Диссертация (1174337), страница 29
Текст из файла (страница 29)
// Clin. Exp.Immunol. - 2002. - Vol. 128. - № 1. - P. 36-43.121. Korn, T. Myelin-specific regulatory T cells accumulate in the CNS but failto control autoimmune inflammation / T. Korn, J. Reddy, W. Gao et al. // Nat. Med. 2007. - Vol. 13. - № 4. - P. 423–431.122. Korn, T.IL-17 and Th17 Cells / T. Korn, E. Bettelli, M. Oukka,V.K. Kuchroo // Annu. Rev. Immunol. – 2009. - Vol.
27. - P. 485–517.123. Krebs, C.F. T helper type 17 cells in immune-mediated glomerular disease/ C.F. Krebs, T. Schmidt, J.H. Riedel, U. Panzer // Nat. Rev. Nephrol. - 2017. - Vol. 13.- № 10. - P. 647-659.124. Krieg, A.M. CpG motifs in bacterial DNA and their immune effects/ A.M. Krieg // Annu. Rev. Immunol. - 2002. - Vol. 20. - P.
709-760.125. Krieg, A.M. CpG motifs in bacterial DNA trigger direct B-cell activation/ A.M. Krieg, A.K. Yi, S. Matson et al. // Nature. - 1995. - Vol. 374. - P. 546-549.126. Kronbichler, A. The Influence and Role of Microbial Factors inAutoimmune Kidney Diseases: A Systematic Review / A. Kronbichler, J. Kerschbaum,G.
Mayer // J. Immunol. Res. - 2015; 2015:858027. Published online 2015 May18. doi: 10.1155/2015/858027127. Krug, A. Herpes simplex virus type 1 activates murine natural interferonproducing cells through toll-like receptor 9 / A. Krug, G.D. Luker, W. Barchet et al.// Blood. - 2004. - Vol. 103. - P.
1433-1437.172128. Krutzik S.R. The role of Toll-like receptors in host defense against microbialinfection / S.R. Krutzik, P.A. Sieling, R.L. Modlin //Current Opinion in Immunology2001. – Vol. 13. – P. 104–108.129. Kurts, C. The immune system and kidney disease: Basic concepts andclinical implications / C. Kurts, U.
Panzer, H.J. Anders, A.J. Rees// Nat. Rev.Immunol. - 2013. - Vol. 13. - № 10. - P. 738–753.130. Kwilasz, A.J. The therapeutic potential of interleukin-10 in neuroimmunediseases / A.J. Kwilasz, P.M. Grace, P. Serbedzija et al. // Neuropharmacology. - 2015. Vol. 96 (Pt A). - P. 55-69.131. Lai, K.N. Podocyte injury induced by mesangial-derived cytokines in IgAnephropathy / K.N. Lai, J.C. Leung, L.Y. Chan et al. // Nephrol.
Dial. Transplant. 2009. - Vol. 24. - № 1. - P. 62-72.132. Lan, H.Y. Suppression of experimental crescentic glomerulonephritis by theinterleukin-1 receptor antagonist / H.Y. Lan, D.J. Nicolic-Paterson, M. Zarama et al.// Kidney Int. - 1993. - Vol. 43. - № 2. - P. 479–485.133. Lanier, L.L. Up on the tightrope: Natural killer cell activation and inhibition/ L.L. Lanier // Nat. Immunol. - 2008.
- Vol. 9. - P. 495–502.134. Latz, E. TLR9 signals after translocating from the ER to CpG DNA in thelysosome / E. Latz, A. Schoenemeyer, A. Visintin et al. // Nat. Immunol. - 2004. Vol. 5. - P. 190-198.135. Lau, Y.K. Renin-angiotensin system gene polymorphisms: its impact onIgAN and its progression to end-stage renal failure among Chinese in Singapore/ Y.K. Lau, K.T. Woo, H.L. Choong et al.
// Nephron. Physiol. - 2004. - Vol. 97. P. 1-8.136. Lech, M. The pathogenesis of lupus nephritis / M. Lech, H.J. Anders// J. Am. Soc. Nephrol. - 2013. - Vol. 24. - P. 1357–1366.137. Lichtnekert, J. Anti-GBM glomerulonephritis involves IL-1 but isindependent of NLRP3/ASC inflammasome-mediated activation of caspase-1/ J. Lichtnekert, O.P. Kulkarni, S.R.
Mulay et al. // PLoS One. - 2011. - Vol. 6. - № 10.- e.26778.173138. Lichtnekert, J. Trif is not required for immune complex glomerulonephritis:dying cells activate mesangial cells via Tlr2/Myd88 rather than Tlr3/Trif/ J. Lichtnekert, V. Vielhauer, D. Zecher et al.// American Journal of Physiology:Renal Physiology. - 2009. - Vol. 296. - P. 867–874.139.
Liu, Q. The molecular mechanisms of TLR-signaling cooperation incytokine regulation. / Q. Liu, J.L. Ding // Cell. Biol. - 2016. - Vol. 94. - № 6. - P. 538542.140. Lorenz, G. Neutrophils, dendritic cells, toll-like receptors, and interferonalpha in lupus nephritis / G. Lorenz, H.J. Anders // Semin. Nephrol. -2015. - Vol. 35. P. 410–426.141. Łukawska, E. The role of the alternative pathway of complement activationin glomerular diseases / E. Łukawska, M. Polcyn-Adamczak, Z.I. Niemir // Clinical andExperimental Medicine. - 2018.
- Vol. 18. - № 3. - P. 297-318.142. Luo, C. Long-term prognosis for Chinese adult patients with acutepostinfectious glomerulonephritis / C. Luo, Z. Tang, D. Chen et al. // Clin. Nephrol. 2011. - Vol. 76. - P. 186–194.143. Maillard, N. Current understanding of the role of complement in IgAnephropathy / N. Maillard, R.J.
Wyatt, B.A. Julian et al. // J. Am. Soc. Nephrol. - 2015.- Vol. 26. – Р. 1503–1512.144. Malafronte, P. Paulista Registry of glomerulonephritis: 5-year data report/ P. Malafronte, G. Mastroianni-Kirsztajn, G.N. Betônico et al. // Nephrol. Dial.Transplant.
- 2006. - Vol. 21. - P. 3098-3105.145. Martinon, F. The inflammasome: a molecule platform triggering activationof inflammatory caspases and processing of proIL-1β /F. Martinon, K. Burns,J. Tschopp //Mol. Cell. – 2002. - Vol. 10. – P. 417-426.146. Maruyama, K. Polymorphisms of renin-angiotensin system genes inchildhood IgA nephropathy / K. Maruyama, M.
Yoshida, H. Nishio et al. // Pediatr.Nephrol. – 2001. - Vol. 16. - P. 350-355.174147. Meng, X.M. Inflammatory processes in renal fibrosis / X.M. Meng,D.J. Nikolic-Paterson, H.Y. Lan // Nat. Rev. Nephrol. - 2014. - Vol. 10. - № 9. P. 493–503.148. Mills, J.A. Systemic lupus erythematosus / J.A. Mills // N. Engl. J. Med. 1994. - Vol.
330. - P. 1871–1879.149. Montseny,J.J. The current spectrum of infectious glomerulonephritis.Experience with 76 patients and review of the literature / J.J. Montseny, A. Meyrier,D. Kleinknecht et al. // Medicine (Baltimore). - 1995. - Vol. 74. - P. 63–73.150. Mori, H. Monocyte chemoattractant protein-1 A-2518G gene polymorphismand renal survival of Japanese patients with immunoglobulin A nephropathy / H.
Mori,Y. Kaneko, I. Narita et al. // Clin. Exp. Nephrol. - 2005. - Vol. 9. - № 4. - P. 297-303.151. Moroni, G. Long-term prognosis of diffuse proliferative glomerulonephritisassociated with infection in adults / G. Moroni, C. Pozzi, S. Quaglini et al. // Nephrol.Dial. Transplant. - 2002. - Vol. 17.
- P. 1204–1211.152. Moroni, G. Rapidly progressive crescentic glomerulonephritis: Earlytreatment is a must / G. Moroni, C. Ponticelli // Autoimmun. Rev. - 2014. - Vol. 13. № 7. - P. 723-729.153. Murali-Krishna K. Counting antigen-specific CD8 T cells: a reevaluation ofbystander activation during viral infection / K. Murali-Krishna, J.D. Altman, M. Sureshet al. // Immunity.
- 1998. - Vol. 8. - № 2. - P. 177-187.154. Naciute, M. Increased Numbers of CD4+CD25+ and CD8+CD25+ T-Cellsin Peripheral Blood of Patients with Rheumatoid Arthritis with Parvovirus B19Infection / M. Naciute, G. Maciunaite, D. Mieliauskaite et al. // In Vivo. - 2017. Vol. 31. - № 2. - P. 181-185.155. Nasr, S.H. Acute postinfectious glomerulonephritis in the modern era:experience with 86 adults and review of the literature / S.H. Nasr, G.S. Markowitz,M.B. Stokes et al.
// Medicine (Baltimore). - 2008. - Vol. 87. - P. 21–32.156. Nasr, S.H. Bacterial infection-related glomerulonephritis in adults/ S.H. Nasr, J. Radhakrishnan, V.D. D'Agati // Kidney Int. – 2013. – Vol. 83. - № 5. –P. 792-803.175157. Nasr, S.H. IgA-dominant postinfectious glomerulonephritis: a new twist onan old disease / S.H. Nasr, V.D.
D’Agati // Nephron. Clin. Pract. - 2011. - Vol. 119. P. 18–25.158. Nawijn, M.C. TLR-2 activation induces regulatory T cells and long-termsuppression of asthma manifestations in mice / M.C. Nawijn, A.C. Motta, R. Gras et al.// PLoS One. - 2013. - Vol. 8. - № 2. – e.55307.159. Nayer, A. IgA-dominant post-infectious glomerulonephritis; making anothercase in support of renal biopsy in type 2 diabetic nephropathy / A.
Nayer, G. Davda,R. Pai, L.M.Ortega // J. Renal Inj. Prev. - 2016. - Vol. 5. - № 1. - P. 45-47.160. Nguyen, M.T. Lipoproteins of Gram-Positive Bacteria: Key Players in theImmune Response and Virulence / M.T. Nguyen, F. Götz // Microbiol. Mol. Biol. Rev.- 2016.
- Vol. 80. - № 3. - P. 891-903.161. Noris, M. Genetics of Immune-Mediated Glomerular Diseases: Focus onComplement / M. Noris, G. Remuzzi // Semin. Nephrol. - 2017. - Vol. 37. - № 5. P. 447-463.162. Noronha, I.L. Cytokines and growth factors in renal disease / I.L. Noronha,Z. Niemir, H. Stein, R.
Waldherr // Nephrol. Dial. Transplant. - 1995. - Vol. 10. - № 6. P. 775–786.163. Novak, J. Glycosylation of IgA1 and pathogenesis of IgA nephropathy/ J. Novak, B.A. Julian, J. Mestecky, M.B. Renfrow // Semin. Immunopathol. - 2012. Vol. 34. - № 3. - P. 365-382.164. Ohyama, K. Proteomic profiling of antigens in circulating immunecomplexes associated with each of seven autoimmune diseases / K. Ohyama, M. Baba,M. Tamai et al.
// Clin Biochem. - 2015. - Vol. 48. - № 3. - P. 181-185.165. Okamoto, A. Kidney-infiltrating CD4+ T-cell clones promote nephritis inlupus-prone mice / A. Okamoto, K. Fujio, N.H. Tsuno et al. // Kidney Int. - 2012. Vol. 82. - № 9. - P. 969-979.166. Omokawa, A. Distribution of glomerular IgG subclass deposits in patientswith membranous nephropathy and anti-U1 ribonucleoprotein antibody / A. Omokawa,176A. Komatsuda, M. Nara et al. // Nephrol.
Dial. Transplant. - 2012. - Vol. 27. - № 5. –Р.1937-1941.167. Ortega, L.M. Role of cytokines in the pathogenesis of acute and chronickidney disease, glomerulonephritis, and end-stage kidney disease / L.M. Ortega,A. Fornoni // Int. J. Interferon Cytokine Mediator Res. - 2010. - Vol. 2. – Р. 49-62.168.
Ostmann, A. Regulatory T cell-derived IL-10 ameliorates crescentic GN/ A. Ostmann, H.J. Paust, U. Panzer et al. // J. Am. Soc. Nephrol. - 2013. - Vol. 24. №6. - P. 930-942.169. Ozinsky, A. The repertoire for pattern recognition of pathogens by theinnate immune system is defined by cooperation between toll-like receptors/ A. Ozinsky, D.M.