Диссертация (1174262), страница 21
Текст из файла (страница 21)
I. Wallhagen, E.S. Froelicher // J Adv Nurs. - 2008. - Vol.61. - P. 326-335.146. Lynch, A.I. Pharmacogenetic association of the NPPA T2238C geneticvariant with cardiovascular disease outcomes in patients with hypertension / A.I.Lynch, E. Boerwinkle, B.R. Davis // JAMA. - 2008. - Vol. 299(3). - P.296-307.147. Ma, F. The Association of Sport Performance with ACE and ACTN3Genetic Polymorphisms: A Systematic Review and Meta-Analysis / F.
Ma, Y.Yang, X. Li // PLoS One. - 2013. - Vol. 8(1). - P.54685.148. Mahmood, M.S. G protein beta 3 subunit gene 825CT dimorphism isassociated with left ventricular hypertrophy but not essential hypertension / M.S.Mahmood[et al.] // Med Sei Monit. - 2004. - Vol. 22(l). - P. 6 - 9.149. Major cardiovascular events in hypertensive patients randomized todoxazosin vs chlorthalidone:theAntihypertensiveandLipid-Lowering Treatment to Prevent Heart Attack Trial (ALLHAT): ALLHATCollaborative Research Group. JAMA.
- 2000. - Vol. 283. - P.1967-1975.150. Martin, M.M. The human angiotensin II type 1 receptor +1166 A/Cpolymorphism attenuates microrna - 155 binding // J Biol Chem. - 2007. - Vol.282(33). - P.24-29.151. Martinez, E. Threonines at position 174 and 235 of the angiotensinogenpolypeptide chain are related to familial history of hypertension in a Spanish-130Mediterranean population / E. Martinez, A. Puras, J. Escribano // Br J BiomedSci.
- 2002. - Vol. 59(2). - P.95-100.152. Matthew, R.W. Influence of race and dietary salt on the antihypertensiveefficacy of an angiotensin-converting enzyme inhibitor or a calcium channelantagonist in salt-sensitive hypertensives / R.W. Matthew, C.G. Steven //Hypertension. - 1998. - Vol. 3. - P. 1088-1096.153. Mingyu, L. Renal medullary genes in salt-sensitive hypertension: achromosomal substitution and cDNA microarray study / L. Mingyu, B. Yuan, E.Rute // Physiol Genomics. - 2002. - Vol.
8. - P.139-149.154. Nazzaro,P.Insulinresistanceinessentialhypertension:apsychophysiological approach to the «chicken and egg» question/ P. Nazzaro, R.Triggiani [et al.]// Nutr. Metab. Cardiovasc. Dis. - 2000. - Vol. 10(5). - P.75-86.155. Neal, B. Effects of ACE inhibitors, calcium antagonists, and otherbloodpressurelowering drugs / B. Neal, S. MacMahor, N. Chapman // Lancet.2000. - Vol.
356. - P.1955-1964.156. Ned, R.M. The ACE I/D polymorphism in US adults: limited evidence ofassociation with hypertension-related traits and sex-specifi c effects byrace/ethnicity / R.M. Ned, A. Yesupriya, G. Imperatore // Am J Hypertens. —2012. - Vol. . 25(2). - P. 209-215.157. Nestel, P.J. Enhanced blood pressure response to dietary salt in elderlywomen, especially those with small waist/hip ratio / P.J. Nestel, P.M. Clifton,M. Noakes // J. Hypertension. - 1993. - Vol.
11. - P.1387-1395.158. Overlack, A. Divergent hemodynamic and hormonal responses to varyingsalt intake in normotensive subjects / A. Overlack, M. Ruppert, R. Kolloch //Hypertension. - 1993. - Vol. 22. - P.331-338.159. Osterop, A.P. AT1 receptor A/C1166 polymorphism contributes tocardiac hypertrophy in subjects with hypertrophic cardiomyopathy //Hypertension. - 1998. - Vol.
32(5). - P. 825-830.131160. Paillard, F. Genotype - phenotype relationships for the rennin –angiotensin - aldosterone system in a normal population // Hypertension. - 1999.- Vol. 34(3). - P.423-429.161. Park, S. Association of central hemodynamics with estimated 24 - hurinary sodium in patients with hypertension / S. Park, J. Park, E. Lakatta // JHypertens.- 2011. - Vol. 29. - P.1502-1509.162. Pamies – Andreu, E. Renin-angiotensin-aldosterone system and G-proteinbeta-3 subunit gene polymorphisms in salt-sensitive essential hypertension / E.Pamies - Andreu, R. Ramirez - Lorca, P.
Stiefel // J. Hum. Hypertension. –2003. - Vol. 17(3). - P.187-191.163. Pilbrow, A.P. Angiotensinogen M235T and T174M gene polymorphismsin combination doubles the risk of mortality in heart failure. Hypertension. 2007. - Vol.49(2). - P.322-327.164. Pitt, B. Effect of amlodipine on the progression of atherosclerosis and theoccurrence of clinical events. PREVENT Investigators / B. Pitt, R.P.
Byington,C.D. Furberg // Circulation. - 2000 Sep 26. - Vol. 102(13). - P.1503 - 1510.165. Poch, E.Molecular basis of salt sensitivity in human hypertension.Evaluation of renin - angiotensin - aldosterone system gene polymorphisms //Hypertension. – 2001. - Vol. 38(5). - P. 1204 - 1209.166. Pontremoli, R.
Genetic Polymorphism of the Renin - Angiotensin Systemand Organ Damage in Essential Hypertension / R. Pontremoli, M. Ravera, F.Viazzi // Kidney Int. – 2000. - Vol. 57( 2). P. 561 – 569.167. PROGRESS Collaborative Group. Randomised trial of a perindopril based blood – pressure - lowering regimen among 6105 individuals withprevious stroke or transient ischaemic attack // Lancet. - 2001. - Vol.358. –-P.1033 - 1041.168.
Psaty, B.M. Diuretic therapy, the alpha - adducin gene variant, and therisk of myocardial infarction or stroke in persons with treated hypertension /132B.M. Psaty, N.L. Smith, S.R. Heckbert // JAMA. - 2002. - Vol. 287(13). P.1680 - 1689.169. Resnick, L. M. Calciotropic hormones in salt-sensitive essentialhypertension: 1,25 dihydroxyvitamin D and parathyroid hypertensive factor // J.H ypertens.-1994.- Vol. 12 (suppl.
1).- Р. 1- 9.170. Resnick, L.M. Intracellular ionic consequences of dietary salt loading inessential hypertension // J. Clin. Invest. - 1994.- Vol. 94.- P. 1269-1276.171. Riddle, E.L. Polymorphisms and Haplotypes of the Regulator of GProtein Signaling - 2 Gene in Normotensives and Hypertensives / E.L. Riddle,B.K. Rana, K.K. Murthy // Hypertension. - 2006. - Vol. 47. - P. 415 - 420.172. Rosei, E.A. Hypertension, atherosclerosis and LVH in ELSA // BloodPressure. - 1996. - Vol. 5 (Suppl 4). - P. 44 - 47.173.
Rotimi, C. Polymorphisms of rennin - angiotensin genes amongNigerians, Jamaicans, and African Americans / C. Rotimi, A. Puras, R. Cooper// Hypertension. - 1996. - Vol.27. - P. 558 - 563.174. Saab, Y.B. Renin - angiotensin - system gene polymorphisms anddepression // Prog Neuropsychopharmacol Biol Psychiatry. - 2007. - Vol. 31(5):P. 1113 - 1118.175. Sacks, F. Effects on blood pressure of reduced dietary sodium and theDietary Approaches to Stop Hypertension diet.
DASH Sodium CollaborativeResearch Group / F. Sacks, L. Svetkey, W. Vollmer // New Eng J Med. – 2001. Vol.344(1). - P. 3 - 10.176. Salvi, E. Target sequencing, cell experiments, and a population studyestablish endothelial nitric oxide synthase (eNOS) gene as hypertensionsusceptibility gene / E. Salvi, T. Kuznetsova // Hypertension. - 2013. - Vol. .62(5). - P.844-852.177.Sasaki, M. Relationship between the angiotensin converting enzyme genepolymorphism and the effects of enalapril on left ventricular hypertrophy andimpaired diastolic filling in essential hypertension: M - mode and pulsed133Doppler echocardiographic studies / M.
Sasaki, T. Oki, A. Iuchi // Hypertens. –1996. - Vol.14. - P. 1403 -1408.178. Sethi, A.A. Angiotensinogen single nucleotide polymorphisms, elevatedblood pressure, and risk of cardiovascular disease // Hypertens. -2003. -Vol.41(6). - Р. 1202 - 1211.179. Sethi, A.A. Angiotensinogen polymorphisms and elevated blood pressurein the general population: the Copenhagen City Heart Study // Hypertension. 2001. - Vol.37 (3).
- Р. 875 - 81.180. Sethi, A.A. Angiotensinogen mutations and risk for ischemic heartdisease, myocardial infarction, and ischemic cerebrovascular disease. Six case control studies from the Copenhagen City Heart Study // Ann Intern Med. 2001. - Vol.134 (10). - Р. 941 - 54.181. Sham, P.C. Monte Carlo tests for associations between disease and allelesat highly polymorphic loci / P.C. Sham, D.
Curtis // Ann Hum Genet. - 1995. Vol. (1). - Р. 97 - 105.182. Shcherbak,N.S. The C825T polymorphism in the G-protein beta 3subunit gene and diabetic complications in IDDM patients / N.S. Shcherbak, E.I.Schwartz // J Hum Genet. - 2001. - Vol. 46.4.- P.188 - 191.183. Schwartz, G.L. Pharmacogenetics of Antihypertensive Drug Responses/ G.L.
Schwartz, S.T. Turner // Pharmacogenomics. - 2004. – Vol.4 (3). –P.151-160.184. Sethupathy, P. Human microRNA - 155 on chromosome 21 differentiallyinteracts with its polymorphic target in the AGTR1 3-prime untranslated region:a mechanism for functional single-nucleotide polymorphisms related tophenotypes / P. Sethupathy, C. Borel, M. Gagnebin // Am. J. Hum. - 2007. -Vol.81. - P.405 - 413.185.
Singh, D. Gender Specific Association of RAS Gene Polymorphism withEssential Hypertension: A Case-Control Study / D. Singh, A. Jajodia, H. Kaur //BioMed Research International. - 2014. - Vol. 2014. - Р. 1-10.134186. Stamler, J. Findings of the international cooperative INTERSALT study /J. Stamler, G. Rose, P. Elliot // Hypertension.















