Диссертация (1154347), страница 33
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— 2013. — Vol. 242. — P. 1099-1103.190. Epidemiology of stone disease across the world / Sorokin I., Mamoulakis C.,Miyazawa K. [et al.] // World J. Urol. — 2017. — Vol. 35. — № 9. — P. 1301-1320.191. Epidemiology of urolithiasis for improving clinical practic / Yasui T., AndoR., Okada A. [et al.] // Hinyokika Kiyo. — 2012. — Vol. 58. — № 12. — P.697-701.192. Epidemiology of urolithiasis with emphasis on ultrasound detection: aretrospective analysis of 5371 cases in Saudi Arabia / Ahmad F., Nada M.O., Farid A.B.[et al.] // Saudi J.
Kidney Dis. Transpl. — 2015. — Vol. 26. — Р. 386-391.193. Etiologic and Epidemiologic Pattern of Urolithiasis in North Iran; Review of10-Year Findings / Mohammadjafari H., Barzin M., Salehifar E. [et al.] // Iran. J.Pediatr. — 2014. — Vol. 24. — P. 69–74.194. Evan, A.P. Renal Stone Disease: A Commentary on the Nature andSignificance of Randall's Plaque / Evan A.P., Unwin R.J., Williams Jr. J.C. // Nephron.Physiol. — 2011. — Vol. 119. — P. 49–53.195. Everything you need to know about distal renal tubular acidosis inautoimmune disease / Both T., Zietse R., Hoorn E.J. [et al.] // Rheumatol. Int. — 2014. —Vol. 34.
— P. 1037–1045.196. Extensive characterizations of bacteria isolated from catheterized urine andstone matrices in patients with nephrolithiasis / Tavichakorntrakool R., PrasongwattanaV., Sungkeeree S. [et al.] // Nephrol. Dial. Transplant. — 2012. — Vol. 27. — № 11. —P. 4125-4130.197. Extracorporeal shock wave lithotripsy (ESWL) versus percutaneousnephrolithotomy (PCNL) or retrograde intrarenal surgery (RIRS) for kidney stones /Srisubat A., Potisat S., Lojanapiwat B.
[et al.] // Cochrane Database Syst. Rev. —2142014. — Vol. 24. — N 11.198. Extracorporeal shock wave lithotripsy: An opinion on its future /Rassweiler J., Rassweiler M.-C., Frede Th., Alken P. // Indian J. Urol. — 2014. —Vol. 30. — P. 3–79.199. Extracorporealshockwavelithotripsywithoutradiation:Ultrasoundlocalization is as effective as fluoroscopy / Smith H.E., Bryant D.A., KooNg J.
[et al.] //Urol. Ann. — 2016. — Vol. 8. — P. 454–457.200. Formation of struvite urinary stones and approaches towards the inhibition-Areview / Das P., Gupta G., Velu V. [et al.] // Biomed. Pharmacother. — 2017. —Vol. 96. — P. 361-370.201. Fourier transform infrared spectroscopy for analysis of kidney stones / HabibKhan A., Imran Sh., Talati J., Jafri L. // Investig. Clin. Urol. — 2018.
— Vol. 59. —P. 32–37.202. Galen, R.S. Beyond Normality: The Predictive Value and Efficiency ofMedical Diagnosis / Galen R.S., Gambino S.R. // A Wiley Biomedical Publication —1975.203. Griner, P.F. Selection and interpretation of diagnostic tests and procedures /Griner P.F., Mayewski R.J., Mushlin A.I. [et al.] // Ann. Intern. Med. — 1981. — 94(4,Part 2): 553-600.204. Guimarães Penido, M.G. Pediatric primary urolithiasis: Symptoms, medicalmanagement and prevention strategies / Guimarães Penido M.G, Tavares M. // World J.Nephrol.
— 2015. — Vol. 4. — P. 444–454.205. Gul, Z. Medical and Dietary Therapy for Kidney Stone Prevention / Gul Z.,Monga M. // Korean J. Urol. — 2014. — Vol. 55. — P. 775–779.206. Hamamoto, S. Molecular mechanism of renal stone formation / Hamamoto S.,Taguchi K., Fujii Y. // Clin. Calcium. — 2011. — Vol. 21. — P. 1481-1487.207. Hereditary causes of kidney stones and chronic kidney disease /Edvardsson V.O., Goldfarb D.S., Lieske J.C. [et al.] // Pediatr. Nephrol.
— 2013. —Vol. 28. — P. 1923-1942.208. High-dose supplementation with vitamin C--induced pediatric urolithiasis: the215first case report in a child and literature review / Chen X., Shen L., Gu X. [et al.] //Urology. — 2014. — Vol. 84. — P. 922-924.209. Holmes, R.P. Lowering urinary oxalate excretion to decrease calcium oxalatestone disease / Holmes R.P., Knight J., Assimos D.G. // Urolithiasis. — 2016.
— Vol. 44.— P. 27–32.210. Incidence and Risk Factors for Urolithiasis in Patients with Crohn's Disease /Kim M.J., Woo S.Y., Kim E.R. [et al.] // Urol. Int. — 2015. — Vol. 95. — P. 314.211. Individualized treatment strategies for hyperuricemia informed by a semi‐mechanistic exposure‐response model of uric acid dynamics / Aksenov S., Peck C.C.,Eriksson U.G., Stanski D.R.
// Physiol. Rep. — 2018. — Vol. 6.212. Induced urinary crystal formation as an analytical strategy for the predictionand monitoring of urolithiasis and other metabolism-related disorders / Laube N., BergW., Bernsmann F. [et al.] // EPMA J. — 2014. — Vol. 5. — P. 13.213. Julka, S. Protocol-based metabolic evaluation in high-risk patients with renalstones in North India / Julka S., Gupta S.K., Srivastava A. / Indian J. Endocrinol. Metab.— 2012. — Vol. 16. — P. 283–287.214. Khan, S.R.
A Unified Theory on the Pathogenesis of Randall’s Plaques andPlugs / Khan S.R., Canales B.K. // Urolithiasis. — 2015. — Vol. 43. — P. 109–123.215. Kidney stone analysis: “Give me your stone, I will tell you who you are!” /Cloutier J., Villa L., Traxer O., Daudon M. // World J. Urol. — 2015. — Vol. 33. —P. 157–169.216. Kidney stones / Khan S.R., Pearle M.S., Robertson W.G. [et al.] // Nat.
Rev.Dis. Primers. — 2016. — Vol. 2.217. Knoll, T. Epidemiology, Pathogenesis, and Pathophysiology of Urolithiasis /Knoll T. // Eur. Urol. — 2010. — Vol. 9. — P. 802–806.218. Krishna Reddy, S.V. Effect of Potassium Magnesium Citrate and Vitamin B6 Prophylaxis for Recurrent and Multiple Calcium Oxalate and Phosphate Urolithiasis /Krishna Reddy S.V., Basha Shaik A., Bokkisam S.
// Korean J. Urol. — 2014. — Vol.55. — P. 411–416.219. Kurtz, I. Molecular Mechanisms and Regulation of Urinary Acidification /216Kurtz I. // Compr. Physiol. — 2014. — Vol. 4. — P. 1737–1774.220. Long-term prescription of α-blockers decrease the risk of recurrent urolithiasisneeded for surgical intervention-a nationwide population-based study / Liu C.C., HsiehH.M., Wu C.F. [et al.] // PLoS One. — 2015. — Vol.
10.221. Management of large renal stones: laparoscopic pyelolithotomy versuspercutaneous nephrolithotomy / Bai Y., Tang Y., Deng L. [et al.] // BMC Urol. —2017. — Vol.31. — P. 75.222. Marked increase in urinary excretion of apolipoproteins in children withnephrolithiasis associated with hypercalciuria / Kovacevic L., Lu H., Caruso J.A. [et al.] //Pediatr. Nephrol. — 2017. — Vol. 32. — P. 1029-1033.223.
Mechanisms of human kidney stone formation / Evan A.P., Worcester E.M.,Coe F.L. [et al.] // Urolithiasis. — 2015 — Vol. 43. — P. 19–32.224. Metabolic disorders in Turkish children with urolithiasis / Celiksoy M.H.,Yilmaz A., Aydogan G. [et al.] // Urology. — 2015.
—Vol. 85. — P. 909-913.225. Metabolic evaluation and recurrence prevention for urinary stone patients:EAU guidelines / Skolarikos A., Straub M., Knoll T. [et al.] // Eur. Urol. — 2015. —Vol.67. — Vol. 4. — P. 750-763.226. Metabolic evaluation of children with urolithiasis / Gouru V.R., Pogula V.R.,S.P. Vaddi [et al.] // Urol. Ann. — 2018. — Vol. 10. — P. 94–99.227. Microbial communities on kidney stones / Romanova Y.M., Mulabaev N.S.,Tolordava E.R.
[et al.] // Mol. Gen. Mikrobiol. Virusol. — 2015. — Vol. 33. — P. 20-25.228. Micropercutaneous nephrolithotripsy: initial experience / Ölçücüoğlu E.,Kasap Y., Ölçücüoğlu Es. [et al.] // Wideochir. Inne Tech. Maloinwazyjne. — 2015. —Vol. 10. — P. 368–372.229. Mineralogical composition of urinary stones, risk factors and metabolicdisturbances in patients with calcium-oxalate urolithiasis / Kustov A.V., Strelnikov A.I.,Moryganov M.A. [et al.] // Urologiia. — 2017. — Vol 4.
— P. 22-26.230. Modular training for percutaneous nephrolithotripsy: The safe way to go /Kallidonis P., Kyriazis I., Vasilas M. [et al.] // Arab. J. Urol. — 2015. — Vol. 13. —P. 270–276.217231. Morgan, M.S. Medical management of renal stones / Morgan M.S., PearleM.S.
// BMJ. — 2016. — Vol. 352.232. Nazzal, L. Enteric hyperoxaluria: an important cause of end-stage kidneydisease / Nazzal L., Puri S., Goldfarb D.S. // Nephrol. Dial. Transplant. — 2016. — Vol.3. — P. 375-382.233. Nephrolithiasis: Molecular Mechanism of Renal Stone Formation and theCritical Role Played by Modulators / Aggarwal K.P., Narula Sh., Kakkar M.,Tandon Ch. // Biomed. Res. Int.
— 2013.234. Nephropathy in dietary hyperoxaluria: A potentially preventable acute orchronic kidney disease / Glew R.H., Sun Y., Horowitz B.L. [et al.] // World J. Nephrol. —2014. — Vol. 3. — P. 122–142.235. New Insights Into the Pathogenesis of Renal Calculi / Bagga H.S., Chi Th.,Miller J., Stoller M.L. // Urol. Clin. North Am. — 2013.
— Vol. 40. — P. 1–12.236. Nutritional Management of Kidney Stones (Nephrolithiasis) / Han H., SegalA.M., Seifter J.L., Dwyer J.T. // Clin. Nutr. Res. — 2015. — Vol. 4. — P. 137–152.237. Oral Consumption of Cranberry Juice Cocktail Inhibits Molecular-ScaleAdhesion of Clinical Uropathogenic Escherichia coli / Tao Y., Pinzón-Arango P.A.,Howell A.B., Camesano T.A. // Med. Food. — 2011. — Vol. 14. — № 7-8. — P. 739–745.238. Phosphaturia as a Promising Predictor of Recurrent Stone Formation inPatients with Urolithiasis / Ha Y-S., Tchey D-U., Kang H.W.
[et al.] // Korean J. Urol. —2010. — Vol. 51. — P. 54–59.239. Prado de Oliveira, E. High plasma uric acid concentration: causes andconsequences / Prado de Oliveira E., Carlos Burini R. // Diabetol. Metab. Syndr. — 2012.— Vol. 4. — P. 12.240. Preventive fluid and dietary therapy for urolithiasis: An appraisal of strength,controversies and lacunae of current literature / Agarwal M.M., Singh Sh.K.,Mavuduru R., Mandal A.K. // Indian J. Urol. — 2011. — Vol. 27.
— Р. 310–319.241. Pseudomonas aeruginosa as an Etiologic Agent of Nephrolithiasis in DeepWater Divers / Bird V.Y., Chastain-Gross R., Sutkowski R. [et al.] // J. Endourol. Case.218Rep. — 2017. — Vol. 3. — P. 4–6.242. Ratkalkar, V.N. Mechanisms of Stone Formation / Ratkalkar V.N.,Kleinman J.G. // Clin. Rev.
Bone Miner. Metab. — 2011. — Vol. 3-4. —P. 187–197.243. Recurrentepisodesofbrushitenephrolithiasisrevealingprimaryhyperparathyroidism / Simon I., Roumeguère T., Devuyst F. [et al.] // Rev. Med. Brux. —2015. — Vol. 36. — P. 172-176.244. Recurrent exercise-induced acute renal failure in a young Pakistani man withsevere renal hypouricemia and SLC2A9 compound heterozygosity / Jeannin G., ChiarelliN., Gaggiotti M.