Диссертация (1144759), страница 33
Текст из файла (страница 33)
Ca2+ influx via the Na+/Ca2+ exchanger is enhanced in malignanthyperthermia skeletal muscle // J. Biol. Chem. 2014. Vol. 289, № 27. P. 19180-90.44. Ambery AG, Tackett L, Penque BA, Brozinick JT, Elmendorf JS. Exercise trainingprevents skeletal muscle plasma membrane cholesterol accumulation, cortical actinfilament loss, and insulin resistance in C57BL/6J mice fed a western-style high-fatdiet // Physiol. Rep.
2017 Vol. 5, № 16. pii: e13363.19745. Antonov S. M., I. I. Krivoi, T. M. Drabkina, Mironova EV, Evstratova AA.Neuroprotective effect of ouabain and Bcl-2 peptide expression duringhyperactivation of NMDA receptors in rat brain cortical neurons in vitro // Dokl.Biol. Sci. 2009. Vol. 426. P. 207-209.46.
Aperia A., Intern. J. New roles for an old enzyme: Na, K-ATPase emerges as aninteresting drug target // Med. 2007. Vol. 261. P. 44–52.47. Arnon A., Hamlyn J.M., Blaustein M.P. Ouabain augments Ca2+ transients inarterial smooth muscle without raising cytosolic Na+ // Am. J. Physiol. Heart Circ.Physiol. 2000. Vol. 279. P. H679-H691.48. Bagrov A. Y., Shapiro J. I., Fedorova O. V. Endogenous cardiotonic steroids:physiology, pharmacology, and novel therapeutic targets // Pharmacol.
Rev. 2009.Vol. 61, № 1. P. 9-38.49. Bagrov A.Y., Fedorova O.V., Dmitrieva R.Y., Howald W.N., Hunter A.P.,Kuznetsova E.A., Shpen V.M. Characterization of a urinary bufodienolide Na+,K+ATPase inhibitor in patients after acute myocardial infarction // Hypertens. 1998.Vol. 31. P. 1097-1103.50. Balasubramanian N., Scott D.W., Castle J.D., Casanova J.E., Schwartz M.A.
Arf6and microtubules in adhesion dependent trafficking of lipid rafts // Nat Cell Biol.2007. Vol. 9. № 12. P. 1381-91.51. Baldwin K.M., Haddad F., Pandorf C.E., Roy R.R., Edgerton V.R. Alteration inmuscle mass and contractile phenotype in response to unloading models: role oftranscriptional/pretranslational mechanisms // Front. Physiol. 2013. Vol. 4. P. 284.52. Balzan S., D'Urso G., Ghione S., Martinelli A., Montali U.
Selective inhibition ofhuman erythrocyte Na+/K+ ATPase by cardiac glycosides and by a mammaliandigitalis like factor // Life Sci. 2000. V. 67. P. 1921-1928.53. Bao H., Sun H., Xiao Y., Zhang Y., Wang X., Xu X., Liu Z., Fang J., Li Z.Functional interaction of nicotinic acetylcholine receptors and Na +/K+ATPase fromLocusta migratoria manilensis (Meyen) // Sci. Rep. 2015. Vol. 5, P.
8849.54. Barrientos G., Sánchez-Aguilera P., Jaimovich E., Hidalgo C., Llanos P.Membrane Cholesterol in Skeletal Muscle: A Novel Player in Excitation-198Contraction Coupling and Insulin Resistance // J. Diabetes Res. 2017. Vol. 2017.Article ID 3941898.55. Benjamin W. Hughes, Linda L. Kusner, Henry J. Kaminski , Moleculararchitecture of the neuromuscular junction // Muscle Nerve. 2006. Vol.
33, № 4. P.445-461.56. Benowitz N.L., Zevin S., Jacob P. Sources of variability in nicotine and cotininelevels with use of nicotine nasal spray, transdermal nicotine, and cigarette smoking// Br. J. Clin. Pharmacol. 1997. Vol. 43. P. 259-267.57. Benziane B, U. Widegren, S. Pirkmajer, Henriksson J., Stepto N.K., Chibalin A.V.Effect of exercise and training of phospholemman phosphorylation in humanskeletal muscle // Am.
J. Physiol. Endocrinol. Metab. 2011. Vol. 301. P. E456.58. Benziane B., Chibalin A.V. Skeletal muscle sodium pump regulation: atranslocation paradigm // Am. J. Physiol. Endocrinol. Metab. 2008. Vol. 295. P.E553–E558.59. Benziane B., M. Bjornholm, S.
Pirkmajer, Austin R.L., Kotova O., Viollet B.,Zierath J.R., Chibalin A.V. Activation of AMP-activated protein kinase stimulatesNa+,K+-ATPase activity in skeletal muscle cells // ActiJ. Biol. Chem. 2012. Vol.287. P. 23451–23463.60. Berchtold M.W., Brinkmeier H., Muntener M. Calcium ion in skeletal muscle: itscrucial role for muscle function, plasticity, and disease // Physiol. Rev. 2000. Vol.80. P. 1216-1263.61. Bibert S., Roy S., Schaer D., Horisberger J.D., Geering K. Phosphorylation ofphospholemman (FXYD1) by protein kinases A and C modulates distinct Na, KATPase isozymes // J. Biol. Chem.
2008. Vol. 283. P. 476–486.62. Biser P.S., Thayne K.A., Fleming W.W., Taylor D.A. Na, K-ATPase α-subunitisoform distribution and abundance in guinea-pig longitudinal muscle/myentericplexus after exposure to morphine // Brain Res. 2002. Vol. 931. P. 186–193.63. Blanco G., Mercer R.W. Isozymes of the Na-K-ATPase: heterogeneity in structure,diversity in function // Am. J. Physiol. 1998. Vol. 275. P.
F633-F655.19964. Blanton M.P., Lala A.K., Cohen J.B. Identification and characterization ofmembrane-associated polypeptides in Torpedo nicotinic acetylcholine receptorrich membranes by hydrophobic photolabeling // Biochim. Biophys. Acta. 2001.Vol. 1512. P. 215-224.65. Blaustein M.P. How does pressure overload cause cardiac hypertrophy anddysfunction? High-ouabain affinity cardiac Na+ pumps are crucial // Am.
J.Physiol. Heart. Circ. Physiol. 2017. Vol. 313. № 5. P. H919-H930.66. Blaustein M.P. Livin’ with NCX and lovin’ it: A 45 year romance // Adv. Exp.Med. Biol. 2013. Vol. 961. P. 3–15.67. Blaustein M.P. Physiological effects of endogenous ouabain: control ofintracellular Ca2+ stores and cell responsiveness // Am. J. Physiol. (Cell Physiology33).
1993. Vol. 264. P. C1367- C1387.68. Blaustein M.P. The pump, the exchanger, and the holy spirit: origins and 40-yearevolution of ideas about the ouabain-Na+pump endocrine system // Am J PhysiolCell Physiol. 2018 (Epub 2017 Nov 7). Vol. 314, № 1. P. C3-C26.69. Blaustein M.P., Golovina V.A. Structural complexity and functional diversity ofendoplasmic reticulum Ca2+ stores // Trends Neurosci. 2001. Vol.
24. P. 602–608.70. Blaustein MP, Chen L, Hamlyn JM, Leenen FH, Lingrel JB, Wier WG, Zhang J.Pivotal role of α2 Na+ pumps and their high affinity ouabain binding site incardiovascular health and disease // J Physiol. 2016. Vol. 594, № 21. P. 6079-6103.71. Bodine S.C. Disuse-induced muscle wasting // Int. J. Biochem. Cell. Biol.
2013.Vol 45. P. 2200.72. Boon H., Kostovski E., Pirkmajer S., Song M., Lubarski I., Iversen P.O., HjeltnesN., Wideqren U., Chibalin A.V. Influence of chronic and acute spinal cord injuryon skeletal muscle Na+-K+-ATPase and phospholemman expression in humans //Am. J. Physiol. Endocrinol. Metab. 2012. Vol. 302. P. E864.73. Bossuyt, J., Despa, S., Han, F., Hou, Z., Robia, S.L., Lingrel, J.B.,Bers D.M.Isoform specificity of the Na/K-ATPase association and regulation byphospholemman // J. Biol. Chem. 2009. Vol. 284, P.
26749–26757.20074. Boulter J., Evans K., Goldman G., Martin G., Trecco D., Heinemann S., Patrick J.Isolation of a cDNA clone coding for a possible neural nicotinic acetylcholinereceptor α–subunit // Nature. 1986. Vol. 319. P. 368–374.75. Boyd N.D., Cohen J.B. Desensitization of membrane-bound Torpedo acetylcholinereceptor by amine noncompetitive antagonists and aliphatic alcohols: studies of[3H]acetylcholine binding and 22Na+ ion fluxes // Biochemistry. 1984. Vol. 23.
P.4023-4033.76. Brannigan G, LeBard D.N., Hénin J., Eckenhoff R.G., Klein M.L. Multiple bindingsites for the general anesthetic isoflurane identified in the nicotinic acetylcholinereceptor transmembrane domain // Proc Natl Acad Sci USA. 2010. Vol. 107, № 32.P. 14122-14127.77. Brannigan G., Hénin J., Law R., Eckenhoff R., Klein M.L. Embedded cholesterolin the nicotinic acetylcholine receptor // Proc Natl Acad Sci USA 2008. Vol. 105.P. 14418–14423.78.
Brunzell D.H., Russell D.S., Picciotto M.R. In vivo nicotine treatment regulatesmesocorticolimbic CREB and ERK signaling in C57Bl/6J mice // J. Neurochem.2003. Vol. 84, №6. P. 1431–1441.79. Bryndina I.G., Shalagina M.N., Sekunov A.V., Zefirov A.L., Petrov A.M.Clomipramine counteracts lipid raft disturbance due to short-term muscle disuse //Neurosci Lett. 2018. Vol. 18, №664.
P. 1-6.80. Burger K., Gimpl G., Fahrenholz F. Regulation of receptor function by cholesterol// Cell Mol. 2000. Vol. 57. № 11. P. 1577-1592.81. Butt A.N., Tennant B.P., Gillingwater S.D., Shepherd P.S., Swaminathan R.Binding of ouabain and human ouabainlike substance to different Na +,K+-ATPaseisoforms // Hypertens. Res. 2000. Vol. 23. P. 45-50.82. Caldwell, J. H. Clustering of sodium channels at the neuromuscular junction //Microsc. Res.
Tech. 2000. Vol. 49. P. 84-89.83. Canato M., DalMaschio M., Sbrana F., Raiteri R., Reggiani C., Vassanelli S.,Megighian A. Mechanical and electrophysiological properties of the sarcolemma201of muscle fibers in two Murine models of muscle dystrophy: Col6a1 −/− and mdx //Journal of Biomedicine and Biotechnology. 2010. P. 13.84. Carlson C.G., Roshek D.M. Adult dystrophic (mdx) endplates exhibit reducedquantal size and enhanced quantal variation // Eur.
J. Physiol. 2001. Vol. 442. P.369 – 375.85. Carnio S., LoVerso F., Baraibar M.A., Longa E., Khan M.M., Maffei M., ReischlM., Canepari M., Loefler S., Kern H., Blaauw B., Friguet B., Bottinelli R., RudolfR., Sandri M. Autophagy impairment in muscle induces neuromuscular junctiondegeneration and precocious aging // Cell Rep. 2014. Vol. 8. P. 1509–1521.86.