Диссертация (Патофизиологическое значение ацетилхолина в пролиферации опухолевых клеток толстой кишки in vitro и in vivo), страница 21
Описание файла
Файл "Диссертация" внутри архива находится в папке "Патофизиологическое значение ацетилхолина в пролиферации опухолевых клеток толстой кишки in vitro и in vivo". PDF-файл из архива "Патофизиологическое значение ацетилхолина в пролиферации опухолевых клеток толстой кишки in vitro и in vivo", который расположен в категории "". Всё это находится в предмете "биология" из Аспирантура и докторантура, которые можно найти в файловом архиве РУДН. Не смотря на прямую связь этого архива с РУДН, его также можно найти и в других разделах. , а ещё этот архив представляет собой кандидатскую диссертацию, поэтому ещё представлен в разделе всех диссертаций на соискание учёной степени кандидата биологических наук.
Просмотр PDF-файла онлайн
Текст 21 страницы из PDF
1987. (47). C. 936–942.57. Cell P.B. [et al.]. Detection and Analysis of a Glucose 6-PhosphateDehydrogenase 1979. № 3 (63). C. 635–645.13458. Champaneria M.C. [et al.]. Friedrich Feyrter: A precise intellect in a diffusesystem // Neuroendocrinology. 2006. № 5–6 (83). C. 394–404.59. Cheng K. [et al.].
Acetylcholine release by human colon cancer cells mediatesautocrine stimulation of cell proliferation // AJP: Gastrointestinal and LiverPhysiology. 2008. № 3 (295). C. G591–G597.60. Cheng K. [et al.]. Acetylcholine release by human colon cancer cells mediatesautocrine stimulation of cell proliferation 2008. (21201). C. 591–597.61. Cheng K., Raufman J.-P. Bile acid-induced proliferation of a human coloncancer cell line is mediated by transactivation of epidermal growth factor receptors// Biochemical Pharmacology. 2005. № 7 (70). C.
1035–1047.62. Cheng K., Xie G., Raufman J.-P. Matrix metalloproteinase-7-catalyzed releaseof HB-EGF mediates deoxycholyltaurine-induced proliferation of a human coloncancer cell line // Biochemical Pharmacology. 2007. № 7 (73). C. 1001–1012.63. Chiba K., Kawakami K., Tohyama K. Simultaneous evaluation of cell viabilityby neutral red, MTT and crystal violet staining assays of the same cells //Toxicology in Vitro. 1998.
№ 3 (12). C. 251–258.64. Christie M. [et al.]. Different APC genotypes in proximal and distal sporadiccolorectal cancers suggest distinct WNT/β-catenin signalling thresholds fortumourigenesis // Oncogene. 2013. № 39 (32). C. 4675–4682.65. Clark I. [et al.]. The regulation of matrix metalloproteinases and their inhibitors// The International Journal of Biochemistry & Cell Biology. 2008.
№ 6–7 (40). C.1362–1378.66. Cooke J.P. Angiogenesis and the role of the endothelial nicotinic acetylcholinereceptor // Life Sciences. 2007. № 24–25 (80). C. 2347–2351.67. Corpet D.E. [et al.]. Most Effective Colon Cancer Chemopreventive Agents inRats: A Systematic Review of Aberrant Crypt Foci and Tumor Data , 2009. №October 2014. C. 37–41.68. Corpet D.E., Pierre F.
Point: From Animal Models to Prevention of ColonCancer . Systematic Review of Chemoprevention in Min Mice and Choice of theModel System // Cancer Epidemiology Biomarkers & Prevention. 2003. № May135(12). C. 391–400.69. Corpet D.E., Pierre F. How good are rodent models of carcinogenesis inpredicting efficacy in humans? A systematic review and meta-analysis of colonchemoprevention in rats , mice and men 2005. (41). C. 1911–1922.70.
Cottrell S. [et al.]. Molecular analysis of APC mutations in familialadenomatous polyposis and sporadic colon carcinomas. // Lancet. 1992. № 8820(340). C. 626–30.71. Cucina A. [et al.]. Nicotine stimulates proliferation and inhibits apoptosis incolon cancer cell lines through activation of survival pathways // Journal of SurgicalResearch. 2012. № 1 (178). C. 233–241.72. Cusick M.F., Libbey J.E., Fujinami R.S. Molecular Mimicry as a Mechanism ofAutoimmune Disease // Clinical Reviews in Allergy & Immunology. 2012.
№ 1(42). C. 102–111.73. Day F.L. [et al.]. PIK3CA and PTEN Gene and Exon Mutation-SpecificClinicopathologic and Molecular Associations in Colorectal Cancer // ClinicalCancer Research. 2013. № 12 (19). C. 3285–3296.74. Decock J. [et al.]. Matrix metalloproteinases: protective roles in cancer // Journalof Cellular and Molecular Medicine. 2011. № 6 (15).
C. 1254–1265.75. Delbro D.S. Do neuro-humoral signaling molecules participate in colorectalcarcinogenesis/cancer progression? // Neurogastroenterology & Motility. 2012. № 2(24). C. 96–99.76. Deloose E. [et al.]. The migrating motor complex: control mechanisms and itsrole in health and disease // Nature Reviews Gastroenterology & Hepatology. 2012.№ 5 (9).
C. 271–285.77. Duffy M.J. [et al.]. Tumor markers in colorectal cancer, gastric cancer andgastrointestinal stromal cancers: European group on tumor markers 2014 guidelinesupdate // International Journal of Cancer. 2014. № 11 (134). C. 2513–2522.78. Dufour A., Overall C.M. Missing the target: matrix metalloproteinase antitargetsin inflammation and cancer // Trends in Pharmacological Sciences. 2013.
№ 4 (34).C. 233–242.13679. Dunn G.P. [et al.]. Cancer immunoediting: from immunosurveillance to tumorescape. // Nature immunology. 2002. № 11 (3). C. 991–998.80. Edmondson J.M., Armstrong L.S., Martinez A.O. A rapid and simple MTTbased spectrophotometric assay for determining drug sensitivity in monolayercultures // Journal of Tissue Culture Methods. 1988. № 1 (11). C.
15–17.81. Egleton R.D., Brown K.C., Dasgupta P. Nicotinic acetylcholine receptors incancer: multiple roles in proliferation and inhibition of apoptosis // Trends inPharmacological Sciences. 2008. № 3 (29). C. 151–158.82. Español A.J. [et al.]. Role of non-neuronal cholinergic system in breast cancerprogression // Life Sciences. 2007. № 24–25 (80). C. 2281–2285.83.
Fiala E.S. Investigation into the metabolism and mode of action of the coloncarcinogen 1,2-dimethylhidrazine // Cancer. 1975. (36). C. 2407–2412.84. Friedman J.M. Leptin at 14 y of age: An ongoing story // American Journal ofClinical Nutrition. 2009. № 3 (89). C. 973–979.85. Frucht H. [et al.]. Human Colon Cancer Cell Proliferation Mediated by the M 3Muscarinic Cholinergic Receptor 1 // Clinical Cancer Research. 1999.
№ September(5). C. 2532–2539.86. Gahring L.C., Rogers S.W. Neuronal nicotinic acetylcholine receptor expressionand function on nonneuronal cells. // The AAPS journal. 2005. № 4 (7). C. E885–E894.87. Gialeli C., Theocharisa. D., Karamanos N.K. Roles of matrixmetalloproteinases in cancer progression and their pharmacological targeting // TheFEBS Journal. 2011. № 1 (278).
C. 16–27.88. Guizzetti M. [et al.]. Acetylcholine as a mitogen: Muscarinic receptor-mediatedproliferation of rat astrocytes and human astrocytoma cells // European Journal ofPharmacology. 1996. № 3 (297). C. 265–273.89. Gulubova M., Vlaykova T. Chromogranin A-, serotonin-, synaptophysin- andvascular endothelial growth factor-positive endocrine cells and the prognosis ofcolorectal cancer: An immunohistochemical and ultrastructural study // Journal ofGastroenterology and Hepatology. 2008. № 10 (23). C.
1574–1585.13790. Gunawardene A.R., Corfe B.M., Staton C. a. Classification and functions ofenteroendocrine cells of the lower gastrointestinal tract // International Journal ofExperimental Pathology. 2011. № 4 (92). C. 219–231.91. Guo Y., Lu N., Bai A. Treatment of inflammatory bowel disease with neuralstem cells expressing choline acetyltransferase // Medical Hypotheses. 2012. № 5(79). C.
627–629.92. Gupta G.P., Massagué J. Cancer Metastasis: Building a Framework // Cell.2006. № 4 (127). C. 679–695.93. Halbersztadt A. [et al.]. The role of matrix metalloproteinases in tumor invasionand metastasis // Ginekologia polska. 2006. № 1 (77). C. 63–71.94. Halpern B., Amache N. Diagnosis of drug allergy in vitro with the lymphocytetransformation test // J Allergy.
1967. № 3 (40). C. 168–181.95. Holla V.R. [et al.]. Regulation of prostaglandin transporters in colorectalneoplasia // Cancer Prevention Research. 2008. № 2 (1). C. 93–99.96. Humphries A. [et al.]. OC-016 Human colorectal adenoma growth ischaracterised by periods of quiescence and rapid clonal expansion // Gut.
2012. №Suppl 2 (61). C. A7–A8.97. Hylemon P.B. [et al.]. Bile acids as regulatory molecules // The Journal of LipidResearch. 2009. № 8 (50). C. 1509–1520.98. Improgo M.R. [et al.]. Nicotinic acetylcholine receptors mediate lung cancergrowth 2013. № September (4). C. 1–6.99.
Ishiyama M. [et al.]. A combined assay of cell viability and in vitro cytotoxicitywith a highly water-soluble tetrazolium salt, neutral red and crystal violet. //Biological & pharmaceutical bulletin. 1996. № 11 (19). C. 1518–1520.100. Itagaki H. [et al.]. An in vitro alternative to the Draize eye-irritation test:Evaluation of the crystal violet staining method. // Toxicology in vitro : aninternational journal published in association with BIBRA. 1991.
№ 2 (5). C. 139–143.101. Jeffery N. [et al.]. The matrix metalloproteinase/tissue inhibitor of matrixmetalloproteinase profile in colorectal polyp cancers // Histopathology. 2009. № 7138(54). C. 820–828.102. Jones S. Mini-review: Endocrine actions of fibroblast growth factor 19 //Molecular Pharmaceutics. 2008.
№ 1 (5). C. 42–48.103. Keely S., Talley N.J., Hansbro P.M. Pulmonary-intestinal cross-talk in mucosalinflammatory disease // Mucosal Immunology. 2012. № 1 (5). C. 7–18.104. Kim M.S., Lee J., Sidransky D. DNA methylation markers in colorectal cancer// Cancer and Metastasis Reviews. 2010. № 1 (29). C. 181–206.105. Klapproth H. [et al.]. Non-neuronal acetylcholine, a signalling moleculesynthezised by surface cells of rat and man // Naunyn-Schmiedeberg’s Archives ofPharmacology.
1997. № 4 (355). C. 515–523.106. Koshimizu H. [et al.]. Adenomatous polyposis coli heterozygous knockoutmice display hypoactivity and age-dependent working memory deficits // Frontiersin Behavioral Neuroscience. 2011. № December (5). C. 1–10.107. Kouji H. [et al.].