Диссертация (1174300), страница 36
Текст из файла (страница 36)
471-480.186. Martini, A. It is time to rethink juvenile idiopathic arthritis classification andnomenclature / A. Martini // Ann Rheum Dis. – 2012. – Vol. 71. – P. 1437–1439.187. Mathew, C. G. P. The Isolation of High Molecular Weight Eukaryotic DNA /C.
G. P. Mathew // Methods Mol Biol. – 1984. – Vol. 2. – P. 31-34.188. McDonald, J. H. Handbook of Biological Statistics / J. H. McDonald. ‒ 3rded. ‒ Baltimore, Maryland : Sparky House Publishing, 2014. ‒ 299 p.189. McHugh, M. L. The Chi-square test of independence / M. L. McHugh// Biochem Med (Zagreb). – 2013. ‒ Vol.
23, № 2. – P. 143-149.190. Meta-analysis confirms association between TNFA-G238A variant and JIA,and between PTPN22-C1858T variant and oligoarticular, RF-polyarticular and RFpositive polyarticular JIA [Электронный ресурс] / M. J. Kaalla [et al.] // PediatrRheumatol. – 2013.
– Vol. 11. – ID 40. – URL: https://doi.org/10.1186/1546-0096-1140. (10.04.2017).191. Meta-Analysis: Diagnostic Accuracy of Anti-Cyclic Citrullinated PeptideAntibody for Juvenile Idiopathic Arthritis / Y. Wang [et al.] // J Immunol Res. – 2015. –Vol. 2015. – ID 915276.192. Methotrexate improves the health-related quality of life of children withjuvenile idiopathic arthritis / A. Cespedes-Cruz [et al.] // Ann Rheum Dis. – 2008.
– Vol.18167, № 3. – P. 309-314.193. Moore, J. H. Epistasis analysis using multifactor dimensionality reduction / J.H. Moore, P. C. Andrews // Methods Mol. Biol. – 2015. ‒ Vol. 1253. – P. 301-314.194. Mukaka M. M. Statistics corner: A guide to appropriate use of correlationcoefficient in medical research / M. M. Mukaka // Malawi Med J. – 2012. ‒ Vol.
24, №3. – P. 69-71.195. Multiple Autoimmune-Associated Variants Confer Decreased IL-2RSignaling in CD4+CD25hi T Cells of Type 1 Diabetic and Multiple Sclerosis Patients[Электронный ресурс] / K. Cerosaletti [et al.] // PLoS ONE. – 2013. – Vol. 8, № 12. –e83811. – URL: https://doi.org/10.1371/journal.pone.0083811. (10.04.2017).196. Mutation screening of the macrophage migration inhibitory factor gene:positive association of a functional polymorphism of macrophage migration inhibitoryfactor with juvenile idiopathic arthritis / R.
Donn [et al.] // Arthritis Rheum. – 2002. –Vol. 46, № 9. – P. 2402-2409.197. NETs are a source of citrullinated autoantigens and stimulate inflammatoryresponses in rheumatoid arthritis / R. Khandpur [et al.] // Sci Transl Med. – 2013. – Vol.5, № 178. – P. 178ra40.198. NFKB1 promoter variation implicates shear-induced NOS3 gene expressionand endothelial function in prehypertensives and stage I hypertensives / J. Y. Park [et al.]// Am J Physiol Heart Circ Physiol. – 2007. – Vol. 293, № 4. – H2320–H2327.199.
Nishimoto, N. Interleukin 6: from bench to bedside / N. Nishimoto, T.Kishimoto // Nat Clin Pract Rheumatol. – 2006. – Vol. 2, № 11. – P. 619-626. – Review.– Erratum in: Nat Clin Pract Rheumatol. – 2006. – Vol. 2, № 12. – P. 691.200. Niu, X. IL-21 and Related Diseases / X. Niu, G. Chen // J Clin Cell Immunol.– 2013. – Vol. 4, Special № 1. – pii: 008.201. No association of the NFKB1 promoter polymorphism with ulcerative colitisin a British case control cohort / M. M.
Mirza [et al.] // Gut. – 2005. – Vol. 54, № 8. – P.1205-1206.202. Novel IL10 gene family associations with systemic juvenile idiopathicarthritis [Электронный ресурс] / M. S. Fife [et al.] // Arthritis Res Ther. – 2006. – Vol.1828, № 5. – R148. – URL: https://doi.org/10.1186/ar2041. (10.04.2017).203. Novel self-epitopes derived from aggrecan, fibrillin, and matrixmetalloproteinase-3 drive distinct autoreactive T-cell responses in juvenile idiopathicarthritis and in health [Электронный ресурс] / S. Kamphuis [et al.] // Arthritis Res Ther.– 2006.
– Vol. 8. – R178. – URL: https://doi.org/10.1186/ar2088. (10.04.2017).204. Outcome in adults with juvenile idiopathic arthritis: a quality of life study / H.E. Foster [et al.] // Arthritis Rheum. ‒ 2003. ‒ Vol. 48, № 3. ‒ P. 767–775.205. PAD4-Mediated Neutrophil Extracellular Trap Formation Is Not Required forImmunity against Influenza Infection [Электронный ресурс] / S. Hemmers [et al.] //PLoSONE.–2011.–Vol.6,№7.–e22043.–URL:https://doi.org/10.1371/journal.pone.0022043. (10.04.2017).206.
Parvovirus B19 may have a role in the pathogenesis of juvenile idiopathicarthritis / B. Gonzalez [et al.] // J Rheumatol. – 2007. – Vol. 34, № 6. – P. 1336-1340.207. Pastinen, T. Influence of human genome polymorphism on gene expression /T. Pastinen, B. Ge, T. J. Hudson // Hum Mol Genet. – 2006. – Vol. 15, Suppl. 1. – R9R16.208. Pathogenic implications for autoimmune mechanisms derived by comparativeeQTL analysis of CD4+ versus CD8+ T cells / S.
Kasela [et al.] // PLoS Genet. – 2017. –Vol. 13, № 3. – e1006643.209. Pathologic patterns of interleukin 10 expression – A review / J. Trifunović [etal.] // Biochem Med (Zagreb). – 2015. – Vol. 25, № 1. – P. 36-48.210. Patterns of expression of tumor necrosis factor alpha, tumor necrosis factorbeta, and their receptors in synovia of patients with juvenile rheumatoid arthritis andjuvenile spondylarthropathy / A. A. Grom [et al.] // Arthritis Rheum. – 1996. – Vol. 39,№ 10. – P. 1703-1710.211. Peptidylarginine deiminase 4 and citrullination in health and disease / C.Anzilotti [et al.] // Autoimmun Rev. – 2010. – Vol.
9, № 3. – P. 158-160.212. Pereira, S. G. Nuclear factor-kappaB1: regulation and function / S. G. Pereira,F. Oakley // Int J Biochem Cell Biol. – 2008. – Vol. 40, № 8. – P. 1425-1430.213. Perinatal and early childhood risk factors associated with rheumatoid factor183positivity in a healthy paediatric population / K. A. Young [et al.] // Ann Rheum Dis.
–2007. – Vol. 66, № 2. – P. 179-183.214. Perinatal characteristics, early life infections and later risk of rheumatoidarthritis and juvenile idiopathic arthritis / C. Carlens [et al.] // Ann Rheum Dis. – 2009. –Vol. 68, № 7. – P. 1159-1164.215. Perkins, N. D. Integrating cell-signalling pathways with NF-kappaB and IKKfunction / N. D. Perkins // Nat Rev Mol Cell Biol. – 2007. – Vol.
8, № 1. – P. 49-62.216. Pharmacogenetics: can genes determine treatment efficacy and safety in JIA?/ H. Schmeling [et al.] // Nat. Rev. Rheumatol. ‒ 2014. ‒ Vol. 10, № 11. ‒ P. 682-690.217. Plasma concentrations of soluble IL-2 receptor α (CD25) are increased in type1 diabetes and associated with reduced C-peptide levels in young patients / K. Downes[et al.] // Diabetologia.
– 2014. – Vol. 57, № 2. – P. 366-372.218. Polymorphic haplotypes of the interleukin-10 5′ flanking region determinevariable interleukin-10 transcription and are associated with particular phenotypes ofjuvenile rheumatoid arthritis / E. Crawley [et al.] // Arthritis Rheum. – 1999.
– Vol. 42,№ 6. – P. 1101–1108.219. Polymorphic structure of the tumor necrosis factor (TNF) locus: an NcoIpolymorphism in the first intron of the human TNF-b gene correlates with a variant aminoacid in position 26 and a reduced level of TNF-b production / G. Messer [et al.] // J ExpMed.
– 1991. – Vol. 173. – P. 209–219.220. Polymorphic variant at the IL2 region is associated with type 1 diabetes andmay affect serum levels of interleukin-2 / M. Fichna [et al.] // Mol Biol Rep. – 2013. –Vol. 40, № 12. – P. 6957-6963.221. Polymorphisms at position -308 in the promoter region of the TNF-alpha andin the first intron of the TNF-beta genes and spontaneous and lipopolysaccharide-inducedTNF-alpha release in sarcoidosis / A. Somoskövi [et al.] // Cytokine. – 1999. – Vol.
11,№ 11. – P. 882–887.222. Polymorphisms of the interleukin-1beta gene affect the risk of myocardialinfarction and ischemic stroke at young age and the response of mononuclear cells tostimulation in vitro / L. Iacoviello [et al.] // Arterioscler Thromb Vasc Biol. – 2005. –184Vol. 25, № 1. – P. 222-227.223. Polymorphisms within Genes Involved in Regulation of the NF-κB Pathwayin Patients with Rheumatoid Arthritis / K. Gębura [et al.] // Int J Mol Sci. – 2017. – Vol.18, № 7.
– pii: E1432.224. Prahalad, S. A comprehensive review of the genetics of juvenile idiopathicarthritis [Электронный ресурс] / S. Prahalad, D. N. Glass // Pediatr Rheumatol. – 2008.– Vol. 6. – ID 11. – URL: https://doi.org/10.1186/1546-0096-6-11. (10.04.2017).225. Prahalad, S. Genetic analysis of juvenile rheumatoid arthritis: approaches tocomplex traits / S.
Prahalad // Curr Probl Pediatr Adolesc Health Care. – 2006. – Vol. 36.– P. 83–90.226. Prediction of clinical non-response to methotrexate treatment in juvenileidiopathic arthritis / M. Bulatovic [et al.] // Ann Rheum Dis. – 2012. – Vol. 71, № 9. – P.1484-1489.227. Predictive value of selected biomarkers, polymorphisms, and clinical featuresfor oligoarticular juvenile idiopathic arthritis-associated uveitis / L. Pelegrín [et al.] //Ocul Immunol Inflamm. – 2014. – Vol. 22, № 3. – P. 208-212.228.
Predictors of poor response to methotrexate in polyarticular-course juvenileidiopathic arthritis: analysis of the PRINTO methotrexate trial / I. Vilca [et al.] // AnnRheum Dis. – 2010. – Vol. 69, № 8. – P. 1479-1483.229. Preliminary definition of improvement in juvenile arthritis / E. H. Giannini [etal.] // Arthritis Rheum. ‒ 1997. ‒ Vol. 40, № 7. – P. 1202-1209.230. Pretreatment serum levels of anti-cyclic citrullinated peptide antibodies areassociated with the response to methotrexate in recent-onset arthritis / K.