Диссертация (1154338), страница 40
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46, № 6. – P. 731-739.423. Janssen, L. J. Calcium Homeostasis and Ionic Mechanisms in PulmonaryFibroblasts / L. J. Janssen, S. Mukherjee, K. Ask // Am. J. Respir. Cell Mol. Biolog. –2015. – Vol. 53, № 2. – P. 135-148.424. Jimenez, S. A.
Role of endothelial to mesenchymal transition in thepathogenesis of the vascular alterations in systemic sclerosis [Electronic resource] //ISRN rheumatol. – 2013. – DOI: 10.1155/2013/835948.425. Jing, C. Double-edged effects of neuropeptide substance P on repair ofcutaneous trauma / C. Jing, W. J ia-Han, Z.
Hong-Xing // Wound Repair Regen. – 2010.– Vol. 18, № 3. – P. 319-324.426. Juckett, G. Management of keloids and hypertrophic scars / G. Juckett, H.Hartman-Adams // Am. Fam. Physician. – 2009. – Vol. 80, № 3. – P. 253-260.248427. Kaneko, Y. Transient receptor potential (TRP) channels: a clinicalperspective / Y. Kaneko, A. Szallasi // Br. J. Pharmacol. – 2014.
– Vol. 171. – № 10. –С. 2474-2507.428. Karin, M. NF-κB at the cross roads of life and death / M. Karin, A. Lin //Nat. Immunol. – 2002. – Vol. 3, № 3. – P. 221–227.429. Keloid‐derived, plasma/fibrin‐based skin equivalents generate de novodermal and epidermal pathology of keloid fibrosis in a mouse model / Y. S. Lee [et al.]// Wound Repair Regen.
– 2016. – Vol. 24, № 2. – P. 302-316.430. Ki-67 proliferation index but not mitotic thresholds integrates themolecular prognostic stratification of lower grade gliomas / E. Duregon [et al.] //Oncotarget. – 2016. – Vol. 7, № 16. – P. 21190-8.431. Kim, M. The enhanced anti-tissue adhesive effect of injectable pluronicHA hydrogel by poly (γ-glutamic acid) / M.Kim, Y. Hwang, G. Tae // Int. J. Biol.lMacromol. – 2016. – Vol. 93, (pt. B). – P. 1603-1611.432. Klingberg, F.
The myofibroblast matrix: implications for tissue repair andfibrosis / F. Klingberg, B. Hinz, E. S. White // J. Pathol. – 2013. – Vol. 229, № 2. – P.298-309.433. Kong, P. The pathogenesis of cardiac fibrosis / P. Kong, P. Christia, N. G.Frangogiannis // Cell. Mol. Life Sci. – 2014. – Vol. 71, № 4. – P. 549-574.434. Krämer, B. Erlanger OP-Workshop Adhäsionen: Was können wir tun, wasmüssen wir tun? / B. Krämer // Geburtshilfe und Frauenheilkunde.
– 2013. – Bd. 73, №1. – S. R3-R22.435. Kubota S. Cellular and molecular actions of CCN2/CTGF and its roleunder physiological and pathological conditions / S. Kubota, M. Takigawa // Clin. sci. –2015. – Vol. 128, № 3. – P. 181-196.436. Kwan, P.O. Biological principles of scar and contracture/ P.O. Kwan, E.E.Tredget // Hand Clin. – 2017.
– Vol. 33, № 2. – P. 277-292.437. Lee, J. H. Effects of interferon-alpha2b on keloid treatment withtriamcinolone acetonide intralesional injection / J. H. Lee, S. E. Kim, A. Y. Lee // Int J.Dermatol. – 2008. – Vol. 47, № 2. – P. 183-186.249438. Lee, R. C. The response of burn scars to intralesional verapamil / R. C.Lee, H. Doong, A. F. Jellema // Arch. Surg. – 1994. – Vol. 129. – P. 107.439.
Lee, Y. A. Pathobiology of liver fibrosis: a translational success story / Y.A. Lee, M. C. Wallace, S. L. Friedman // Gut. – 2015. – Vol. 64, № 5. – P. 830-841.440. Lenna, S. The role of endoplasmic reticulum stress and the unfoldedprotein response in fibrosis / S. Lenna, M. Trojanowska // Curr. Opin. Rheumatol. –2012.
– Vol. 24, № 6. – P. 663-668.441. Levine, T. B. Inflammation / T. B. Levine, A. B. Levine // Metabolicsyndrome and cardiovascular disease. – 2nd ed. – 2012. – P. 192-227.442. Li, M. Functional analysis of keratinocyte and fibroblast gene expression inskin and keloid scar tissue based on deviation analysis of dynamic capabilities / M.
Li,L. Wu // Exp. Ther. Med. – 2016. – Vol. 12, № 6. – P. 3633-3641.443. Libby, P. Inflammation in atherosclerosis / P. Libby // Arteriosclerosis,thrombosis, and vascular biology. – 2012. – Vol. 32, № 9. – P. 2045-2051.444. Lieber, R. L. Cellular mechanisms of tissue fibrosis. 4. Structural andfunctional consequences of skeletal muscle fibrosis / R.
L. Lieber, S. R. Ward // Am. J.Physiol.-Cell Physiol. – 2013. – Vol. 305, № 3. – P. C241-C252.445. Liu, W. Calcium channel blockers and modulation of innate immunity / W.Liu, A. Matsumori // Curr. Opin. Infect. Dis. – 2011. – Vol. 24, № 3. – P. 254-8.446. Love, P. B. Keloids: an update on medical and surgical treatments / P. B.Love, R. V. Kundu // J.
Drugs Dermatol. – 2013. – Vol. 12, № 4. – P. 403-409.447. Low-level laser (light) therapy (LLLT) in skin: stimulating, healing,restoring / P. Avci [et al.] // Semin. Cutan. Med. Surg. – 2013. – Vol. 32, № 1. – P. 4152.448. Lung beractant increases free cytosolic levels of Ca 2+ in human lungfibroblasts / A.
Guzmán-Silva [et al.] // PloS One. – 2015. – Vol. 10, № 7. – P.e0134564.449. Malta, M. C. Open palm technique in Dupuytren's disease treatment / M. C.Malta, M. P. T. Alves, Malta L. M. de Azevedo // Rev. Bras. Ortop (English Edition). –2013. – Vol. 48, № 3. – P. 246-250.250450. Management of "difficult" wounds / K. Neuhaus [et al.] // Eur. J. Pediatr.Surg. – 2013. – Vol. 23, № 5. – P. 365-374.451.
Margaret Shanthi, F. X. Comparison of intralesional verapamil withintralesional triamcinolone in the treatment of hypertrophic scars and keloids / F. X.Margaret Shanthi, K. Ernest, P. Dhanraj // Indian J. Dermatol. Venereol. Leprol. – 2008.– Vol. 74, № 4. – P. 343-348.452. Mass cytometry analysis reveals hyperactive NF Kappa B signaling inmyelofibrosis and secondary acute myeloid leukemia [Electronic resource] / D. A.Fisher [et al.] // Leukemia. – 2017. – DOI: 10.1038/leu.2016.377.453. Matrix stiffness–induced myofibroblast differentiation is mediated byintrinsic mechanotransduction / X. Huang [et al.] // Am. J. Respir. Cell mol.
Biol. –2012. – Vol. 47, № 3. – P. 340-348.454. Mechanisms and effects of biosynthesis and apoptosis in repair of fullthickness skin defect with collagen-chitosan dermal stent / S. J. Xu [et al.] // ZhonghuaZheng Xing Wai Ke Za Zhi. – 2009. – Vol. 25, № 3. – P. 208-212.455. Medical ozone therapy decreases postoperative uterine adhesion formationin rats / B. Uysal [et al.] // Arch. Gynecol. Obstet. – 2012. – Vol.
286, № 5. – P. 12011207.456. Mehdi, M.Z. MCM-2 and Ki-67 as proliferation markers in renal cellcarcinoma: a quantitative and semi-quantitative analysis / M. Z. Mehdi, A. H. Nagi, N.Naseem // Int. Braz. J. Urol. – 2016. – Vol. 42, № 6. – P.
1121-1128.457. Mei-ling, A. J. CaMKII and stress mix it up in mitochondria [Elecnronicresource] / A. J. Mei-ling, O. M. Koval // Front Pharmacol. – 2014. – DOI:10.3389/fphar.2014.00067.458. Mitochondrial Ca 2+ uptake induces cyclic AMP generation in the matrixand modulates organelle ATP levels / G. Di Benedetto [et al.] // Cell metabolism. –2013.
– Vol. 17, № 6. – P. 965-975.459. Momordin Ic induces HepG2 cell apoptosis through MAPK and PI3K/Aktmediated mitochondrial pathways / J. Wang [et al.] // Apoptosis. – 2013. – Vol. 18, №6. – P. 751-765.251460. Morphological study of burn wound healing with the use of collagenchitosan wound dressing / A. K. Kirichenko [et al.] // Bull. Exp. Biol. Med. – 2013. –Vol. 154, № 5.
– P. 692-696.461. Multiple Ca2+ signaling pathways regulate intracellular Ca2+ activity inhuman cardiac fibroblasts / J. B. Chen [et al.] // J. Cell Physiol. – 2010. – Vol. 223, №1. – P. 68-75.462. Myofibroblast-mediated mechanisms of pathological remodelling of theheart / K. T. Weber [et al.] // Nat. Rev. Cardiol. – 2013. – Vol. 10, № 1. – P. 15-26.463. N-Acetylglucosamine suppress collagenases activation in ultraviolet Birradiated human dermal fibroblasts: involvement of calcium ions and mitogen-activatedprotein kinases / Y.
P. Hwang [et al.] // J. Dermatol. Sci. – 2011. – Vol. 63, № 2. – P.93-103.464. Namazi, M. R. Strategies for prevention of scars: what can we learn fromfetal skin? / M. R. Namazi, M. K. Fallahzadeh, R. A. Schwartz // Int. J. Dermatol. –2011. – Vol. 50, № 1. – P. 85-93.465. New therapeutics for the prevention and reduction of scarring / N. L.Occleston [et al.] // Drug Discov. Today. – 2008. – Vol. 13, № 21-22.
– P. 973-981.466. NF-κB: an essential transcription factor in psoriasis / A. M. Goldminz [etal.] // J. Dermatol. Sci. – 2013. – Vol. 69, № 2. – P. 89-94.467. Nilius, B. Amazing T-type calcium channels: updating functionalproperties in health and disease / B. Nilius, E. Carbone // Pflügers Archiv. – 2014. –Vol.
466, № 4. – P. 623-626.468. N-methyl-D-aspartate (NMDA) antagonists--S(+)-ketamine, dextrorphan,and dextromethorphan--act as calcium antagonists on bovine cerebral arteries / I. R.Kamel [et al.] // J. Neurosurg. Anesthesiol.
– 2008. – Vol. 20, № 4. – P. 241-248.469. Novel approach to reduce postsurgical adhesions to a minimum:administration of losartan plus atorvastatin intraperitoneally / P. Dinarvand [et al.] // J.Surg. Res. – 2013. – Vol. 181, № 1. – P. 91-98.470. Novel insights on understanding of keloid scar: article review / W. Mari [etal.] // J. Am. Coll Clin. Wound Spec. – 2016. – Vol. 7, № 1-3. – P. 1-7.252471. Nuclear factor I-C links platelet-derived growth factor and transforminggrowth factor beta1 signaling to skin wound healing progression / G.
Plasari [et al.] //Mol. Cell Biol. – 2009. – Vol. 29, № 22. – P. 6006-6017.472. Ogawa, R. Keloid and hypertrophic scars are the result of chronicinflammation in the reticular dermis [Electronic resource] / R. Ogawa // Int. J. Mol. Sci.– 2017. – Vol. 18, № 3. – DOI: 10.3390/ijms18030606.473. Ohashi, N. Development of newer calcium channel antagonists: therapeuticpotential of efonidipine in preventing electrical remodelling during atrial fibrillation / N.Ohashi, H. Mitamura, S.
Ogawa // Drugs. – 2009. – Vol. 69, № 1. – P. 21-30.474. Opposing effects of bone morphogenetic protein-2 and endothelin-1 onlung fibroblast chloride currents / V. Shlyonsky [et al.] // Am. J. Respire. Cell mol. Biol.– 2011. – Vol. 45, № 6. – P. 1154-1160.475. Origin and function of myofibroblasts in kidney fibrosis / V.
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