Диссертация (1141136), страница 18
Текст из файла (страница 18)
- 2001. – Vol. 12. – P. 61–65.103. Rudy Y. and Plonsey R. A note on the Brody-Effect. // J Electrocardiology. –1978. – Vol. 11. – P. 87-90.104. RudyY.,Messinger-RapportB.J.,TheInverseProbleminElectrocardiography: Solutions in terms of Epicardial Potentials. // CRC CritRev Bioeng. – 1988.
- Vol. 16. – P. 215-268.105. Sacher F, Probst V, Iesaka Y, et al. Outcome after implantation of acardioverter–defibrillator inpatients with Brugada syndrome: a multicenterstudy. // Circulation. – 2006. – Vol. 114. – P. 2317–2324.106. Shimizu W, Matsuo K, Takagi M., et al. Body surface distribution andresponse to drugs of ST segment elevation in Brugada syndrome: clinicalimplication of eighty-seven-lead body surface potential mapping and itsapplication to twelve-lead electrocardiograms.
// J Cardiovasc Electrophysiol.– 2000. – Vol. 11(4). – P. 396-404.107. Sinner MF, Pfeufer A, Perz S., et al. Spontaneous Brugada electrocardiogrampatterns are rare in the German general population: results from the KORAstudy. // Europace.
– 2009. – Vol. 11. – P. 1338–1344.108. Smits JP, Eckardt L, Probst V, et al. Genotype–phenotype relationship inBrugada syndrome: electrocardiographic features differentiate SCN5Arelated patients from non-SCN5A-related patients. // J Am Coll Cardiol. 2002. – Vol. 40. – P. 350–356.109. Splawski I, Timothy KW, Decher N, et al. Severe arrhythmia disorder causedby cardiac L-type calcium channel mutations. // Proc Nat Acad Sci USA. 2005. – Vol. 102.
– P. 8089–8096.110. Takimoto K, Li D, Nerbonne JM, Levitan ES. Distribution, splicing andglucocorticoid-induced expression of cardiac alpha 1C and alpha 1D voltage133gated Ca21 channel mRNAs. // J Mol Cell Cardiol. - 1997. – Vol. 29. -P.3035–3042.111. Tsuchiya T1, Ashikaga K, Honda T, et al.Prevention of ventricularfibrillation by cilostazol, an oral phosphodiesterase inhibitor, in a patient withBrugada syndrome. // J Cardiovasc Electrophysiol. - 2002. – Vol.
13(7). P.698-701.112. Tukkie R, Sogaard P, Vleugels J, et al. Delay in right ventricular activationcontributes to Brugada syndrome. // Circulation. – 2004. – Vol. 109. – P.1272–1277.113. Van PF, Clark KA, Chatelain FC, Minor Jr DL. Structure of a complexbetween a voltage-gated calcium channel beta-subunit and an alpha-subunitdomain. // Nature.
- 2004. – Vol. 429. – P. 671–675.114. Van den Berg MP, Wilde AA, Viersma TJW, et al. Possible bradycardic modeof death and successful pacemaker treatment in a large family with features oflong QT syndrome type 3 and Brugada syndrome. // J CardiovascElectrophysiol. – 2001. - Vol. 12 – P. 630-636.115. Veltmann C, Schimpf R, Echternach C, A prospective study on spontaneousfluctuations between diagnostic and non-diagnostic ECGs in Brugadasyndrome: implications for correct phenotyping and risk stratification. //European Heart Journal. - 2006. Vol.
27. – P. 2544–2552.116. Vernooy K, Delhaas T, Cremer OL, Di Diego JM, Oliva A, Timmermans C,et al. Electrocardiographic changes predicting sudden death in propofolrelated infusion syndrome. // Heart Rhythm. – 2006. – Vol. 3. – P. 131–137.117. Vernooy K, Sicouri S, Dumaine R, Hong K, Oliva A, Burashnikov E, et al.Genetic and biophysical basis for bupivacaine-induced ST segment elevationand VT/VF. Anesthesia unmasked Brugada syndrome.
// Heart Rhythm. –2006. – Vol. 3. – P. 1079–1781.118. Virk HUH, Farooq S, Ghani AR, Arora S. QRS fragmentation: its role insherlocking the arrhythmogenic heart. // Journal of Community HospitalInternal Medicine Perspectives. – 2016. – Vol. 6(3). – P. 1230-1235. -134119. Wilde AA, Antzelevitch C, Borggrefe M, et al. Proposed Diagnostic Criteriafor the Brugada Syndrome: Consensus Report. // Circulation. – 2002. – Vol.106. – P.
2514-2519.120. Wilde AA, Viskin S. EP testing does not predict cardiac events in Brugadasyndrome. // Heart Rhythm. – 2011. - Vol. 8. – P.1598 –1600.121. Wolpert C, Echternach C, Veltmann C, et al. Intravenous drug challenge usingflecainide and ajmaline in patients with Brugada syndrome. // Heart Rhythm.– 2005. – Vol. 2. – P. 254–260.122. Yan GX, Antzelevitch C. Cellular Basis for the Brugada Syndrome and OtherMechanisms of Arrhythmogenesis Associated With ST-Segment Elevation. //Circulation.
– 1999. – Vol. 100. – P. 1660-1666.123. Yan GX, Antzelevitch C. Cellular basis for the electrocardiographic J wave.// Circulation. – 1996. – Vol. 93. – P. 372–379.124. Zhang J, Sacher F, Hoffmayer K, et al. Cardiac Electrophysiologic SubstrateUnderlying the ECG Phenotype and Electrogram Abnormalities in BrugadaSyndrome Patients.
// Circulation. 2015. – Vol. 131. – P. 1950-1959.125. Zipes DP, Camm JA, Borggrefe M. ACC/AHA/ESC 2006 guidelines formanagement of patients with ventricular arrhythmias and the prevention ofsudden cardiac death—executive summary. // European Heart Journal. –2006. – Vol. 27. – P. 2099–2140..
![](https://s.studizba.com/z.php?f=/templates/si/i/noavatar.png&w=100&h=100&t=1"){kind=avatar}
