Диссертация (1139576), страница 53
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— 2010. — Vol. 28. —P. 3496–3505.322. Liu, X. L. Analysis of resistance phenotype and homology of Klebsiellapneumoniae in burn Patients [Text] / X. L. Liu, D. Z. Peng, L. Xue [et al.] // ZhonghuaShao Shang Za Zhi. — 2012. — Vol. 28 (2). — P. 96–100.323. Liu, Y. C. Cytokine responses against periodontal infection: protective anddestructive roles [Text] / Y.
C. Liu, U. H. Lerner, Y. T. Teng // Periodontology 2000.— 2010. — Vol. 52. — P. 163–206.324. Lockhart, P. B. Periodontal disease and atherosclerotic vascular disease: doesthe evidence support an independent association?: a scientific statement from theAmerican Heart Association [Text] / P. B. Lockhart, A. F. Bolger, P. N.
Papapanou [etal.] // Circulation. — 2012. — Vol. 125 (20). — P. 2520–2544. —doi:10.1161/CIR.0b013e31825719f3. Epub 2012 Apr 18.325. Lopatin, D. E. Modulation of immunoreactivity to periodontal diseaseassociated microorganisms during pregnancy [Text] / D. E. Lopatin, K. S. Kornman,W. J. Loesche // Infection and Immunity. — 1980. — Vol. 28. — P. 713–718.326. Loppnow, H. Cytokine induction by lipopolysaccharide (LPS) corresponds tolethal toxicity and is inhibited by nontoxic Rhodobacter capsulatus LPS [Text] / H.Loppnow, P. Libby, M. Freudenberg [et al.] // Infection and Immunity.
— 1990. —Vol. 58 (11). — P. 3743–3750.327. Maekawa, T. Porphyromonas gingivalis manipulates complement and TLRsignaling to uncouple bacterial clearance from inflammation and promote dysbiosis[Text] / T. Maekawa, J. L. Krauss, T. Abe [et al.] // Cell Host & Microbe. — 2014. —11 june. — Vol.
15(6). — P. 768–778. — doi: 10.1016/j.chom.2014.05.012.328. Mah, T. F. A genetic basis for Pseudomonas aeruginosa biofilm antibioticresistance [Text] / T. F. Mah, B. Pitts, B. Pellock [et al.] // Nature. — 2003. — Vol.426. — P. 306–310.320329. Mahanonda, R. IL-8 and IDO expression by human gingival fibroblasts viaTLRs [Text] / R. Mahanonda, N. Sa-Ard-Iam, P. Montreekachon [et al.] // Journal ofImmunology.
— 2007. — Vol. 178. — P. 1151–1157.330. Mahanonda, R. Toll-like receptors and their role in periodontal health anddisease [Text] / R. Mahanonda, S. Pichyangkul // Periodontology 2000. — 2007. —Vol. 43. — P. 41–55.331. Mandrell, R. E. Lipooligosaccharides (LOS) of some Haemophilus speciesmimic human glycosphingolipids, and some LOS are sialylated [Text] / R. E. Mandrell,R. McLaughlin, Y. Aba Kwaik [et al.] // Infection and Immunity. — 1992. — Vol. 60.— P.
1322–1328.332. Mariani-Kurkdjian,P.Extended-spectrumbeta-lactamaseproducing-enterobacteriaceae [Text] / P. Mariani-Kurkdjian, C. Doit, E. Bingen // Archives ofPediatrics. — 2012. — Vol. 19 (3). — P. 93–96.333. Mariotti, A. Sex steroid hormones and cell dynamics in the periodontium [Text]/ A. Mariotti // Critical Reviews in Oral Biology & Medicine. — 1994. — Vol. 5. —P. 27–53.334. Marsh, P. D. How is the development of dental biofilms influenced by the host?[Text] / P.
D. Marsh, D. A. Devine // Journal of Clinical Periodontology. — 2011. —Vol. 38 (11). — P. 28–35.335. Marsh, P. D. Microbiology of dental plaque biofilms and their role in oral healthand caries [Text] / P. D. Marsh // Dental Clinics of North America. — 2010. — Vol.54 (3). — P. 441–454.336. May, T. B. Alginate synthesis by Pseudomonas aeruginosa: a key pathogenicfactor in chronic pulmonary infections of cystic fibrosis patients [Text] / T. B.
May, D.Shinabarger, R. Maharaj [et al.] // Clinical Microbiology Reviews. — 1991. — Vol. 4.— P. 191–206.337. McDougald, D. Should we stay or should we go: Mechanisms and ecologicalconsequences for biofilm dispersal [Text] / D. McDougald, S. A. Rice, N. Barraud [etal.] // Nature Reviews Microbiology. — 2012. — Vol. 10. — P. 39–50.321338. McGaha, T.
L. Amino acid catabolism: a pivotal regulator of innate and adaptiveimmunity [Text] / T. L. McGaha, L. Huang, H. Lemos [et al.] // ImmunologicalReviews. — 2012. — Vol. 249. — P. 135–157.339. McKenzie, R. M. Differential response of Porphyromonas gingivalis to varyinglevels and duration of hydrogen peroxide-induced oxidative stress [Text] / R. M.McKenzie, N. A.
Johnson, W. Aruni [et al.] // Microbiology. — 2012. — Vol. 158. —P. 2465–2479.340. McLaughlin, R. A. Interleukin-1beta-induced growth enhancement ofStaphylococcus aureus occurs in biofilm but not planktonic cultures [Text] / R. A.McLaughlin, A. J.
Hoogewerf // Microbial Pathogenesis. — 2006. — Vol. 41 (2–3).— P. 67–79.341. Medzhitov, R. Innate immune induction of the adaptive immune response [Text]/ R. Medzhitov, C. A. Jr. Janeway // Cold Spring Harbor Symposia on QuantitativeBiology. — 1999. — Vol. 64. — P. 429–435.342. Medzhitov, R.
Innate immunity [Text] / R. Medzhitov, C. Jr. Janeway // NewEngland Journal of Medicine. — 2000. — Vol. 343. — P. 338–344.343. Mezyk-Kopec, R. Inactivation of membrane tumor necrosis factor alpha bygingipains from Porphyromonas gingivalis [Text] / R. Mezyk-Kopec, M. Bzowska, J.Potempa [et al.] // Infection and Immunity. — 2005. — Vol. 73. — P. 1506–1514.344. Mikolajczyk-Pawlinska, J. Modulation of interleukin-8 activity by gingipainsfrom Porphyromonas gingivalis: implications for pathogenicity of periodontal disease[Text] / J. Mikolajczyk-Pawlinska, J.
Travis, J. Potempa // FEBS Letters. — 1998. —Vol. 440. — P. 282–286.345. Millhouse, E. Development of an in vitro periodontal biofilm model for assessingantimicrobial and host modulatory effects of bioactive molecules [Text] / E. Millhouse,A. Jose, L. Sherry [et al.] // BMC Oral Health. — 2014. — Vol. 14. — P. 80. —doi:10.1186/1472-6831-14-80.346. Miossec, P. Interleukin-17 and type 17 helper T cells [Text] / P. Miossec, T.Korn, V. K. Kuchroo // New England Journal of Medicine. — 2009. — Vol.
361. —P. 888–898.322347. Moller, D. E. Potential role of TNF-alpha in the pathogenesis of insulinresistance and type 2 diabetes. Trends Endocinol [Text] / D. E. Moller // Metabolism.— 2000. — Vol. 11. — P. 212–217.348. Mombelli, A. On the symmetry of periodontal disease [Text] / A. Mombelli, C.Meier // Journal of Clinical Periodontology. — 2001.
— Vol. 28. — P. 741–745.349. Montagner, F. Beta-lactamic resistance profiles in Porphyromonas, Prevotella,and Parvimonas species isolated from acute endodontic infections [Text] / F.Montagner, R. Castilho Jacinto, F. G. Correa Signoretti [et al.]Endodontics.——2014.Vol.40(3).—P.// Journal of339–344.—doi:10.1016/j.joen.2013.10.037.
Epub 2013 Dec 12.350. Mostefaoui, Y. Candida albicans and Streptococcus salivarius modulate IL-6,IL-8, and TNF-α expression and secre-tion by engineered human oralmucosa cells[Text] / Y. Mostefaoui, C. Bart, M. Frenette [et al.] // Cellular Microbiology. — 2004.— Vol. 6 (11). — P. 1085–1096.351. Moter, A.
Molecular epidemiology of oral treponemes in patients withperiodontitis and in periodontitis-resistant subjects [Text] / A. Moter, B. Riep, V.Haban [et al.] // Journal of Clinical Microbiology. — 2006. — Vol. 44 (9). — P. 3078–3085.352. Murakami, Y. Inhibitory effects of human salivary histatins and lysozyme oncoaggregation between Porphyromonas gingivalis and Streptococcus mitis [Text] / Y.Murakami, H. Nagata, A. Amano [et al.] // Infection and Immunity. — 1991. — Vol.59. — P.
3284–3286.353. Myneni, S. R. TLR2 signaling and Th2 responses drive Tannerella forsythiainduced periodontal bone loss [Text] / S. R. Myneni, R. P. Settem, T. D. Connell [Text]// Journal of Immunology. — 2011. — Vol. 187 (1). — P. 501–509.354. Mysak, J. Porphyromonas gingivalis: major periodonto-pathic pathogenoverview [Text] / J. Mysak, S. Podzimek, P. Sommerova [et al.] // Journal ofImmunologyResearch.doi:10.1155/2014/476068—2014.—Vol.2014.—476068.—323355. Navarro-San Francisco, M. Bacteraemia due to OXA-48-carbapenemaseproducing Enterobacteriaceae: a major clinical challenge [Text] / M.
Navarro-SanFrancisco, C. Mora-Rillo1, M. P. Romero-Gomez [et al.] // Clinical Microbiology andInfection. — 2012. — Vol. 19 (2). — E72-9. — doi:10.1111/1469-0691.12091356. Newman, M. G. Антимикробные препараты в стоматологической практике[Текст] / M. G. Newman, A. J. van Vinkelhoff. — М.: Азбука, 2001. — 328 с.357. Niederman, R. Infection-mediated early-onset periodontal disease in P/Eselectin-deficient mice [Text] / R. Niederman, T.
Westernoff, C. Lee [et al.] // Journalof Clinical Periodontology. — 2001. — Vol. 28. — P. 569–575.358. Nishimura, E. Oral streptococci exhibit diverse susceptibility to human betadefensin-2: antimicrobial effects of (hBD-2) on oral streptococci [Text] / E. Nishimura,A. Eto, M.
Kato [et al.] // Current Microbiology. — 2004. — Vol. 48. — P. 85–87.359. Nobbs, A. H. Streptococcus adherence and colonization [Text] / A. H. Nobbs,R. J. Lamont, H. F. Jenkinson // Microbiology and Molecular Biology Reviews. —2009. — Vol. 73. — P. 407–450.360. Novak, M. T. Biomimetic strategies based on viruses and bacteria for thedevelopment of immune evasive biomaterials [Text] / M.
T. Novak, J. D. Bryers, W.M. Reichert // Biomaterials. — 2009. — Vol. 30 (11). — P. 1989–2005.361. Nussbaum, G. How has neutrophil research improved our understanding ofperiodontal pathogenesis? [Text] / G. Nussbaum, L. Shapira // Journal of ClinicalPeriodontology. — 2011. — Vol. 38. — P. 49–59. — doi:10.1111/j.1600051X.2010.01678.x362.
Offenbacher, S. Changes in gingival crevicular fluid inflammatory mediatorlevels during the induction and resolution of experimental gingivitis in humans [Text]/ S. Offenbacher, S. Barros, L. Mendoza [et al.] // Journal of Clinical Periodontology.— 2010. — Vol. 37. — P.
324–333.363. Offenbacher, S. Gingival transcriptome patterns during induction and resolutionof experimental gingivitis in humans [Text] / S. Offenbacher, S. P. Barros, D. W.Paquette [et al.] // Journal of Periodontology. — 2009. — Vol. 80. — P. 1963–1982.324364. Okano, S. Proteomics-based analysis of a counter-oxidative stress system inPorphyromonas gingivalis [Text] / S.
Okano, Y. Shibata, T. Shiroza [et al.] //Proteomics. — 2006. — Vol. 6. — P. 251–258.365. Oringer, R. J. Modulation of the host response in periodontal therapy [Text] /R.J. Oringer // Journal of Periodontology. — 2002. — Vol. 73. — P.
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