Диссертация (1174337), страница 30
Текст из файла (страница 30)
Underhill, J.D. Fontenot et al. // Proc. Natl. Acad. Sci. USA. 2000. - Vol. 97. - P. 13766-13771.170. Pan, Q. Role of basophils in the pathogenesis of minimal change nephroticsyndrome: a literature review / Q. Pan, J. Wu, J.Tao et al. // Exp. Ther. Med. - 2014. Vol. 8. - № 4. - P. 1027–1031.171. Patole, P.S. Expression and regulation of Toll-like receptors in lupus-likeimmune complex glomerulonephritis of MRL-Fas(lpr) mice / P.S. Patole,R.D. Pawar, M.
Lech et al. // Nephrol. Dial. Transplant. - 2006. - Vol. 21. - № 11. P. 3062–3073.172. Patole, P.S. Viral double-stranded RNA aggravates lupus nephritis throughToll-like receptor 3 on glomerular mesangial cells and antigen-presenting cells/ P.S. Patole, H.J. Gröne, S. Segerer et al.
// J. Am. Soc. Nephrol. - 2005. - Vol. 16. P. 1326-1338.173. Pawar,R.D.Toll-likereceptor-7modulatesimmunecomplex glomerulonephritis / R.D. Pawar, P.S. Patole, D. Zecher et al. // J. Am. Soc.Nephrol. - 2006. - Vol. 17. - № 1. - P. 141-149.174. Pedigo, C.E. Local TNF causes NFATc1-dependent cholesterol-mediatedpodocyte injury / C.E. Pedigo, G.M. Ducasa, F. Leclercq et al. // J. Clin. Invest. - 2016.- Vol.
126. - № 9. - P. 3336-3350.177175. Pei, Y. Association of angiotensinogen gene T235 variant with progressionof immunoglobin A nephropathy in Caucasian patients / Y. Pei, J. Scholey, K. Thai etal. // J. Clin. Invest. – 1997. - Vol. 100. - P. 814-820.176. Pereira, Wde F.
The role of the immune system in idiopathic nephroticsyndrome: a review of clinical and experimental studies / Wde F. Pereira, G.E. BritoMelo, F.T. Guimarгes et al. // Inflamm. Res. - 2014. - Vol. 63. - № 1. - P. 1–12.177. Peters, H. Angiotensin II blockade and low-protein diet produce additivetherapeutic effects in experimental glomerulonephritis / H. Peters, W.A. Border,N.A. Noble // Kidney Int.
- 2000. - Vol. 57. - № 4. - P. 1493-1501.178. Phoon, R.K. T-bet deficiency attenuates renal injury in experimentalcrescentic glomerulonephritis / R.K. Phoon, A.R. Kitching, D. Odobasic et al. // J. Am.Soc. Nephrol. - 2008. - Vol. 19. - P.
477–485.179. Pisetsky, D.S. Systemic lupus erythematosus: a matter of life and death/ D.S. Pisetsky, L. Rönnblom // Arthritis Rheum. - 2009. - Vol. 60. - P. 1567–1570.180. Placier, S. Renal hemodynamics and development of renal fibrotic lesionsduring hypertension / S. Placier, M.
Flamant, J.J. Boffa et al. //Arch. Mal. Coeur. Vaiss.- 2006. - Vol. 99. - № 7-8. - P. 697-700.181. Polhill, T. IL-2/IL-2Ab complexes induce regulatory T cell expansion andprotect against proteinuric CKD / T. Polhill, , G.Y. Zhang, M. Hu et al. // J. Am. Soc.Nephrol. - 2012. - Vol. 23. - № 8. - P. 1303-1308.182. Prakash, S. Prediction of susceptible biomarkers for end stage renal diseaseamong North Indians / S. Prakash, A.N.
Sarangi, G. Tripathi et al. // Nephrology(Carlton). - 2016. - Vol. 21. - № 7. - P. 592-600.183. Prasad, N.Infection-Induced Kidney Diseases / N. Prasad, M.R. Patel// Front. Med. (Lausanne). - 2018. - Vol. 5. - P. 327.184. Remuzzi, A. Effect of angiotensin II antagonism on the regression of kidneydisease in the rat / A. Remuzzi, E. Gagliardini, C. Donadoni et al. // Kidney Int. - 2002.- Vol. 62. - № 3. - P. 885-894.185. Ricardo, S.D. Macrophage diversity in renal injury and repair / S.D.
Ricardo,G.H. Van, A.A. Eddy // J. Clin. Invest. - 2008. - Vol. 118. - P. 3522–3530.178186. Rijnink, E.C. Idiopathic non-lupus full-house nephropathy is associated withpoor renal outcome / E.C. Rijnink, Y.K. Teng, T. Kraaij et al.// Nephrol. Dial.Transplant. - 2017.
- Vol. 32. - № 4- P. 654–662.187. Ring, T. Use of eculizumab in crescentic IgA nephropathy: proof ofprinciple and conundrum? / T. Ring, B.B. Pedersen, G. Salkus et al. // Clin. Kidney J. 2015. - Vol. 8. – Р. 489–491.188. Ritz,E.Effectofrenovascularhypertensiononexperimentalglomerulonephritis in rats / E.
Ritz, S. Orth, T. Weinreich, J. Wagner // J. Lab. Clin.Med. - 1999. - Vol. 134. - № 3. - P. 292-303.189. Rodriguez-Iturbe, B. Attack rate of poststreptococcal nephritis in families.A prospective study / B. Rodriguez-Iturbe, L. Rubio, R. Garcia // Lancet. - 1981. Vol. 1. - Р. 401–403.190. Rosenblad, T. Eculizumab treatment for rescue of renal function in IgAnephropathy / T. Rosenblad, J. Rebetz, M. Johansson et al. // Pediatr. Nephrol. - 2014. Vol.
29. – Р. 2225–2228.191. Rutishauser, R.L. Generating diversity: transcriptional regulation of effectorand memory CD8 T-cell differentiation / R.L. Rutishauser, S.M. Kaech // Immunol.Rev. - 2010. - Vol. 235. - № 1. - P. 219-233.192. Salant,D.J. Membranousnephropathy.Chapter20/D.J.Salant,D.C. Cattran // In: Comprehensive Clinical Nephrology, 5th Ed., edited by Floege J.,Johnson R.J., Feehally J. - St. Louis, MI, Saunders, an imprint of Elsevier Inc., 2015. –Р. 239–251.193. Satoskar,A.A.Acutepostinfectiousglomerulonephritisandglomerulonephritis caused by persistent bacterial infection / Satoskar A.A., Nadasdy T.,Silva F.G.In: Jennette J.C., Olson J.L., Silva F.G., D’Agati V.D., eds.
Heptinstall’sPathology of the Kidney. 7th Ed. Philadelphia, PA: Wolters Kluwer, 2015. - P. 367436.194. Schena, F.P. Progression of renal damage in human glomerulonephritis: Isthere sleight of hand in winning the game? / F.P. Schena, L. Gesualdo, G. Grandaliano,V. Montinaro // Kidney Int. - 1997. - Vol. 52. - P. 1439–1457.179195. Sean, E.K. Macrophages and progressive tubulointerstitial disease/ E.K. Sean, P. Cockwell // Kidney Int. - 2005. - Vol. 68. - P. 437–455.196. Sekine, H. Enrichment of anti-glomerular antigen antibody-producing cellsin the kidneys of MRL/MpJ-Fas(lpr) mice / H.
Sekine, H. Watanabe, G. S. Gilkeson// J. Immunol. - 2004. - Vol. 172. - P. 3913–3921.197. Sethi, S. C3 Glomerulonephritis: clinicopathologic findings, complementabnormalities, glomerular proteomic profile, treatment and follow-up / S. Sethi,F.C. Fervenza., Y. Zhang et al. // Kidney Int. – 2012. – Vol. 82. – P. 465-473.198. Sethi, S. Mayo Clinic/Renal Pathology Society Consensus Report onPathologic Classification, Diagnosis, and Reporting of GN / S.
Sethi, M. Haas,G.S. Markowitz et al. // J. Am. Soc. Nephrol. - 2015. - Vol. 27. - № 5. - P. 1278-1287.199. Sethi, S. Standardized classification and reporting of glomerulonephritis/ S. Sethi, F.C. Fervenza // Nephrol. Dial. Transplant. - 2018. doi: 10.1093/ndt/gfy220.[Epub ahead of print]200. Shahzad, K. Nlrp3-inflammasome activation in non-myeloid-derived cellsaggravates diabetic nephropathy / K. Shahzad, F. Bock, W. Dong et al. // Kidney Int.2015. - Vol. 87. - № 1. - P. 74-84.201.
Shao, X.S. The prevalence of Th17 cells and FOXP3 regulate T cells (Treg)in children with primary nephrotic syndrome / X.S. Shao, X.Q. Yang, X.D. Zhao et al.// Pediatr. Nephrol. - 2009. - Vol. 24. - № 9. – P. 1683–1690.202. Simmons, E.M. Plasma cytokine levels predict mortality in patients withacute renal failure / E.M. Simmons, J. Himmelfarb, M.T. Sezer et al. // Kidney Int.
2004. - Vol. 65. - № 4. - P. 1357-1365.203. Shin, J.I. Inverse relationship between soluble urokinase receptors andestimated glomerular filtration rate: a role for IL-2? / J.I. Shin // Kidney Int. - 2015. Vol. 87. - № 5. - P. 1074.204. Shishido, S. Combination of pulse methylprednisolone infusions withcyclosporine-based immunosuppression is safe and effective to treat recurrent focalsegmental glomerulosclerosis after pediatric kidney transplantation / S. Shishido,180H.
Satou, M. Muramatsu et al. // Clin. Transplant. - 2013. - Vol.27. - № 2. -P. 143–150.205. Smeltz, R.B. Inhibition of autoimmune T cell responses in the DA rat bybone marrow-derived NK cells in vitro: Implications for autoimmunity / R.B. Smeltz,N.A. Wolf, R.H. Swanborg // J. Immunol. - 1999. - Vol. 163. - P. 1390–1397.206. Solak, Y. Glomerulonephritis associated with tuberculosis: a case report andliterature review / Y.
![](https://s.studizba.com/z.php?f=/templates/si/i/noavatar.png&w=100&h=100&t=1"){kind=avatar}
