Диссертация (1144759), страница 40
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Sharma G., Vijayaraghavan S. Nicotinic cholinergic signaling in hippocampalastrocytes involves calcium-induced calcium release from intracellular stores //Proc. Natl. Acad. Sci. 2001. Vol. 98. P. 4148–4153.353. Shenkman B. S., Nemirovskaya T. L. Calcium-dependent signaling mechanismsand soleus fiber remodeling under gravitational unloading // J. Muscle Res. CellMotil. 2008.
Vol. 29, № 6–8. P. 221–230.354. Shenkman B.S., Litvinova K.S., Nemirovskaya T.L., Podlubnaya Z.A.,Vikhlyantsev I.M., Kozlovskaya I.B. Afferent and peripheral control of musclefiber properties during gravitational unloading // Journ. Gravit. Physiol. 2004. Vol.11. P. 111–114.355. Shenkman B.S., Nemirovskaya T.L., Lomonosova Y.N. No-dependent signalingpathways in unloaded skeletal muscle // Front.
Physiol. 2015. Vol. 6. P. 1-8.356. Shestopalov V.I., Panchin Y., Tarasova O.S., Gaynullina D., Kovalzon V.M.Pannexins Are Potential New Players in the Regulation of Cerebral Homeostasisduring Sleep-Wake Cycle. // Front. Cell Neurosci. 2017. Vol. 11. P. 210.357. Sibarov D.A., Bolshakov A.E., Abushik P.A., Krivoi I.I., Antonov S.M. The Na +,K+-ATPase functionally interacts with the plasma membrane Na+, Ca2+-exchangerto prevent Ca2+ overload and neuronal apoptosis in excitotoxic stress // J.Pharmacol. Exp. Ther. 2012.
Vol. 343. P. 596–607.358. Silverman BdZ, Fuller W., Eaton P., Deng J., Moorman J.R., Cheung J.Y., JamesA.F., Shattock M.J. Serine 68 phosphorylation of phospholemman: acute isoformspecific activation of cardiac Na/K ATPase // Cardiovasc. Res. 2005. Vol.
65. P.93–103.359. Simasko S.M., Soares J.R., Weiland G.A. Two components of carbamylcholineinduced loss of nicotinic acetylcholine receptor function in the neuronal cell linePC12 // Mol. Pharmacol. 1986. Vol. 30. P. 6–12.230360. Simons K., Gerl M.J. Revitalizing membrane rafts: new tools and insights // Nat.Rev. Mol. Cell Biol. 2010.
Vol. 11, № 10. P. 688–699.361. Sine S.M. End-plate acetylcholine receptor: structure, mechanism, pharmacology,and disease // Physiol. Rev. 2012. Vol. 92, № 3. P. 1189–1234.362. Skou J.C. The influence of some cations on an adenosine triphosphatase fromperipherical nerves // Biochem. Biophys. Acta. 1957.
Vol. 23. P. 394–401.363. Song X.Z., Pedersen S.E. Electrostatic interactions regulate desensitization of thenicotinic acetylcholine receptor // Biophys. J. 2000. Vol. 78. P. 1324–1334.364. Sonnino S., Mauri L., Chigorno V., Prinetti A. Gangliosides as components of lipidmembrane domains // Glycobiology. 2007. Vol. 17, № 10. P. 1R–13R.365. Sooksawate T., Simmonds M. A. Effects of membrane cholesterol on thesensitivity of the GABA(A) receptor to GABA in acutely dissociated rathippocampal neurons // Neuropharmacol. 2001. Vol. 40, № 2. P. 178–184.366.
Sparks J.A., Pauly J.R. Effects of continuous oral nicotine administration on brainnicotinic receptors and responsiveness to nicotine in C57Bl/6 mice //Psychopharmacology (Berl). 1999. Vol. 141, № 2. P. 145-53.367. Sperelakis, NIn: Cell Physiology Sourcebook, 3rd Ed., Academic Press, New York.2001. pp. 231–236.368. Sweadner K.J. Na, K-ATPase and its isoforms / In: Neuroglia (eds. Kettenmann H.,Ransom B.R.). Oxford University Press. New York, Oxford.
1995. P.259–272.369. Sweadner, K.J., Rael, E. The FXYD gene family of small ion transport regulatorsor channels: cDNA sequence, protein signature sequence, and expression //Genomics. 2000. Vol. 68. P. 41–56.370. Takeuchi A., Reyes N., Artigas P., Gadsby D.C.
The ion pathway through theopened Na+, K+-ATPase pump // Nature. 2008. Vol. 456, № 7220. P. 413-416.371. Tanaki H., Klink R., Lena C., Korn H., Changeux J.-P. Calcium mobilizationelicited by two types of nicotinic acetylcholine receptors in mouse substantia nigrapars compacta // Eur. J. Neurosci. 2000. Vol. 12.
P. 2475–2485.231372. Tao Q.F., Hollenberg N.K., Price D.A., Graves S.W. Sodium pump isoformspecificity for the digitalis-like factor isolated from human peritoneal dialysate //Hypertension. 1997. Vol. 29. P. 815–821.373. Therien A.G., Pu H.X., Karlish S.J., Blostein R. Molecular and functional studiesof the gamma subunit of the sodium pump // J. Bioenerg. Biomembr. 2001. Vol.33, № 5.
P. 407–714.374. Thomas R. Electrogenic sodium pump in nerve and muscle cells // Physiol. Rev.1972. Vol. 52. P. 563–594.375. Thompson C.B., Choi C., Youn J.H., McDonough A.A. Temporal responses ofoxidative vs. glycolytic skeletal muscles to K+ deprivation: Na+ pumps and cellcations // Am. J. Physiol. 1999. Vol.
276 (Cell Physiol. 45). P. C1411–C1419.376. Thompson C.B., Dorup I., Ahn J., Leong P.K.K., McDonough A.A.Glucocorticoids increase sodium pump α2- and β1-subunit abundance and mRNAin rat skeletal muscle // Am. J. Physiol. Cell Physiol. 2001. Vol. 280. P. C509–C516.377. Thompson C.B., McDonough A.A. Skeletal muscle Na,K-ATPase and β subunitprotein levels respond to hypokalemic challenge with isoform and muscle typespecificity // J. Biol. Chem. 1996. Vol. 271.
P. 32653–32658.378. Tian J., Cai T., Yuan Z., Wang H., Liu L., Haas M., Maksimova E., Huang X.Y.,Xie Z.J. Binding of Src to Na+,K+-ATPase forms a functional signaling complex //Mol. Biol. Cell. 2006. Vol. 17, № 1. Р.317–326.379. Tian J., Xie Z. J. The Na-K-ATPase and calcium-signaling microdomains //Physiology. 2008. Vol. 23. P. 205–211.380. Tian L., Prior C., Dempster J., Marshall I.G. Nicotinic antagonist-producedfrequency-dependent changes in acetylcholine release from rat motor nerveterminals // J. Physiol. 1994. Vol. 476.
P. 517–529.381. Tikhonov D.B., Zhorov B.S.. Kinked-helices model of the nicotinic acetylcholinereceptor ion channel and its complexes with blockers: simulation by the MonteCarlo minimization method // Biophys J. 1998. Vol. 74, № 1. P. 242–55.232382. Tintignac L.A., Brenner H.R., Rüegg M.A. Mechanisms regulating neuromuscularjunction development and function and causes of muscle wasting // Physiol. Rev.2015. Vol. 95. P. 809–852.383. Tricarico D., Mele A., Camerino G. M., Bottinelli R., Brocca L., Frigeri A., SveltoM., George A. L.
Jr., Conte Camerino D. The KATP channel is a molecular sensorof atrophy in skeletal muscle // J. Physiol. 2010. Vol. 588, № 5. Р. 773–784.384. Tyapkina O., Volkov E., L. Nurullin L., Shenkman B., Kozlovskaya I., NikolskyE., Vyscocil F. Resting membrane potential and Na+, K+ - ATPase of rat and slowmuscles during modeling of hypogravity// Physiol. Res. 2009. Vol. 58. P. 599–603.385. Vagin O., Dada L.A., Tokhtaeva E., Sachs G.
The Na-K-ATPase α₁β₁ heterodimeras a cell adhesion molecule in epithelia // Am. J. Physiol. Cell Physiol. 2012. Vol.302, № 9. P. C1271–C1281.386. van der Pijl E.M., van Putten M., Niks E.H., Verschuuren J.J., Aartsma-Rus A.,Plomp J.J. Characterization of neuromuscular synapse function abnormalities inmultiple Duchenne muscular dystrophy mouse models // Eur. J. Neurosci. 2016.Vol. 43, № 12.
P. 1623–1635.387. Vilchinskaya N.A., Mirzoev T.M., Lomonosova Y.N., Kozlovskaya I.B.,Shenkman B.S. Human muscle signaling responses to 3-day head-out dryimmersion // J. Musculoskelet. Neuronal. Interact. 2015. Vol. 15, № 3. P. 286-293.388. Vilchinskaya N.A., Mochalova E.P., Nemirovskaya T.L., Mirzoev T.M., TurtikovaO.V., Shenkman B.S. Rapid decline in MyHC I(β) mRNA expression in rat soleusduring hindlimb unloading is associated with AMPK dephosphorylation // J.Physiol. 2017. Vol.
595, № 23. P. 7123–7134.389. Vyskocil F., Malomouzh A.I., Nikolsky E.E. Non-quantal acetylcholine release atthe neuromuscular junction // Physiol. Res. 2009. Vol.58. P.763 – 784.390. Vyskocil F., Nikolsky E., Edwards C. An analysis of the mechanisms underlyingthe non–quantal release of acetylcholine at the mouse neuromuscular junction //Neurosci. 1983. Vol. 9, № 2. P. 429–435.391. Walaas S.I., Czernik A.J., Olstad O.K., Sletten K., Walaas O. Protein kinase C andcyclic AMP-dependent protein kinase phosphorylate phospholemman, an insulin233and adrenaline-regulated membrane phosphoprotein, at specific sites in thecarboxy terminal domain // Biochem.
J. 1994. Vol. 304 (Pt 2). P.635–40.392. Wang H., Haas M., Liang M., Cai T., Tian J., Li S., Xie Z. Ouabain assemblessignaling cascades through the caveolar Na+/K+-ATPase // J. Biol. Chem. 2004.Vol. 279, № 17. P. 17250–17259.393. Wang H., Sun X. Desensitized nicotinic receptors in brain // Brain ResearchReviews. 2005. Vol. 48. P. 420–437.394. Wang J., Velotta J.B., McDonough A., Farley R.A.
All human Na+-K+-ATPasealpha-subunit isoforms have a similar affinity for cardiac glycosides// Am. J.Physiol. 2001. Vol. 281, № 14. P. C1336–C1343.395. Wang L., McComb J.G., Weiss M.H., McDonough A.A, Zlokovic B.V. Nicotinedownregulates 2 isoform of Na,K-ATPase at the blood-brain barrier and brain inrats // Biochem. Biophys. Res. Commun. 1994.
Vol. 199. P. 1422–1427.396. Wang X., Bogdanov M., Dowhan W. Topology of polytopic membrane proteinsubdomains is dictated by membrane phospholipid composition // EMBO J. 2002.Vol. 21, № 21. P. 5673–5681.397. Willadt S., Nash M., Slater C.R. Age-related fragmentation of the motor endplate isnot associated with impaired neuromuscular transmission in the mouse diaphragm// Sci Rep. 2016. Vol. 6. P. 24849.398.
Williams M.W., Resneck W.G., Kaysser T. Ursitti J.A., Birkenmeier C.S., BarkerJ.E., Bloch R.J. Na,K–ATPase in skeletal muscle: two populations of –spectrincontrol localization in the sarcolemma but not partitioning between thesarcolemma and the transverse tubule // J. Cell Sci. 2001. Vol. 114. Р. 751–762.399. Willmann R., Pun S., Stallmach L., Sadasivam G., Santos A.F., Caroni P., FuhrerC. Cholesterol and lipid microdomains stabilize the postsynapse at theneuromuscular junction // EMBO J.
2006. Vol. 25. P. 4050–4060.400. Wilson G.G., Karlin A. Acetylcholine receptor channel structure in the resting,open, and desensitized states probed with the substituted-cysteine-accessibilitymethod // PNAS. 2001. Vol. 98. P. 1241–1248.234401. Wilson M.H., Deschenes M.R.
The neuromuscular junction: anatomical featuresand adaptations to various forms of increased, or decreased neuromuscular activity// Int. J. Neurosci. 2005. Vol. 115, № 6. P. 803-28.402. Woo A.L., James P.F., Lingrel J.B. Sperm motility is dependent on a uniqueisoform of the Na, K-ATPase // J. Biol. Chem. 2000.
Vol. 275. P. 20693–20699.403. Wood S.J., Slater C.R. Safety factor at the neuromuscular junction Prog //Neurobiol. 2001. Vol. 64. P. 393–429.404. Wyckelsma VL, McKenna MJ, Effects of Age on Na+,K+-ATPase Expression inHuman and Rodent Skeletal Muscle // Front. Physiol. 2016. Vol. 7. P. 316.405. Xie Z., Askari A. Na+/K+-ATPase as a signal transducer // Eur. J. Biochem. 2002.Vol. 269. P.
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