Диссертация (1174214), страница 59
Текст из файла (страница 59)
Predictors of mortality in insulin dependent diabetes: 10 yearobservational follow up stud / P. Rossing, P. Hougaard, K. Borch-Johnsen et al. // BMJ.–1996. – Vol.313. – P. 779–784.633. Roth, A. Telomerase levels control the lifespan of human T lymphocytes / A. Roth,H.
Yssel, J. Pene et al. // Blood. –2003. – Vol.102. – P. 849.634. Rozing, M.P. Familial longevity is associated with decreased thyroid function /M.P. Rozing, J.J. Houwing-Duistermaat, P.E. Slagboom et al. // J Clin Endocrinol Metab.– 2010. – Vol.95. – P. 4979–4984.329635. Rozing, M.P. Low serum free triiodothyronine levels mark familial longevity: theLeiden Longevity Study / M.P. Rozing, R.G. Westendorp, A.J. de Craen et al.
// JGerontol A Biol Sci Med Sci. – 2010. – Vol.65. – P. 365–368.636. Rozing, M.P. Serum triiodothyronine levels and inflammatory cytokine productioncapacity / M.P. Rozing, R.G. Westendorp, A.B. Maier et al. // Age. – 2012. – Vol.34. –P. 195–201.637. Russo, A. Telomere length variation in juvenile acute myocardial infarction / A.Russo, L. Palumbo, C. Fornengo et al. // PLoS One. – 2012. – Vol.7.
– P. 49206.638. Sabharwal, S. Telomere length dynamics in early life: the blood-and-muscle model/ S. Sabharwal, S. Verhulst, G. Guirguis et al. // FASEB J. – 2017. – Vol. 32(1). – P. 529534.639. Saenz de Tejada, I. Pathophysiology of erectile dysfunction / I. Saenz de Tejada, J.Angulo, S. Cellek et al. // J Sex Med. – 2005. – Vol. 2.
– P. 26-39.640. Sahin, E. Telomere dysfunction induces metabolic and mitochondrial compromise/ E. Sahin, S. Colla, M. Liesa et al. //Nature. – 2011. – Vol. 470(7334). – P. 359–65.641. Saito, H. Effects of aging on mortality, hypothermia, and cytokine induction inmice with endotoxemia or sepsis / H. Saito, E.R. Sherwood, T.K.
Varma et al. // MechAgeing Dev. – 2003. – Vol.124. – P. 1047–1058.642. Sakurai, K. Induction of endothelin-1 production in endothelial cells viacooperative action between CD40 and lectin-like oxidized LDL receptor (LOX-1) / K.Sakurai, L. Cominacini, U. Garbin et al. // J Cardiovasc Pharmacol. – 2004. – Vol.44. –P. 173–180.643. Saliques, S. Circulating leukocyte telomere length and oxidative stress: a newtarget for statin therapy / S. Saliques, J.R. Teyssier, C.
Vergely et al. // Atherosclerosis. –2012. – Vol.219. – P. 753–760.644. Salpea, K. Association of telomere length with type 2 diabetes, oxidative stress andUCP2 gene variation / K. Salpea, P.J. Talmud, J.A. Cooper et al. // Atherosclerosis. –2010. – Vol.209(1). – P. 42-50.645. Salpea, K.D. Тelomere length in atherosclerosis and diabetes / K.D. Salpea, S.E.Humphries // Atherosclerosis. – 2010. – Vol.209. – P.
35-8.330646. Salpea, K.D. The association of telomere length with paternal history of prematuremyocardial infarction in the European Atherosclerosis Research Study II / K.D. Salpea,V. Nicaud, L. Tiret et al. // J Mol Med. – 2008. – Vol.86. – P. 815–824.647. Samani, N.J. Telomere shortening in atherosclerosis / N.J. Samani, R. Boultby, R.Butler et al. // Lancet. – 2001. – Vol.358.
– P. 472–3.648. Samper E. Restoration of telomerase activity rescues chromosomal instability andpremature aging in Terc−/− mice with short telomeres / E. Samper, J.M. Flores, M.A.Blasco // EMBO Rep. – 2001. – Vol.2. – P. 800–807.649.Sampson, M.J. Monocyte telomere shortening and oxidative DNA damage in type2 diabetes / M.J. Sampson, M.S. Winterbone, J.C.
Hughes et al. // Diabetes Care. – 2006.– Vol.29. – P. 283–89.650. Sano, H. Receptors for proteins modified by advanced glycation endproducts(AGE)—Their functional role in atherosclerosis / H. Sano, R. Nagai, K. Matsumoto et al.// Mech. Ageing Dev. – 1999. – Vol.107. – P. 333–346.651.
Santhanam, L. Arginase and vascular aging / L. Santhanam // J Appl Physiol. –2008. – Vol.105. – P. 1632-1642.652. Sarin, K. Y. Conditional telomerase induction causes proliferation of hair folliclecells / K. Y. Sarin, P. Cheung, D. Gilison et al. // Nature. – 2005. – Vol.436.
– P. 1048–1052.653. Satoh, M. Association between oxidative DNA damage and telomere shortening incirculating endothelial progenitor cells obtained from metabolic syndrome patients withcoronary artery disease / M. Satoh, Y. Ishikawa, Y. Takahashi et al. // Atherosclerosis. –2008. – Vol.198. – P. 347–353.654. Satoh, M. Effect of intensive lipid-lowering therapy on telomere erosion inendothelial progenitor cells obtained from patients with coronary artery disease / M.Satoh, Y. Minami, Y.
Takahashi et al. // ClinSci. – 2009. – Vol.116. – P. 827-35.655. Saurwein-Teissl, M. Lack of antibody production following immunization in oldage:association with СD8(+)CD28(−) T cell clonal expansions and an imbalance in theproduction of Th1 and Th2 cytokines / M. Saurwein-Teissl, T.L. Lung, F. Marx et al. //J. Immunol.
– 2002. – Vol.168. – P. 5893.331656. Savage, S. A. Genetic variation, nucleotide diversity, and linkage disequilibrium inseven telomere stability genes suggest that these genes may be under constraint / S. A.Savage, B.J. Stewart, A. Eckert et al. // Hum. Mutat. – 2005. – Vol.26. – P. 343–350.657. Schulz, C. Adipocyte-brain: Crosstalk / C. Schulz, K. Paulus, H.
Lehnert // ResultsProbl. Cell Differ. – 2010. – Vol.52. – P. 189–201.658. Schutte, S. A meta-analytic review of the effects of mindfulness meditation ontelomerase activity / S. Schutte, J.M. Malouff // Psychoneuroendocrinology. – 2014. –Vol.42. – P. 45–48.659. Sell, D.R. Molecular basis of arterial stiffening: role of glycation— a mini-review/ D.R.
Sell, V.M. Monnier // Gerontology. –2012. – Vol.58. – P. 227– 237.660. Sengstock, D. Dominance of traditional cardiovascular risk factors over renalfunction in predicting arterial stiffness in subjects with chronic kidney disease / D.Sengstock, R.L. Sands, B.W. Gillespie et al. // Nephrol Dial Transplant.
– 2010. – Vol.25.– P. 853–861.661. Sengstock, D.M. Arterial Stiffness Is Related to Insulin Resistance in NondiabeticHypertensive Older Adults / D.M. Sengstock, P.V. Vaitkevicius, M.A. Supiano // TheJournal of Clinical Endocrinology & Metabolism. – 2005. – Vol.90(5). – P. 2823–2827.662. Serra, V.
Telomere length as a marker of oxidative stress in primary humanfibroblast cultures / V. Serra, T. Grune, N. Sitte et al. // Ann NY Acad Sci. – 2000. – Vol.908. – P. 327-330.663. Serra, V. Extracellular superoxide dismutase is a major antoxidant to humanfibroblasts and slows telomere shortening / V. Serra, T. Von Zglinicki, M.
Lorenz et al.// J Biol Chem. – 2003. – Vol.278. – P. 6824-6830.664. Shargorodsky, M. Long-term thyrotropin-suppressive therapy with levothyroxineimpairs small and large artery elasticity and increases left ventricular mass in patientswith thyroid carcinoma / M. Shargorodsky, S. Serov, D. Gavish et al. // Thyroid. – 2006.– Vol.16.
– P. 381–386.665. Shay, J.W. Hayflick, his limit, and cellular ageing / J.W. Shay, W.E. Wright // NatRev Mol Cell Biol. – 2000. – Vol.1(1). – P. 72-6.332666. Shen, W. The effects of simvastatin on angiogenesis: studied by an original modelof atherosclerosis and acute myocardial infarction in rabbit / W. Shen, H.M. Shi, W.H.Fan et al. // MolBiol Rep.
– 2011. – Vol.38. – P. 3821-8.667. Shih, C.H. Thyroid hormone receptor dependent transcriptional regulation offibrinogen and coagulation proteins / C.H. Shih, S.L. Chen, C.C. Yen et al. //Endocrinology. – 2004. – Vol.145. – P. 2804-2814.668. Shimada, T. Angiogenesis and vasculogenesis are impaired in the precociousaging klotho mouse / T. Shimada, Y. Takeshita, T. Murohara et al.
// Circulation. – 2004.– Vol.110. – P. 1148–1155.669. Shintani, S. Mobilization of endothelial progenitor cells in patients with acutemyocardial infarction / S. Shintani, T. Murohara, H. Ikeda et al. // Circulation. – 2001. –Vol.103.
– P. 2776-2779.670. Siddiqi, A. Serum cytokines in thyrotoxicosis / A. Siddiqi, J.P. Monson, D.F. Woodet al. //J Clin Endocrinol Metab. – 1999. – Vol.84. – P. 435–439.671. Sidorov, I. Leukocyte telomere dynamics and human hematopoietic stem cellkinetics during somatic growth / I. Sidorov, M. Kimura, A. Yashin et al. // Exp. Hematol.– 2009. – Vol.37. – P. 514–524.672. Simes, R.J. Relationship between lipid levels and clinical outcomes in the Longterm Intervention with Pravastatin in the Ischemic Disease (LIPID) Trial: to what extentis the reduction in coronary events with pravastatin explained by on-study lipid levels? /R.J.
Simes, I.C. Marschner, D. Hunt et al. // Circulation. – 2002. – Vol.105. – P. 1162–9.673. Simoncini, T. Interaction of oestrogen receptor with the regulatory subunit ofphosphatidylinositol-3-OH kinase / T. Simoncini, A. Hafezi-Moghadam, D.P. Brazil etal. // Nature. – 2000. – Vol.407. – P. 538–541.674. Singh, B.M. Interactions between the renin-angiotensin system and dyslipidemia:relevance in the therapy of hypertension and coronary heart disease / B.M.
Singh, J.L.Mehta // Arch Intern Med. – 2003. – Vol.163. – P. 1296-1304.675. Sirakov, M. The thyroid hormones and their nuclear receptors in the gut: fromdevelopmental biology to cancer / M. Sirakov, M. Plateroti // Biochim Biophys Acta. –2011. – Vol.1812. – P. 938–946.333676.