Диссертация (1140189), страница 28
Текст из файла (страница 28)
Human gastric carcinogenesis: a multistep and multifactorial process — First American Cancer Society Award Lecture on Cancer Epidemiology and Prevention / P. Correa // Cancer Res. — 1992. — Vol. 52. —Р. 6735—6740.101. Current concepts in the management of Helicobacter pylori infection: theMaastricht III Consensus Report / P. Malfertheiner, F. Megraud, C. O’Morain[et al.] // Gut.
— 2007. — Vol. 56. — Р. 772—781.102. Defining epithelial cell progenitors in the human oxyntic mucosa / S. M. Karam, T. Straiton, W. M. Hassan, C. P. Leblond // Stem. Cells. — 2003. —Vol. 21. — Р. 322—336.103. Dekigai, H. Mechanism of Helicobacter pylori-associated gastric mucosal injury / H. Dekigai, M. Murakami, T. Kita // Dig Dis Sei. — 1995. — Jun.,Vol. 40 (6).
— P. 1332—1339.104. Delineation of the chemomechanosensory regulation of gastrin secretion usingpure rodent G cells / M. Kidd, O. Hauso, I. Drozdov [et al.] // Gastroenterology. — 2009. — Vol. 137. — Р. 231—241.105. Diagnosis of gastritis by means of a combination of serological analyses /E. Mardh, S. Mardh, B. Mardh, K. Borch // Clin.
Chim. Acta. — 2002. —Vol. 320. — Р. 17—27.106. Ding, S. Z. Helicobacter pylori infection, oncogenic pathways and epigeneticmechanisms in gastric carcinogenesis / S. Z. Ding, J. B. Goldberg, M. Hatakeyama // Future Oncol. — 2010. — Vol. 6. — Р. 851—862.177107. Ding, S. Z. Helicobacter pylori infection induced gastric cancer; advance ingastric stem cell research and the remaining challenges / S. Z. Ding,P. Y. Zheng // Gut. Pathog. — 2012.
— Vol. 4. — Р. 18.108. Discrimination of normal gastric mucosa from Helicobacter pylori gastritis using standard endoscopes and a single observation site: studies in childrenand young adults / Y. Nakayama, A. Horiuchi, T. Kumagai [et al.] // Helicobacter. — 2004. — № 9. — P. 95—99.109. Dockray G. J. Gastrin and gastric epithelial physiology / G. J.
Dockray //J. Physiol. — 1999. — Vol. 518. — Р. 315—324.110. Dunn, B. E. Role of bacterial amines in H. pylori-associated hypergastrinaemia / B. E. Dunn // Helicobacter pylori. Springer Netherlands. —1994. — Р. 273—280.111. E-cadherin gene mutations provide a genetic basis for the phenotypic divergence of mixed gastric carcinomas / J. C. Machado, P. Soares, F.
Carneiro[et al.] // Lab. Invest. — 1999. — Vol. 79, № 4. — P. 459—465.112. Effect of Helicobacter pylori eradication on G-cell and D-cell density in children / D. M. Queiroz, S. B. Moura, E. N. Mendes [et al.] // Lancet. — 1994. —Vol. 14. — Р. 1191—1193.113. Effect of Helicobacter pylori infection on antral gastrin and somatostatin cellsand on serum gastrin concentrations / S. M. Park, H. R.
Lee, J. G. Kim[et al.] // Korean. J. Intern. Med.—1999. — Vol. 14. — Р. 15—20.114. Effect of Helicobacter pylori products and recombinant cytokines on gastrinrelease from cultured canine G cells / I. Beales, M. J. Blaser, S. Srinivasan[et al.] // Gastroenterology. — 1997. — Vol. 113. — Р. 465—471.115. Effects of Helicobacter Pylori gastritis on gastric secretion in healthy humanbeings / M.
Feldman, B. Cryer, E. Lee // Am. J. Physiol. — 1998. — Vol. 274(6). — Р. 1011—1017.116. Ekundayo, A. A. Gastrin and the growth of the gastrointestinal tract / A. A.Ekundayo, C. Y. Lee, R. A. Goodlad // Gut. — 1995. — Vol. 36, № 2. —Р. 203—208.178117. Epstein — Barr virus and Helicobacter pylori co-infection are positively associated with severe gastritis in pediatric patients / M. G. Cárdenas-Mondragón,R.
Carreon-Talavera, M. Camorlinga-Ponce [et al.] // PLoS One. — 2013. —Vol. 8, № 4. — Р. 62850.118. Epstein — Barr virus infection is common in inflamed gastrointestinal mucosa/ J. L. Ryan, Y. J. Shen, D. R. Morgan [et al.] // Dig. Dis. Sci. — 2012. —Vol. 57. — Р. 1887—1898.119. Eradication of Helicobacter pylori for primary gastric cancer and secondarygastric cancer after endoscopic mucosal resection / S. Kato, M. Asaka, S. Ono[et al.] // J. Gastroenterol.
— 2007. — Vol. 42, № 17. — Р. 16—20.120. Eradication of Helicobacter pylori prevents cancer development in subjectswith mild gastric atrophy identified by serum pepsinogen levels /K. Yananoka, M. Oka, H. Ohata [et al.] // Int. J. Cancer. — 2009. —Vol. 125. — Р.
2697—703.121. Eurohepygast Study Group Pepsinogen A, pepsinogen C, and gastrin as markers of atrophic chronic gastritis in European dyspeptics / N. Broutet, M.Plebani, C. Sakarovitch [et al.] // Br. J. Cancer. — 2003. — Vol. 88. —Р. 1239—1247.122. Evaluation of gastric histology in children and adolescents with Helicobacterpylori gastritis using the Update Sydney System / M. Langner, R.
S. Machado,F. R. S. Patrício, E. Kawakami // Arquivos de Gastroenterologia Gastroenterol. — 2009. — Vol. 46, № 4.123. Evidence that loss of sonic hedgehog is an indicator of Helicobacterpyloriinduced atrophic gastritis progressing to gastric cancer / A. Shiotani,H. Iishi, N. Uedo [et al.] // Am. J.
Gastroenterol. — 2005. — Vol. 100. —Р. 581—587.124. Expression and cell-specific localization of the cholecystokinin B/gastrin receptor in the human stomach / H. Kulaksiz, R. Arnold, B. Goke [et al.] // Cell.Tissue. Res. — 2000. — Vol. 299. — Р. 289—298.179125. Expression of antral gastrin and somatostatin mRNAA in Helicobacter pyloriinfected subjects / M.
Sumii, K. Summi, A. Tari [et al.] // Am. J. Gastroenterоl. — 1994. — Vol. 89. — Р. 1515—1519.126. Expression of homeobox gene CDX2 precedes that of CDX1 during the progression of intestinal metaplasia / A. Eda, H. Osawa, I. Yanaka [et al.] //J. Gastroenterol. — 2002. — Vol. 37. — Р. 94—100.127. Factors predicting progression of gastric intestinal metaplasia: results of a randomised trial on Helicobacter pylori eradication / W. K. Leung, S. R. Lin,J. Y. L.
Ching [et al.] // Gut. — 2004. — Vol. 53, №. 9. — Р. 1244—1249.128. Faller, G. Helicobacter pylori and antigastric autoimmunity / G. Faller,T. Kirchner // Pathologe. — 2001. — Jan., № 22 (1). — P. 25—30.129. Fasting levels of serum gastrin in different functional and morphologic states ofthe antrofundal mucosa. An analysis of 860 subjects / P. Sipponen, J. Valle,K.
Varis [et al.] // Scand. J. Gastroenterol. — 1990. — Vol. 25. — Р. 513—519.130. Feldman, M. Effects of Helicobacter pylori gastritis on gastric secretion inhealthy human beings / M. Feldman, B. Cryer, E. Lee // Am. J. Physiol.Gastrointest. Liver. Physiol.
— 1998. — Vol. 274 (6 Pt 1). — Р. 1011—1017.131. Fichman, S. Histological changes in the gastric mucosa after Helicobacter pylori eradication / S. Fichman, Y. Niv // Eur. J. Gastroenterol. Hepatol. —2004. — Vol. 16, № 11. — P. 1183—1188.132. Fox, J. G. Inflammation, atrophy, and gastric cancer / J. G. Fox, T. S. Wang //J. Clin. Invest. — 2007. — Vol. 117. — Р. 60—69.133. Functional role of calcium-sensing receptor on somatostatin release from ratgastric mucosa (Abstract) / E. Nakamura, M. Hasumura, A. S. Gabriel [et al.] //Gastroenterology. — 2010.
— Vol. 138. — Р. 404.134. Gastric acid secretion in cholecystokinin-1 receptor, -2 receptor, and -1, -2 receptor gene knockout mice / S. Kanai, H. Hosoya, S. Akimoto [et al.] //J. Physiol. Sci. — 2009. — Vol. 59. — Р. 23—29.180135. Gastric atrophy and intestinal metaplasia before and after Helicobacter pylorieradication: a meta-analysis / J. Wang, L. Xu, R. Shi [et al.] // Digestion. —2011.
— Vol. 83. — Р. 253—260.136. Gastric cancer risk in patients with premalignant gastric lesions: a nationwidecohort study in the Netherlands / A. C. de Vries, N. C. van Grieken, C. V.Looman [et al.] // Gastroenterology. — 2008. — Vol. 134. — Р. 945—952.137. Gastric endocrine cells: types, function and growth / E. Solcia, G.
Rindi,R. Buffa [et al.] // Regul. Pept. — 2000. — Vol. 93. — Р. 31—35.138. Gastric mucosal atrophy: interobserver consistency using new criteria for classification and grading / M. Rugge, P. Correa, M. F.Dixon [et al.] // AlimentaryPharmacology & Therapeutics. — 2002. — Vol. 16, №. 7. — Р. 1249—1259.139.
Gastric parietal cell antibodies, Helicobacter pylori infection, and chronicatrophic gastritis: evidence from a large population-based study in Germany /Y. Zhang, M. N. Weck, B. Schöttker [et al.] // Cancer Epidemiology Biomarkers & Prevention. — 2013. — Vol. 22, № 5. — Р. 821—826.140. Gastric precancerous process in a high risk population: cross-sectional studies /P. Correa, W. Haenszel, C. Cuello [et al.] // Cancer. Res. — 1990.
—Vol. 50. — Р. 4731—4736.141. Gastrin (G) cells and somatostatin (D) cells in patients with dyspeptic symptoms: Helicobacter pylori associated and non-associated gastritis / Y. Liu,G. D. Vosmaer, G. N. Tytgat [et al.] // J. Clin. Pathol. — 2005. — Vol. 58. —Р. 927—931.142. Gastrin and antral G cells in course of Helicobacter pylori eradication: sixmonths follow up study / A. Sokic-Milutinovic, V.
Todorovic, T. Milosavljevic [et al.] // World J. of Gastroenterology. — 2005. — Vol. 11,№ 27. — Р. 4140.143. Gastrin and somatostatinn in Helicobacter pylori infected antral mucosa /L. Odum, H. D. Petersen, I. B. Andersen [et al.] // Gut. — 1994. — Vol. 35. —Р. 615—618.181144. Gastrin has a specific proliferative effect on the rat enterochromaffin-like cell,but not on the parietal cell: a study by elutriation centrifugation / I.
Bakke,G. Qvigstad, E. Brenna [et al.] // Acta Physiol. Scand. — 2000. —Vol. 169. — Р. 29—37.145. Gastrin stimulates the growth of gastric pit cell precursors by inducing its ownreceptors / T. Nakajima, Y. Konda, Y. Izumi [et al.] // Am. J. Physiol.Gastrointest. Liver Physiol. — 2002.
![](https://s.studizba.com/z.php?f=/templates/si/i/noavatar.png&w=100&h=100&t=1"){kind=avatar}
