Диссертация (Способы коррекции ранних механизмов канцерогенеза в органах репродуктивной системы), страница 32
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(2001). Biochem. Biophys. Res. Commun.,285, 1102–1106.141.He L, Hannon GJ. MicroRNAs: small RNAs with a big role in generegulation. Nat Rev Genet. 2004;5:522–531.196142.Heath E.I., Heilbrun L.K., Li J., Vaishampayan U., Harper F.,Pemberton P., Sarkar F.H. A phase I dose-escalation study of oral BRDIM (BioResponse 3,3'- Diindolylmethane) in castrate-resistant, nonmetastatic prostate cancer. Am J Transl Res. 2010; 2(4):402-11.143.Henken F.E., Wilting S.M., Overmeer R.M., van Rietschoten J.G.,Nygren A.O., Errami A., et al.
Sequential gene promoter methylationduring HPV-induced cervical carcinogenesis. Br J Cancer 2007;97:1457–64.144.Ho G.Y., Bierman R., Beardsley L., Chang C.J., Burk R.D. Naturalhistory of cervicovaginal papillomavirus infection in young women N.Engl. J. Med., 338 (1998), pp. 423–428.145.Hong C., Kim H.A., Firestone G.L., Bjeldanes L.F. 3,3'-Diindolylmethane (DIM) induces a G(1) cell cycle arrest in human breastcancer cells that is accompanied by Sp1-mediated activation ofp21(WAF1/CIP1) expression. Carcinogenesis. 2002; 23(8):1297-305.146.Hong W.K., Sporn M.B.
Recent Advances in Chemoprevention ofCancer. Science, 1997; 278:1073–77.147.Hoshino H, Miyoshi N, Nagai K, Tomimaru Y, Nagano H,Sekimoto M, Doki Y, Mori M, Ishii H. Epithelial-mesenchymaltransition with expression of SNAI1-induced chemoresistance incolorectal cancer.
Biochem Biophys Res Commun 2009; 390: 1061–1065.148.Hudson D.L. Epithelial stem cells in human prostate growth anddisease. Prostate Cancer Prostatic. Dis., 2004; 7, 188–194.149.Hussain T., Gupta S., Adhami V.M., Mukhtar H. Green teaconstituent epigallocatechin-3-gallate selectively inhibits COX-2without affecting COX-1 expression in human prostate carcinoma cells.Int J Cancer. 2005; 113(4):660-669.197150.Hwang C., Sethi S., Heilbrun L.K., Gupta N.S., Chitale D.A., SakrW.A., Menon M., Peabody J.O., Smith D.W., Sarkar F.H., Heath E.I.Anti-androgenic activity of absorption-enhanced 3, 3'-diindolylmethanein prostatectomy patients.
Am J Transl Res. 2016; 8(1):166-76.151.Iczkowski KA, Chen HM, Yang XJ, Beach RA. Prostate cancerdiagnosed after initial biopsy with atypical small acinar proliferationsuspicious for malignancy is similar to cancer found on initial biopsy.Urology. 2002; 60(5):851-4.152.InternationalWorldCancerResearchFund,http://www.wcrf.org/int/cancer-facts-figures/data-specificcancers/endometrial-cancer-cancer-lining-womb-statistics. [access date05/08/2016]153.Iorio MV, Ferracin M, Liu CG, Veronese A, Spizzo R, Sabbioni S,et al. MicroRNA gene expression deregulation in human breast cancer.Cancer Res. 2005; 65:7065–7070.154.Islam S., Islam N., Kermode T., Johnstone B., Mukhtar H.,Moskowitz R.W., Goldberg V.M., Malemud C.J., Haqqi T.M.Involvement of caspase-3 in epigallocatechin-3-gallate-mediatedapoptosis of human chondrosarcoma cells.
Biochem Biophys ResCommun. 2000; 270(3):793-7.155.Iwata T., Fujii T., Morii K., Saito M., Sugiyama J., Nishio H.,Morisada T., Tanaka K., Yaguchi T., Kawakami Y., Aoki D. Cytokineprofile in cervical mucosa of Japanese patients with cervicalintraepithelial neoplasia. Int J Clin Oncol. 2015; 20(1):126-33.156.Jellinck P.H., Forkert PG, Riddick DS, Okey AB, Michnovicz JJ,Bradlow HL. Ah receptor binding properties of indole carbinols andinduction of hepatic estradiol hydroxylation. Biochem Pharmacol.
1993;45(5):1129-36.198157.Jemal A., Bray F., Center M.M., Ferlay J., Ward E., Forman D.Global cancer statistics. CA Cancer J Clin. 2011; 61(2):69-90.158.Jemal A., Siegel R., Xu J., Ward E. Cancer statistics CA Cancer JClin, 60 (2010), pp. 277–300.159.Jiang J., Mo Z.C., Yin K., Zhao G.J., Lv Y.C., Ouyang X.P., JiangZ.S., Fu Y., Tang C.K.
Epigallocatechin-3-gallate prevents TNF-αinduced NF-κB activation thereby upregulating ABCA1 via theNrf2/Keap1 pathway in macrophage foam cells. Int J Mol Med. 2012;29(5):946-56.160.Jones P.A., Baylin S.B. The fundamental role of epigenetic eventsin cancer // Nat Rev Genet. – 2002. – Vol. 3. – P. 415-428.161.Kabat G.C., Chang C.J., Sparano J.A., Sepkovic D.W., Hu X.P.,Khalil A., Rosenblatt R., Bradlow H.L. Urinary estrogen metabolites andbreast cancer: a case-control study. Cancer Epidemiol Biomarkers Prev.1997; 6(7): 505-9.162.Kaei Nasu, Yukie Kawano, Yoshiyuki Tsukamoto, MasayukiTakano, Noriyuki Takai, Haili Li, Yuichi Furukawa, Wakana Abe,Masatsugu Moriyama and Hisashi Narahara. Aberrant DNA methylationstatus of endometriosis: Epigenetics as the pathogenesis, biomarker andtherapeutic target.
Journal of Obstetrics and Gynaecology ResearchVolume 37, Issue 7, pages 683–695, 2011.163.Kaku T., Tsukamoto N. et al. Endometrial carcinoma associated withhyperplasia // Gynecol. Oncol. – 1996. – Vol. 60(1). – P. 22-25.164.Kamb A. Cyclin-dependent kinase inhibitors and human cancer.Curr. Top. Microbiol.
Immunol. 1998; 227, 139.165.Kandala P.K., Srivastava S.K. Regulation of Janus-activatedkinase-2 (JAK2) by diindolylmethane in ovarian cancer in vitro and invivo. Drug Discov Ther. 2012; 6(2):94-101.199166.Kaur M., Agarwal C., Agarwal R. Anticancer and cancerchemopreventive potential of grape seed extract and other grape-basedproducts. J Nutr. 2009; 139(9):1806S-12S.167.Khan N., Afaq F., Saleem M., Ahmad N., Mukhtar H. TargetingMultipleSignalingPathwaysbyGreenTeaPolyphenolEpigallocatechin-3-Gallate, Cancer Res, 2006; 66(5):2500-5.168.Kijima I., Phung S., Hur G., Kwok S.L., Chen S. Grape seed extractis an aromatase inhibitor and a suppressor of aromatase expression.Cancer Res.
2006; 66:5960–7.169.Kikuno N., et al., Genistein mediated histone acetylation anddemethylation activates tumor suppressor genes in prostate cancer cells.Int J Cancer, 2008. 123(3): p. 552-60.170.Kim J., Zhang X., Rieger-Christ K.M., Summerhayes I.C., WazerD.E., et al. (2006) Suppression of Wnt Signaling by the Green TeaCompound (−)-Epigallocatechin 3-Gallate (EGCG) in Invasive BreastCancer Cells. Journal of Biological Chemistry 281: 10865–10875.171.Kiselev V.I., Drukh V.M., Pchelintseva O.I., Muyzhnek E.L.,Kuznetsov I.N., Paltsev M.A.
Preclinical antitumor activity of thediindolylmethane formulation in xenograft mouse model of prostatecancer. Experimental Oncology. - 2014. - Т. 36. - № 2. - P. 90-93.172.Kong D., Heath E., Chen W., et al. Loss of let-7 up-regulates EZH2in prostate cancer consistent with the acquisition of cancer stem cellsignatures that are attenuated by BR-DIM. PLoS ONE 2012;7(3):e33729.173.Kovacs JJ, Murphy PJ, Gaillard S, Zhao X, Wu JT, Nicchitta CV,Yoshida M, Toft DO, Pratt WB, Yao T. P.HDAC6 regulates Hsp90acetylation and chaperone-dependent activationof glucocorticoidreceptor. Mol. Cell.
2005; 18: 601-607.200174.Kramer O.H., Zhu P., Ostendorff H.P. et al. (2003) The histonedeacetylase inhibitor valproic acid selectively induces proteasomaldegradation of HDAC2. EMBO J., 22, 3411-3420.175.Krishnamurthy P, Schuetz JD. Role of ABCG2/ BCRP in biologyand medicine. Annu Rev Pharm macol Toxicol 2006;46:381-410.176.Lacey J. V., Victoria M. C. // Endometrial hyperplasia and the riskof progression to carcinoma. Review. Maturitas. – 2009. – Vol. 63. – P.39–44.177.Lai L.C., Erbas H., Meadows K.A., Lennard T.W., Holly J.M.Insulin-like growth factor binding protein-3 in breast cyst fluid:relationships with insulin-like growth factors I and II and transforminggrowth factor-beta 1 and 2.
Cancer Lett. 1996; 110(1-2):207-12.178.Landen CN, Birrer MJ, Sood AK. Early events in the pathogenesisof epithelial ovarian cancer. J Clin Oncol. 2008; 26(6):995-1005.179.Landis-Piwowar K.R., Huo C., Chen D., Milacic V., Shi G., ChanT.H., Dou Q.P. A novel prodrug of the green tea polyphenol (-)epigallocatechin-3-gallate as a potential anticancer agent. Cancer Res.2007 May 1;67(9):4303-10.180.Larsson S.C., et al. Dietary long-chain n-3 fatty acids for theprevention of cancer: a review of potential mechanisms.
Am J Clin Nutr.,2004; 79:935–45.181.Lavoie G., Estève P.O., Laulan N.B., Pradhan S., St-Pierre Y. PKCisoforms interact with and phosphorylate DNMT1. BMC Biol 2011,9(1):31.182.Lawen A. Apoptosis - an introduction. Bioessays. 2003; 25(9):888-96.183.Le H.T., Schaldach C.M., Firestone G.L., Bjeldanes L.F. Plant-derived 3,3'-Diindolylmethane is a strong androgen antagonist in humanprostate cancer cells.J Biol Chem. 2003; 278(23):21136-21145.201184.Lee W. J., Shim J. Y., Zhu B. T.
Mechanisms for the inhibition ofDNA methyltransferases by tea catechins and bioflavonoids. MolPharmacol 68, 1018-1030 (2005);185.Leong H., Firestone G.L., Bjeldanes L.F. Cytostatic effects of 3,3'-diindolylmethane in human endometrial cancer cells result from anestrogen receptor-mediated increase in transforming growth factor-alphaexpression. Carcinogenesis. 2001; 22(11):1809-17.186.Letyagin V. P. Mastopathy.
RMM Gynaecology. 2000; 8:468-472.187.Li Y., Li X., Guo B. (2010) Chemopreventive agent 3,3’-diindolylmethane selectively induces proteasomal degradation of class Ihistone deacetylases. Cancer Res., 70(2), 646-654.188.Li Y., Yuan Y.Y., Meeran S.M., Tollefsbol T.O. Synergisticepigenetic reactivation of estrogen receptor-alpha (ERalpha) bycombined green tea polyphenol and histone deacetylase inhibitor inERalpha-negative breast cancer cells. Mol Cancer 2010; 9:274.189.Lin J.K.
Cancer chemoprevention by tea polyphenols throughmodulating signal transduction pathways. Arch Pharm Res. 2002;25(5):561-71.190.Lord R.S., Bongiovanni B., Bralley J.A. Estrogen metabolism andthe diet-cancer connection: rationale for assessing the ratio of urinaryhydroxylated estrogen metabolites.