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– 2005. – Vol. 76, № 11. – Р. 1849-1855.253. Gingival crevicular fluid levels of RANKL and OPG in periodontal diseases:implications of their relative ratio / N. Bostanci [et al.] // Journal of clinicalperiodontology. – 2007. – Vol. 34, № 5. – Р. 370-376.254. Gingival tissue transcriptomes in experimental gingivitis / D. Jönsson [et al.]// Journal of clinical periodontology. – 2011.
– Vol. 38, № 7. – Р. 599-611.255. Global oral health inequalities task group-periodontal disease / L. J. Jin [et al.]// Advances in dental research. – 2011. – Vol. 23, № 2. – Р. 221-226.256. González O. A. Oral infectious diseases: a potential risk factor for HIV virusrecrudescence? / O. A. González, J. L. Ebersole, C. B. Huang // Oral diseases.
–2009. – Vol. 15, № 5. – Р. 313-327.257. Gonzalez-Angulo A. M., Hennessy B. T. J., Mills G. B. Future of personalizedmedicine in oncology: a systems biology approach //Journal of Clinical Oncology.– 2010. – Т. 28. – №. 16. – С. 2777-2783.258. Granulocyte chemotactic protein 2 (gcp‐2/cxcl6) complements interleukin‐8in periodontal disease / M. Kebschull [et al.] // Journal of periodontal research. –2009. – Vol. 44, № 4.
– Р. 465-471.259. Graves D. T. The contribution of interleukin-1 and tumor necrosis factor toperiodontal tissue destruction / D. T. Graves, D. Cochran // Journal ofperiodontology. – 2003. – Vol. 74, № 3. – Р. 391-401.260. Green J. C. The simplified oral hygiene index: A metod for classifying oralhygiene status / J. C. Green, J. R. Vermillion // J. Am.
Dent. Assoc. – 1960. – Vol.61. – P. 172-175.261. Hajeer A. H. TNF-alpha gene polymorphism: clinical and biologicalimplications / A. H. Hajeer, I. V. Hutchinson // Microsc Res Tech. – 2000. – Vol.50, № 3. – Р. 216-228.300262. Hajishengallis E. Neutrophil homeostasis and periodontal health in childrenand adults / E. Hajishengallis, G. Hajishengallis // Journal of dental research. – 2014.– Vol. 93, № 3.
– Р. 231-237.263. Hajishengallis G. Endogenous modulators of inflammatory cell recruitment /G. Hajishengallis, T. Chavakis // Trends in immunology. – 2013. – Vol. 34, № 1. –Р. 1-6.264. Hajishengallis G. Immunomicrobial pathogenesis of periodontitis: keystones,pathobionts, and host response / G. Hajishengallis // Trends in immunology. – 2014.– Vol. 35, № 1.
– Р. 3-11.265. Hajishengallis G. Immunomicrobial pathogenesis of periodontitis: keystones,pathobionts, and host response / G. Hajishengallis // Trends in immunology. – 2014.– Vol. 35, № 1. – Р. 3-11.266. Hajishengallis G. The keystone-pathogen hypothesis / G. Hajishengallis, R.P. Darveau, M. A. Curtis // Nature Reviews Microbiology. – 2012.
– Vol. 10, № 10.– Р. 717-725.267. Heitz‐Mayfield L. J. A. Disease progression: identification of high‐risk groupsand individuals for periodontitis / L. J. A. Heitz ‐Mayfield // Journal of ClinicalPeriodontology. – 2005. – Vol. 32, № s6. – Р. 196-209.268. HenningerM.UntersuchungvonAssoziationenderMikrosatellitenpolymorphismen MICA-TM, MICB-Ca_2_A und C1_4_1 mit derchronischen Parodontitis - 2011.269. Henry J. L. The economic and social impact of periodontal disease / J.
L.Henry, J. C. Sinkford // Public Health Reports. – 1979. – Vol. 94, № 2. – Р. 172.270. Herring M. E. Periodontal disease and control of diabetes mellitus / M. E.Herring, S. K. Shah // The Journal of the American Osteopathic Association. – 2006.– Vol. 106, № 7. – Р. 416-421.271. History of treated periodontitis and smoking as risks for implant therapy / I.K. Karoussis [et al.] // Int.
J. Oral. Maxillofac Implants. – 2009. – Vol. 24. – Р. 3968.301272. Hofbauer L. C. Clinical implications of the osteoprotegerin/RANKL/RANKsystem for bone and vascular diseases / L. C. Hofbauer, M. Schoppet // Jama. – 2004.– Vol. 292, № 4. – Р. 490-495.273. Hollegaard M. V. Cytokine gene polymorphism in human disease: on-linedatabases, Supplement 3 / M. V.
Hollegaard, J. L. Bidwell // Genes and immunity.– 2006. – Vol. 7, № 4. – Р. 269-276.274. Hood L., Friend S. H. Predictive, personalized, preventive, participatory (P4)cancer medicine //Nature Reviews Clinical Oncology. – 2011. – Т. 8. – №. 3. – С.184-187.275. Houri‐Haddad Y. T‐cell phenotype as a risk factor for periodontal disease / Y.Houri‐Haddad, A. Wilensky, L. Shapira // Periodontology 2000. – 2007.
– Vol. 45,№ 1. – Р. 67-75.276. Hull M. W. Indigenous microflora and innate immunity of the head and neck/ M. W. Hull, A. W. Chow // Infectious disease clinics of North America. – 2007. –Vol. 21, № 2. – Р. 265-282.277. Human atherosclerotic plaque contains viable invasive Actinobacillusactinomycetemcomitans and Porphyromonas gingivalis / E. V. Kozarov [et al.] //Arteriosclerosis, thrombosis, and vascular biology. – 2005. – Vol. 25, № 3.
– Р. e17e18.278. Identification of single bacterial cells in aqueous solution using confocal lasertweezers Raman spectroscopy / C. Xie [et al.] // Analytical chemistry. – 2005. – Vol.77, № 14. – Р. 4390-4397.279. Identification of the gene (BBS1) most commonly involved in Bardet-Biedlsyndrome, a complex human obesity syndrome / K. Mykytyn [et al.] // Naturegenetics. – 2002. – Vol. 31, № 4. – Р. 435-438.280. IL-1 and TNF antagonists inhibit the inflammatory response and bone loss inexperimental periodontitis / R. Assuma [et al.] // The Journal of Immunology. –1998. – Vol. 160, № 1.
– Р. 403-409.302281. IL‐1 gene polymorphism and smoking as risk factors for peri‐implant boneloss in a well‐maintained population / A. Feloutzis [et al.] // Clinical Oral ImplantsResearch. – 2003. – Vol. 14, № 1. – Р. 10-17.282. IL‐1RN gene polymorphism is associated with peri‐implantitis / M.
L. Laine[et al.] // Clinical Oral Implants Research. – 2006. – Vol. 17, № 4. – Р. 380-385.283. IL-2 is essential for TGF-β to convert naive CD4+ CD25− cells to CD25+Foxp3+ regulatory T cells and for expansion of these cells / S. G. Zheng [et al.] //The Journal of Immunology. – 2007. – Vol. 178, № 4. – Р. 2018-2027.284. IL‐23‐producing CD68+ macrophage‐like cells predominate within an IL‐17‐polarized infiltrate in chronic periodontitis lesions / J.
P. Allam [et al.] // Journal ofclinical periodontology. – 2011. – Vol. 38, № 10. – Р. 879-886.285. IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-relatedprotein ST2 and induces T helper type 2-associated cytokines / J. Schmitz [et al.] //Immunity. – 2005. – Vol. 23, № 5. – Р. 479-490.286. IL-6,RANKL,TNF-alpha/IL-1:interrelationsinboneresorptionpathophysiology / K. T.
Steeve [et al.] // Cytokine & growth factor reviews. – 2004.– Vol. 15, № 1. – Р. 49-60.287. IL-8 and IDO expression by human gingival fibroblasts via TLRs / R.Mahanonda [et al.] // The Journal of Immunology. – 2007. – Vol. 178, № 2. – Р.1151-1157.288. IL-9 induces differentiation of TH17 cells and enhances function of FoxP3+natural regulatory T cells / W.
Elyaman [et al.] // Proceedings of the NationalAcademy of Sciences. – 2009. – Vol. 106, № 31. – Р. 12885-12890.289. Immunohistochemical localization of Toll‐like receptors 1-10 in periodontitis/ A. Beklen [et al.] // Oral microbiology and immunology. – 2008. – Vol. 23, № 5.– Р. 425-431.290. Immunopathogenesis of chronic inflammatory periodontal disease: cellularand molecular mechanisms / G. J. Seymour [et al.] // Journal of periodontal research.– 1993. – Vol.
28, № 6. – Р. 478-486.303291. In situ identification of streptococci and other bacteria in initial dental biofilmby confocal laser scanning microscopy and fluorescence in situ hybridization / I.Dige [et al.] // European journal of oral sciences. – 2007. – Vol. 115, № 6. – Р. 459467.292. Inflammation and bone loss in periodontal disease / D. L. Cochran // Journalof periodontology. – 2008. – Vol. 79, № 8S.
– Р. 1569-1576.293. Influence of subcutaneous administration of recombinant TNF‐α on ligature‐induced periodontitis in rats / R. Gašperšič [et al.] // Journal of periodontal research.– 2003. – Vol. 38, № 2. – Р. 198-203.294. Interaction Between Fibronectin Fragments and Immunoglobulin G inGingival Crevicular Fluid of Patients with Periodontal Disease/Kompleksifibronektinskih fragmenata i imunoglobulina G u gingivalnoj tečnosti osoba obolelihod parodontopatije / G. Brajović [et al.] // Serbian Dental Journal.
– 2015. – Vol. 62,№ 2. – Р. 55-64.295. Interleukin 1 and receptor antagonist levels in gingival crevicular fluid inheavy smokers versus non‐smokers / A. Rawlinson [et al.] // J ournal of clinicalperiodontology. – 2003. – Vol. 30, № 1. – Р. 42-48.296. Interleukin 1 beta (IL-1β) promoter C [- 511] T polymorphism but not C [+3953] T polymorphism is associated with polycystic ovary syndrome / Y. Mu [et al.]// Endocrine. – 2010. – Vol.
37, № 1. – Р. 71-75.297. Interleukin 1B and interleukin 1RN polymorphisms are associated withincreased risk of gastric carcinoma / J. C. Machado [et al.] // Gastroenterology. –2011. – Vol. 121, № 4. – Р. 823-829.298. Interleukin 1B gene (IL-1B) and interleukin 1 receptor antagonist gene (IL1RN) polymorphisms in Helicobacter pylori-negative gastric cancer of intestinal anddiffuse histotype / A. Ruzzo [et al.] // Annals of oncology. – 2005. – Vol.
16, № 6.– Р. 887-892.299. Interleukin gene polymorphisms and breast cancer: a case control study andsystematic literature review / S. P. Balasubramanian [et al.] // BMC cancer. – 2006.– Vol. 6, № 1. – Р. 188.304300. Interleukin-1 and tumor necrosis factor- ALPHA. gene polymorphisms inTurkish patients with localized aggressive periodontitis / E. Guzeldemir [et al.] //Journal of oral science. – 2008. – Vol. 50, № 2. – Р. 151-159.301. Interleukin-1 gene polymorphisms and chronic periodontitis in adult whites:a systematic review and meta-analysis / N.