Диссертация (1139521), страница 51
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Eksp. Ter. – 2011. – N 1. – P. 3–19.629. Lukyanova, L. D. Mitochondria signaling in adaptation to hypoxia // Int. J.Physiol. Pathophys. – 2014. – Vol. 5, N 4. – P. 363–381.630. Lukyanova, L. D. Mitochondria-controlled signaling mechanisms of brainprotection in hypoxia / L. D. Lukyanova, Y. I. Kirova // Front. Neurosci. – 2015. – N 9.– Р. 320.631. Lushchak, V. I. Adaptive response to oxidative stress: Bacteria, fungi, plantsand animals // Comp. Biochem. Physiol.
C Toxicol. Pharmacol. – 2011. – Vol. 153, Iss.2. – P. 175–190.632. Lykkesfeldt, J. Is vitamin C supplementation beneficial? Lessons learnedfrom randomized controlled trials / J. Lykkesfeldt, H. E. Poulsen // Br. J. Nutr. – 2010.– Vol. 103, Iss. 9. – P. 1251–1259.633. Macario, A. J. Sick chaperones, cellular stress, and disease / A. J. Macario,E. Conway de Macario // N. Engl. J. Med. – 2005.
– Vol. 353, Iss. 14. – P. 1489–1501.634. Macrophage antioxidant protections within atherosclerotic plaques /S. P. Gieseg [et al.] // Front. Biosci. – 2009. – N 1 (14). – P. 1230–1246.635. Magnesium and the inflammatory response: potential physiopathologicalimplications / A. Mazur [et al.] // Arch. Biochem. Biophys. – 2007. – Vol. 458, Iss. 1. –P. 48–56.636.
Masdeu, J. C. Pascual B. Genetic and degenerative disorders primarily causing dementia / J. C. Masdeu, B. Pascual // Handbook of Clinical Neurology. Vol. 135.Neuroimaging, Part I // J. C. Masdeu and R.G. Gonzalez (eds.). – Elsevier, 2016. –Chapter 26.
– P. 525–564.637. Mattson, M. P. Roles of the lipid peroxidation product 4-hydroxynonenal inobesity, the metabolic syndrome, and associated vascular and neurodegenerative disorders // Exp. Gerontol. – 2009. – Vol. 44, Iss. 10. – P. 625–633.308638. McKenna, M. A new role for α-ketoglutarate dehydrogenase complex: regulating metabolism through post-translational modification of other enzymes /M. McKenna, C. Rae // J. Neurochem.
– 2015. – Vol. 134, Iss. 1. – P. 3–6.639. Mechanisms of exercise-induced cardioprotection / S. Powers [et al.] //Physiology (Bethesda). – 2014. – Vol. 29, Iss, 1. – P. 27–38.640. Meta-analysis of lymphocytes in schizophrenia: clinical status and antipsychotic effects / B. Miller [et al.] // Biol. Psychiatry. – 2013. – Vol. 73, Iss. 10.
– P. 993–999.641. Mitochondrial complex II prevents hypoxic but not calcium- and proapoptotic Bcl-2 protein-induced mitochondrial membrane potential loss / B. J. Hawkins [et al.]// J. Biol. Chem. – 2010. – Vol. 285, N 34. – P. 26494–26505.642. Mitochondrial diaphorases as NAD+ donors to segments of the citric acidcycle that support substrate-level phosphorylation yielding ATP during respiratory inhibition / G. Kiss [et al.] // FASEB J. – 2014. – Vol. 28, Iss. 4. – P. 1682–1697.643.
Mitochondrial medicine: a metabolic perspective on the pathology of oxidative phosphorylation disorders / J.A. Smeitink [et al.] // Cell Metab. – 2006. – Vol. 3,Iss. 1. – P. 9–13.644. Mitochondrial mutations in atherosclerosis: new solutions in research andpossible clinical applications / I. A. Sobenin [et al.] // Curr. Pharm. Des.
– 2013. – Vol.19, Iss. 33. – P. 5942–5953.645. Mitra, S. Oxidized LDL, LOX-1 and atherosclerosis / S. Mitra, T. Goyal,J. L. Mehta // Cardiovasc Drugs Ther. – 2011. – Vol.25, Iss. 5. – P. 419–429.646. Mohammadi, M. T. Contribution of nitric oxide synthase (NOS) in bloodbrain barrier disruption during acute focal cerebral ischemiain normal rat /M.
T. Mohammadi, S. M. Shid-Moosavi, G. A. Dehghani // Pathophysiology. – 2012. –Vol. 19, Iss. 1. – P. 13–20.647. Mollnes, T. E. Complement in inflammatory tissue damage U and disease /T. E. Mollnes, W-C. Song, J. D. Lambris // Trends Immunol. Today. – 2002. – Vol.
23,Iss, 2. – P. 61–64.309648. Moskowitz, M. A. The science of stroke: mechanisms in search of treatments / M.A. Moskowitz, E.H. Lo, C. Iadecola // Neuron. – 2010. – Vol. 67, Iss. 2.– P.181–198.649. Mosser, D. M. The many faces of macrophage activation // J. Leukoc. Biol.– 2003. – Vol. 73, Iss. 2.
– P. 209–212.650. Murray, P. J. Protective and pathogenic functions of macrophage subsets /P. J. Murray, T. A. Wynn // Nat. Rev. Immunol. – 2011. – Vol. 11, Iss. 11. – P. 723–737.651. Nagle, D. G. Natural Product-Derived Small Molecule Activators of Hypoxia-Inducible Factor-1 (HIF-1) / D. G. Nagle, Y.-D. Zhou // Curr. Pharm. Des. – 2006. –Vol. 12, Iss. 21. – P.
2673–2688.652. Nauseef, W. M. Neutrophils at work / W. M. Nauseef, N. Borregaard // Nat.Immunol. – 2014. – Vol. 15, Iss. 7. – P. 602–611.653. Ncuroinllammation – an early event in both the history and pathogenesis ofAlzheimer's disease / P. Eikelenboom [et al.] // Neurodegener. Dis. – 2010. – Vol. 7, Iss.1-3. – P. 38–4l.654. Neonatal cytolines and chemokines and risk of Autism Spectrum Disorderthe Early Markers for Autism (EMA) study: a case-control study / O.
Zebro [et al.] // J.Neuroinflammation. – 2014. – Vol. 11. – P. 113.655. Neopterine and ige during respiratory-syncytial virus infection in infants /E. T. Kandelaki [et al.] // Georgian Med. News. – 2006. – Vol. 130. – P. 76–80.656. Neuroprotective effect of treatment with galantamine and choline alphoscerate on brain microanatomy in spontaneously hypertensive rats / S. Tayebati [et al.] // J.Neurol. Sci. – 2009. – Vol.
283, Iss. 1–2. – P. 187–194.657. Neuroprotective effect of treatment with galantamine and choline alphоscerate on brain microanatomy in spontaneously hypertensive rats / S. К. Tayebati [et al.] //J. Neurol. Sci. – 2009. – Vol. 283, Iss. 1–2. – P. 187–194.658. Neutrophil-derived cytokines: potential therapeutic targets in inflammation /T. Kasama [et al.] //Curr. Drug Targets Inflamm. Allergy. – 2005. – Vol. 4, Iss. 3. – P.273–279.310659. Neutrophilic myeloid-derived suppressor cells in cord blood modulate innateand adaptive immune responses / N. Rieber [et al.] // Сlin.
Exper. Immunol. – 2013. –Vol. 174, Iss. 1. – P. 45–52.660. Neutrophils in the activation and regulation of innate and adaptive immunity/ A. Mantovani [et al.] // Nat. Rev. Immunol. – 2011. – Vol. 11, Iss. 8. – P. 519–531.661. Niizuma, K. Oxidative stress and mitochondrial dysfunc tion as determinantsof ischemic neuronal death and survival / K. Niizuma, H. Endo, P. Chan //J.
Neurochem. – 2009. – Vol. 109, Suppl. 1. – P. 133–138.662. Nitro-fatty acids reduce atherosclerosis in apolipoprotein E-deficient mice /T. K. Rudolph [et al.] // Arterioscler. Thromh. Vasc. Biol. – 2010, – Vol. 30, Iss. 5. –P. 938–945.663. Nonlinear cardiovascular regulation changes in blood viscosity /B. Y. Salazar Vázquez [et al.] // Clin.
Hemorheol. Microcirc. – 2011. – Vol. 49, Iss. 1-4.– P. 29–36.664. Non-pharmacological cognitive intervention for aging and dementia: Currentperspectives / J. Alves [et al.] // World J. Clin. Cases. – 2013. – Vol. 1, Iss. 8. – P. 233–241.665. Orekhov A. N. Mechanisms of atherogenesis and development of antiatherosclerotic therapy // Curr. Pharm. – 2015. – Vol. 21, Iss.
9. – P. 1103–1106.666. Ovesgharan, S. Hypertensoin, executive dysfunction, and progression todementia / S. Ovesgharan, V. Hachinski // Arch. Neurol. – 2010. – Vol. 67, Iss. 2. – P.187–192.667. Oxidative damage in rat brain during aging: interplay between energy andmetabolic key target protein / F. Di Domenico [et al.] // Neurochem. Res. – 2010. – Vol.35, Iss. 12. – P. 2184–2192.668. Oxidative stress upregulates the NMDA receptor on cerebrovascular endothelium / C.
Betzen [et al.] // Free Radic. Biol. Med. – 2009. – Vol. 47, Iss. 8. –P. 1212–1220.669. Oxidative stress, inflammation, and cancer: How arc they linked? / S. Reuter[et al.] // Free Radic. Biol. Med. – 2010. – Vol. 49, Iss. 11. – P. 1603–1616.311670. Oxidative stress: Biomarkers and novel therapeutic pathways / K. Maiese [etal.] // Exp. Gerontol. – 2010. – Vol. 45, Iss. 3. – P. 217–234.671. OxPL/apoB, lipoprotein(a) and OxLDL biomarkers and cardiovascular disease in chronic hemodialysis patients / M. Bossola [et al.] // J. Nephrol.
– 2011. – Vol.24, Iss. 5. – P. 581–588.672. Pantoni, L. The relation between white matter lesions and cognition /L. Pantoni, A. Poggesi, D. Inzitari // Curr. Opin. Neurol. – 2007. – Vol. 20, Iss. 4. – P.390–397.673. Perinuclear mitochondrial clustering creates an oxidant-reach nuclear domain required for hypoxia-induced transcription / A.-В. Al-Mehdi [et al.] // Sci Signal.– 2012. – Vol.
5, Iss. 231. – P. ra47.674. Phillis, J. W. The role of phospholipases, cyclooxygenases, and lipoxygenases in cerebral ischemic/traumatic injuries / J. W. Phillis, M. H. O’Regan // Crit. Rev.Neurobiol. – 2003. – Vol. 15, Iss. 1. – P. 61–90.675. Pizova, N. V. Valdoxan (agomelatine) in the Treatment of Depression inCerebrovascular Diseases (results of the Russian Resonance multicenter naturalisticstudy) // Neurosci. Behav.
Physiol. –2014. – Vol. 44, Iss. 3. – 315–319.676. Plasma homocysteine and MTHFRC677T polymorphism as risk factors forincident dementia / A. H. Ford [et al.] // J. Neurol. Neurosurg. Psychiatry. – 2012. –Vol. 83, Iss. 1. – P. 70-75.677. Pleiotropic neuroprotective and metabolic effects of Actovegin’s mode ofaction / F. Machicao [et al.] // J.
Neurol. Sci. – 2012. – Vol. 322, Iss. 1. – P. 222–227.678. Polymorphism of the serotonin transporter gene modulates brain and physiological responses to acute stress in Japanese men / H. Ohira [et al.] // Stress. – 2009. –Vol. 12, Iss. 6. – P. 533–543.679. Possible involvement of NO/NOS signaling in hippocampal amyloid-betaproduction induced by transient focalcerebral ischemia in aged rats / S. Li [et al.] //Neurosci. Lett.
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