Диссертация (1139576), страница 48
Текст из файла (страница 48)
N. Role of antibiotic penetration limitation in Klebsiella pneumoniaebiofilm resistance to ampicillin and ciprofloxacin [Text] / J. N. Anderl, M. J. Franklin,P.S. Stewart // Antimicrobial Agents and Chemotherapy. — 2000. — Vol. 44. — P.1818–1824.124. Andrian,E.Porphyromonasgingivalis-epithelialcellinteractionsinperiodontitis [Text] / E. Andrian, D.
Grenier, M. Rouabhia // Journal of DentalResearch. — 2006. — Vol. 85 (5). — P. 392–403.297125. Anwar, H. Dynamic interactions of biofilms of mucoid Pseudomonas aeruginosawith tobramycin and piperacillin [Text] / H. Anwar, J.L. Strap, K. Chen [et al.] //Antimicrobial Agents and Chemotherapy. — 1992. — Vol. 36.
— P. 1208–1214.126. Armitage, G. C. Development of a classification system for periodontal diseasesand conditions [Text] / G. C. Armitage // Annals of Periodontology. — 1999. — Vol.4. — P. 1–6.127. Armitage, P. Statistical methods in medical research / P. Armitage, G. Berry, J.N. S. Matthews. — 4th edn. — Blackwell Science Ltd, London, 2002. — 795 p.128.
Bachni, P. Electron microscopic study of the interaction of oral microorganismswith polymorphonuclear leukocytes [Text] / P. Bachni, M. A. Listgarten, N. S.Taichman [et al.] // Archives of Oral Biology. — 1977. — Vol. 22. — P. 685–692.129. Bahri, R. Candida famata modulates Toll-like receptor, β-defensin, andproinflammatory cytokine expression by normal human epithelial cells [Text] / R.Bahri, D. Saidane-Mosbahi, M. Rouabhia // Journal of Cellular Physiology.
— 2010.— Vol. 222 (1). — P. 209–218.130. Bahri, R. Normal Human Gingival Epithelial Cells Sense C. parapsilosis by TollLike Receptors and Module Its Pathogenesis through Antimicrobial Peptides andProinflammatory Cytokines [Text] / R. Bahri, S. Curt, D. Saidane-Mosbahi [et al.] //Mediators of Inflammation. — 2010. — Vol. 2010. — Article ID 940383. — 13 p.131.
Balaban, N. Q. Bacterial persistence as a phenotypic switch [Text] / N. Q.Balaban, J. Merrin, R. Chait [et al.] // Science. — 2004. — Vol. 305(5690). — P. 1622–1625.132. Barnes, T. C. The Many Faces of Interleukin-6: The Role of IL-6 inInflammation, Vasculopathy, and Fibrosis in Systemic Sclerosis [Text] / T. C. Barnes,M. E. Anderson, R. J. Moots // International Journal of Rheumatology. — 2011. —Vol.
2011 — Article ID 721608. — 6 p.133. Baroud, M. Underlying mechanisms of carbapenem resistance in extendedspectrum β-lactamase-producing Klebsiella pneumoniae and Escherichia coli isolatesat a tertiary care centre in Lebanon: role of OXA-48 and NDM-1 carbapenemases[Text] / M. Baroud, I. Dandache, G. F. Araj [et al.] // International Journal of298AntimicrobialAgents.—2013.—Vol.41(1).—P.75–79.—doi:10.1016/j.ijantimicag.2012.08.010. Epub 2012 Nov 9.134. Bartold, P. M. Periodontitis: A host-mediated disruption of microbialhomeostasis. Unlearning learned concepts [Text] / P. M. Bartold, T. E.
Van Dyke //Periodontology 2000. — 2013. — Vol. 62. — P. 203–217.135. Batoni, G. Use of antimicrobial peptides against microbial biofilms: advantagesand limits [Text] / G. Batoni, G. Maisetta, F. L. Brancatisano [et al.] // CurrentMedicinal Chemistry. — 2011. — Vol. 18 (2). — P. 256–279.136. Beikler, T. Oral biofilm-associated diseases: trends and implications for qualityof life, systemic health and expenditures [Text] / T. Beikler, T. F.
Flemmig //Periodontology 2000. — 2011. — Vol. 55. — P. 87–103.137. Beklen, A. Toll-like receptors 2 and 5 in human gingival epithelial cells cooperate with T-cell cytokine interleukin-17 [Text] / A. Beklen, T. Sorsa, Y. T.Konttinen // Oral Microbiology and Immunology. — 2009. — Vol. 24. — P. 38–42.138. Belibasakis, G. N. Interleukin-8 responses of multi-layer gingival epithelia tosubgingival biofilms: role of the “red complex” species [Text] / G. N. Belibasakis, T.Thurnheer, N. Bostanci // PLoS One. — 2013. — Vol.
10 (8). —doi:10.1371/journal.pone.0081581139. Belibasakis, G. N. Oral biofilm challenge regulates the RANKL-OPG system inperiodontal ligament and dental pulp cells [Text] / G. N. Belibasakis, A. Meier, B.Guggenheim [et al.] // Microbial Pathogenesis. — 2011. — Vol.
50. — P. 6–12.140. Benakanakere, M. TLR2 promoter hypermethylation creates innate immunedysbiosis [Text] / M. Benakanakere, M. Abdolhosseini, K. Hosur, [et al.] // Journal ofDental Research. — 2015. — Vol. 94 (1). — P. 183–191.141. Bender, J. S. Novel rinse assay for the quantification of oral neutrophils and themonitoring of chronic periodontal disease [Text] / J. S. Bender, H.
Thang, M. Glogauer// Journal of Periodontal Research. — 2006. — Vol. 41 (3). — P. 214–220.142. Benzer, D. Vancomycin-resistant enterococcus colonization in neonatalintensive care unit: prevention and eradication experience [Text] / D. Benzer, D.299Yavuzcan Öztürk, T. Gürsoy [et al.] // Mikrobiyoloji Bulteni. — 2012. — Vol. 46 (4).— P. 682–688.143. Beutler, B. How we detect microbes and respond to them: the Toll-like receptorsand their transducers [Text] / B. Beutler, K. Hoebe, X.
Du [et al.] // Journal ofLeukocyte Biology. — 2003. — Vol. 74 (4). — P. 479–485. Epub 2003 Jul 1.144. Bickel, M. Acid-base properties of human gingival crevicular fluid [Text] / M.Bickel, J. L. Munoz, P. Giovannini // Journal of Dental Research. — 1985. — Vol. 64.— P. 1218–1220.145.
Bickel, M. The pH of human crevicular fluid measured by a new microanalyticaltechnique [Text] / M. Bickel, G. Cimasoni // Journal of Dental Research. — 1985. —Vol. 20. — P. 35–40.146. Binnig, G. Atomic Force Microscope [Text] / G. Binnig, C.F. Quate // PhysicalReview Letters. — 1986. — Vol. 56 (9). — P.
930–933.147. Bodet, C. Inflammatory responses of a macrophage/epithelial cell co-culturemodel to mono and mixed infections with Porphyromonas gingivalis, Treponemadenticola, and Tannerella forsythia [Text] / C. Bodet, F. Chandad, D. Grenier //Microbes and Infection. — 2006. — Vol. 8 (1). — P. 27–35.148. Bostanci, N. Porphyromonas gingivalis regulates TREM-1 in humanpolymorphonuclear neutrophils via its gingipains [Text] / N. Bostanci, T.
Thurnheer,J. Aduse-Opoku [et al.] // PLoS ONE. — 2013. — Vol. 8 (10). — e75784. —doi:10.1371/journal.pone.0075784149. Bostanci, N. Porphyromonas gingivalis: an invasive and evasive opportunisticoral pathogen [Text] / N. Bostanci, G. N. Belibasakis // FEMS Microbiology Letters.— 2012. — Vol. 333. — P. 1–9.150. Boyce, B. F. Effects of interleukin-1 on bone turnover in normal mice [Text] /B. F. Boyce, T. B.
Aufdemorte, I. R. Garrett [et al.] // Endocrinology. — 1989. — Vol.125. — P. 1142–1150.151. Bradshaw, D. J. Role of Fusobacterium nucleatum and coaggregation inanaerobe survival in planktonic and biofilm oral microbial communities during300aeration [Text] / D.J. Bradshaw, P. D. Marsh, G. K. Watson [et al.] // Infection andImmunity. — 1998. — Vol. 66.
— P. 4729–4732.152. Brandtzaeg, P. Homeostatic impact of indigenous microbiota and secretoryimmunity [Text] / P. Brandtzaeg // Beneficial microbes. — 2010. — Vol. 1 (3). — P.211–227. — doi:10.3920/BM2010.0009.153. Brandtzaeg, P. The gut as communicator between environment and host:immunological consequences [Text] / P. Brandtzaeg // European Journal ofPharmacology.—2011.—Vol.668.—P.16–32.—doi:10.1016/j.ejphar.2011.07.006. Epub 2011 Jul 28.154. Brocker, C.
Evolutionary divergence and functions of the human interleukin (IL)gene family [Text] / C. Brocker, D. Thompson, A. Matsumoto [et al.] // HumanGenomics. — 2010. — Vol. 5. — P. 30–55.155. Brown, J. TLR-signaling networks: an integration of adaptor molecules, kinases,and cross-talk [Text] / J. Brown, H. Wang, G. N.
Hajishengallis [et al.] // J Dent Res.— 2011. — Vol. 90. — P. 417–427.156. Burton J. P. The rationale and potential for the reduction of oral malodour usingStreptococcus salivarius probiotics [Text] / J.P. Burton // Oral Diseases. — 2005. —Vol. 11 (1).
— P. 29-31.157. Burton, J. P. Beneficial microbes for the oral cavity: time to harness the oralstreptococci? [Text] / J. P. Burton, P. A. Wescombe, P. A. Cadieux [et al.] // BenefMicrobes. — 2011. — Vol. 2. — P. 93–101.158. Cabiscol, E.
Oxidative stress in bacteria and protein damage by reactive oxygenspecies [Text] / E. Cabiscol, J. Tamarit, J. Ros // International Microbiology. — 2000.— Vol. 3. — P. 3–8.159. Calkins, C. C. Inactivation of tumor necrosis factor-alpha by proteinases(gingipains) from the periodontal pathogen, Porphyromonas gingivalis. Implicationsof immune evasion [Text] / C. C. Calkins, K.
Platt, J. Potempa [et al.] // Journal ofBiological Chemistry. — 1998. — Vol. 273. — P. 6611–6614.160. Canabarro, A. Association of subgingival colonization of Candida albicans andother yeasts with severity of chronic periodontitis [Text] / A. Canabarro, C. Valle, M.301R. Farias [et al.] // Journal of Periodontal Research. — 2013. —Vol. 48. — P. 428–432.161.
Cardoso, C. R. Evidence of the presence of T helper type 17 cells in chroniclesions of human periodontal disease [Text] / C. R. Cardoso, G. P. Garlet, G. E. Crippa// Oral Microbiology and Immunology. — 2009. — Vol. 24. — P. 1–6.162. Carrillo-de-Albornoz, A. Gingival changes during pregnancy: II. Influence ofhormonal variations on the subgingival biofilm [Text] / A.