Диссертация (Молекулярно-генетические основы ювенильного артрита), страница 38
Описание файла
Файл "Диссертация" внутри архива находится в папке "Молекулярно-генетические основы ювенильного артрита". PDF-файл из архива "Молекулярно-генетические основы ювенильного артрита", который расположен в категории "". Всё это находится в предмете "медицина" из Аспирантура и докторантура, которые можно найти в файловом архиве РНИМУ им. Пирогова. Не смотря на прямую связь этого архива с РНИМУ им. Пирогова, его также можно найти и в других разделах. , а ещё этот архив представляет собой кандидатскую диссертацию, поэтому ещё представлен в разделе всех диссертаций на соискание учёной степени кандидата медицинских наук.
Просмотр PDF-файла онлайн
Текст 38 страницы из PDF
– Vol. 56, № 2. – P. 425-432.277. The impact of disease activity and tumour necrosis factor‐α inhibitor therapyon cytokine levels in juvenile idiopathic arthritis / H. M. Walters [et al.] // Clin ExpImmunol. – 2016. – Vol. 184, № 3. – P. 308-317.278. The magnitude of early response to methotrexate therapy predicts long-termoutcome of patients with juvenile idiopathic arthritis / M. Bartoli [et al.] // Ann RheumDis. – 2008. – Vol. 67, № 3. – P.
370-374.279. The NFKB1 ATTG ins/del polymorphism and risk of coronary heart diseasein three independent populations / U. Vogel [et al.] // Atherosclerosis. ‒ 2011. ‒ Vol. 219,№ 1. ‒ P. 200-204.280. The pathogenesis of oligoarticular/polyarticular vs systemic juvenileidiopathic arthritis / Y.T. Lin [et al.] // Autoimmun Rev. – 2011. – Vol. 10, № 8. – P. 482489.281. The PTPN22 allele encoding an R620W variant interferes with the removalof developing autoreactive B cells in humans / L. Menard [et al.] // J Clin Invest. – 2011.– Vol.
121, № 9. – P. 3635-3644.282. The role of the interleukin-6 family of cytokines in inflammatory arthritis andbone turnover / P. K. Wong [et al.] // Arthritis Rheum. – 2003. – Vol. 48, № 5. – P. 11771189.283. The susceptibility loci Juvenile Idiopathic Arthritis shares with otherautoimmune diseases extend to PTPN2, COG6 and ANGPT1 / S. D.
Thompson [et al.] //Arthritis Rheum. – 2010. – Vol. 62, № 11. – P. 3265-3276.284. Time to treatment as an important factor for the response to methotrexate injuvenile idiopathic arthritis / H. M. Albers [et al.] // Arthritis Rheum. – 2009. – Vol. 61,№ 1. – P. 46-51.285. TNF-α Polymorphisms in Juvenile Idiopathic Arthritis: Which Potential190Clinical Implications? / A. Scardapane [et al.] // Int J Rheumatol. – 2012. – Vol.
2012. –ID 756291.286. TNF-α promoter gene polymorphisms in Spanish children with persistentoligoarticular and systemic-onset juvenile idiopathic arthritis / C. Modesto [et al.] // ScandJ Rheumatol. – 2005. – Vol. 34, № 6. – P. 451-454.287. Trial of early aggressive therapy in polyarticular juvenile idiopathic arthritis /C. A. Wallace [et al.] // Arthritis Rheum. – 2012. – Vol.
64, № 6. – P. 2012–2021.288. Tumor necrosis factor alpha promoter polymorphisms in patients with juvenileidiopathic arthritis / H. Schmeling [et al.] // Clin Exp Rheumatol. – 2006. – Vol. 24, № 1.– P. 103–108.289. Tumor Necrosis Factor B (TNFB) Genetic Variants and Its IncreasedExpression Are Associated with Vitiligo Susceptibility [Электронный ресурс] / N. C.Laddha [et al.] // PLoS ONE. – 2013. – Vol. 8, № 11.
– e81736. ‒ URL:https://doi.org/10.1371/journal.pone.0081736. (10.04.2017).290. Tumor Necrosis Factor-α –308 Genotypes Influence Inflammatory Activityand TNF-α Serum Concentrations in Children with Juvenile Idiopathic Arthritis / A. F.Mourão [et al.] // J Rheumatol. – 2009. – Vol. 36, № 4. – P. 837-842.291. Tumor necrosis factor-α is a common genetic risk factor for asthma, juvenilerheumatoid arthritis, and systemic lupus erythematosus in a Mexican pediatric population/ S.
Jiménez-Morales [et al.] // Hum Immunol. – 2009. – Vol. 70, № 4. – P. 251–256.292. Tumour necrosis factor alpha and its soluble receptors in juvenile chronicarthritis / M. Rooney [et al.] // Rheumatology (Oxford). – 2000. – Vol. 39, № 4. – P. 432438.293. Tumour necrosis factor α G → A -238 and G → A -308 polymorphisms injuvenile idiopathic arthritis / S. Ozen [et al.] // Rheumatology (Oxford). – 2002. – Vol.41, № 2. – P. 223-227.294. Tumour necrosis factor-alpha gene promoter polymorphism and decreasedinsulin resistance / C.
P. Day [et al.] // Diabetologia. – 1998. –Vol. 41, № 4. – P. 430-434.295. van Dijkhuizen, E. H. Early predictors of prognosis in juvenile idiopathicarthritis: a systematic literature review / E. H. van Dijkhuizen, N. M. Wulffraat // Ann191Rheum Dis. – 2015. – Vol. 74, № 11. – P. 1996-2005.296. van Dijkhuizen, E. H. Prediction of methotrexate efficacy and adverse eventsin patients with juvenile idiopathic arthritis: a systematic literature review [Электронныйресурс] / E.
H. van Dijkhuizen, N. M. Wulffraat // Pediatr Rheumatol. – 2014. – Vol. 12.– ID 51. – URL: https://doi.org/10.1186/1546-0096-12-51. (10.04.2017).297. Variants in TNFAIP3, STAT4, and C12orf30 Loci Associated With MultipleAutoimmune Diseases Are Also Associated With Juvenile Idiopathic Arthritis / S.Prahalad [et al.] // Arthritis Rheum. – 2009. – Vol. 60, № 7. – P. 2124-2130.298. Variation in the CTLA4 3'UTR has phenotypic consequences for autoreactiveT cells and associates with genetic risk for type 1 diabetes / V. M. de Jong [et al.] // GenesImmun. – 2016. – Vol.
17, № 1. – P. 75-78.299. Walker, L. S. Treg and CTLA-4: two intertwining pathways to immunetolerance / L. S. Walker // J Autoimmun. – 2013. – Vol. 45. – P. 49-57.300. Wallace, C. A. Preliminary criteria for clinical remission for select categoriesof juvenile idiopathic arthritis / C. A.
Wallace, N. Ruperto, E. Giannini // J Rheumatol. –2004. ‒ Vol. 31, № 11. – P. 2290–2294.301. Wang, Z. SNPs, protein structure, and disease / Z. Wang, J. Moult // HumMutat. – 2001. – Vol. 17, № 4. – P. 263-270.302. Weber, A. Interleukin-1 (IL-1) pathway / A. Weber, P. Wasiliew, M. Kracht// Sci Signal. – 2010.
– Vol. 3, № 105. – cm1.303. Westfall P. H. Resampling-Based Multiple Testing: Examples and Methodsfor p-Value Adjustment / P. H. Westfall, S. S. Young. – New York : John Wiley & Sons,Inc, 1993 – 360 p.304. Why is PTPN22 a good candidate susceptibility gene for autoimmune disease?/ G. L. Burn [et al.] // FEBS Lett. – 2011. – Vol. 585, № 23.
– P. 3689-3698.305. Wishart, D. S. PepTool and GeneTool: platform-independent tools forbiological sequence analysis / D. S. Wishart, P. Stothard, G. H. Van Domselaar // MethodsMol Biol. – 2000. ‒ Vol. 132. – P. 93-113.306. Woo, P. Cytokines and juvenile idiopathic arthritis / P. Woo // Curr RheumatolRep. – 2002.
– Vol. 4, № 6. – P. 452-457..