldt, D. S. Vascular adaptation in pregnancy and endothelial dysfunction inpreeclampsia / D. S. Boeldt, I. M. Bird // J Endocrinol. – 2017. – Vol. 232, № 1. –Р. 27-44.78. Bombell, S.
Tumour necrosis factor (-308A) polymorphism in pre-eclampsia:meta-analysis of 16 case-control studies / S. Bombell, W. McGuire // Aust N Z JObstet Gynaecol. – 2008. – Vol. 48, № 6. – Р. 547-551.79. Bouças, A.P. Role of innate immunity in preeclampsia: a systematic review / A. P.Bouças [et al.] // Reprod Sci.
– 2017. – Vol. 24, № 10. – P. 1362-1370.80. Brito, J.A. Enzyme activity of superoxide dismutase and zincemia in women withpreeclampsia / Brito J.A. [et al.] // Nutr Hosp. – 2013. – Vol.28, №2. – P.486-490.81. Brockelsby, J. VEGF via VEGF receptor-1 (Flt-1) mimics preeclamptic plasma ininhibiting uterine blood vessel relaxation in pregnancy: implications in thepathogenesis of preeclampsia / Brockelsby J., Hayman R., Ahmed A., Warren A.,Johnson I., Baker P.
// Lab Invest. – 1999. – Vol. 79. – P. 1101-11.82. Brodszki, J. Vascular mechanical properties and endothelial function in preeclampsia with special reference to bilateral uterine artery notch / J. Brodszki [etal.] // Acta Obstet Gynecol Scand. – 2008. – Vol. 87, № 2. – Р. 154-162.83. Brooks, S.A. miRNAs is a common regulator of the transforming growth factor(TGF)-B-pathway in the preeclamptic placenta and cadmium-trated trophoblasta:links between the environment, the epigenome and preeclampsia / Brooks S.A. //Food Chem Toxicol.
– 2016. – Vol. 98(PtA). – P. 50-57.84. Brosens, J.J. The myometrial junctional zone spiral arteries in normal andabnormal pregnancies: a review of the literature / Brosens J.J., Pijnenborg R.,124Brosens I.A. // American Journal of Obstetrics and Gynecology. – 2002. Vol. 187.– P. 1416-1423.85. Bürk, M.R. Severely reduced presence of tissue macrophages in the basal plate ofpre-eclamptic placentae / Bürk MR, Troeger C, Brinkhaus R, Holzgreve W, HahnS. // Placenta.- 2001. – Vol. 22(4). – P. 309–1610.86. Burnett, M.M.
Presentation of reversible posterior leukoencephalopathy syndromein patients on calcineurin inhibitors / Burnett M.M. [et al.] // Clin NeurolNeurosurg. – 2010. – Vol.112. –P.886–889.87. Can, M. Oxidative stress and apoptosis in preeclampsia / Can M. [et al.] // Tissueand Cell.
– 201. – Vol. 43, № 6. – Р. 477-481.88. Casey, S.O Posterior reversible leukoencephalopathy syndrome / Casey S.O. [etal.] // AJNR Am. J. Neuroradiol. – 2000. – Vol. 21. № 7. – P. 1199–1206.89. Chaiworapongsa, T. Evidence supporting a role for blockade of the vascularendothelial growth factor system in the pathophysiology of preeclampsia /Chaiworapongsa T., Romero R, Espinoza J, Bujold E, Mee Kim Y, Goncalves LF,et al.
// Am J Obstet Gynecol. – 2004. – Vol. 190. – P. 1541-47.90. Chaiworapongsa, T. Plasma soluble vascular endothelial growth factor receptor-1concentration is elevated prior to the clinical diagnosis of preeclampsia /Chaiworapongsa T., Romero R, Kim YM, Kim GJ, Kim MR, Espinoza J, et al. // JMatern Fetal Neonatal Med. – 2005. – Vol.
17. – P. 3-18.91. Chaiworapongsa, T. Pre-eclampsia part 1: current understanding of itspathophysiology / Chaiworapongsa T., Chaemsaithong, P., Yeo, L., Romero, R. //Nat. Rev. Nephrol. – 2014. – Vol. 10 (8). – P.466-480.92. Chen, W. Significance of toll-like receptor 4 signaling in peripheral bloodmonocytes of pre-eclamptic patients / W. Chen [et al.] // HypertensPregnancy. – 2015.
– Vol. 34, № 4. – Р. 486-494.93. Chou, M.C. Posterior reversible encephalopathy syndrome: magnetic resonanceimaging and diffusion-weighted imaging in 12 cases / Chou M.C. [et al.] //Kaohsiung J Med Sci. – 2004. – Vol. 20: 8. – P. 381–388.94. Conti, E. Growth factors in preeclampsia: a vascular disease model. A failed125vasodilation and angiogenic challenge from pregnancy onwards? / Conti, E.,Zezza, L., Ralli, E., Caserta, D., Musumeci, M.B., Moscarini, M., et al. // CytokineGrowth Factor Rev. – 2013.
– Vol. 24 (5). – P. 411-425.95. Contro, E. Cell-free fetal DNA for the prediction of pre-eclampsia at the first andsecond trimesters: a systematic review and meta-analysis. / E. Contro, D.Bernabini, A. Farina //Mol Diagn Ther. – 2017. – Vol. 21, № 2. – Р. 125-135.96. Cornelius, D.C. TH17- and IL-17- mediated autoantibodies and placental oxidativestress play a role in the pathophysiology of pre-eclampsia / Cornelius D.C.,Lamarca B. // Minerva Ginecol.
– 2014. – Vol. 66,№ 3. – P. 243-249.97. Covarrubias, D.J. Posterior reversible encephalopathysyndrome: prognostic utilityof quantitative diffusion-weighted MR images / Covarrubias D.J. [et al.] // Am JNeuroradiol. –2002. –Vol.23:6. – P.1038–1048.98. Dai, B. The polymorphism for endothelial nitric oxide synthase gene, the level ofnitric oxide and the risk for pre-eclampsia: a meta-analysis / B. Dai [et al.] // Gene.– 2013. – Vol. 519, № 1. – Р. 187-193.99.
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