Диссертация (Патофизиологическое значение ацетилхолина в пролиферации опухолевых клеток толстой кишки in vitro и in vivo), страница 23
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2013. № 11 (88). C. 1340–1346.157. Rosenvinge E.C. Von, Raufman J.-P. Muscarinic Receptor Signaling in ColonCancer // Cancers. 2011. № 4 (3). C. 971–981.158. Sadallah S., Eken C., Schifferli J. a. Ectosomes as modulators of inflammationand immunity // Clinical & Experimental Immunology. 2011. № 1 (163). C. 26–32.159. Said A., Raufman J.-P., Xie G. The Role of Matrix Metalloproteinases inColorectal Cancer // Cancers. 2014.
№ 1 (6). C. 366–375.160. Saito Y. [et al.]. Epigenetic Alterations and MicroRNA Misexpression in143Cancer and Autoimmune Diseases: a Critical Review // Clinical Reviews in Allergyand Immunology. 2013. C. 1–8.161. Santos D.C.M. Dos [et al.]. Morphologic characterization and distribution ofendocrine cells in the large intestine of the opossum Didelphis aurita (WiedNeuwied, 1826) // Tissue and Cell. 2013. № 5 (45). C. 338–349.162. Saotome K., Morita H., Umeda M. Cytotoxicity test with simplified crystalviolet staining method using microtitre plates and its application to injection drugs.// Toxicology in vitro : an international journal published in association withBIBRA.
1989. № 4 (3). C. 317–321.163. Schuller H.M. [et al.]. Interaction of tobacco-specific toxicants with theneuronal alpha7-nicotinic acetylcholine receptor and its associated mitogenic signaltransduction pathway: potential role in lung carcinogenesis and pediatric lungdisorders. // European Journal of Pharmacology. 2000. № 1–3 (393). C. 265–77.164. Schuller H.M. Is cancer triggered by altered signalling of nicotinicacetylcholine receptors? // Nature Reviews Cancer. 2009.
№ 3 (9). C. 195–205.165. Singh S., Pillai S., Chellappan S. Nicotinic Acetylcholine Receptor Signalingin Tumor Growth and Metastasis // Journal of Oncology. 2011. (2011). C. 1–11.166. Smith L.H. The APUD cell concept // Journal of surgical oncology. 1976. № 2(8). C. 137–142.167. Song P. [et al.]. Choline transporter-like protein 4 (CTL4) links to nonneuronal acetylcholine synthesis // Journal of Neurochemistry. 2013.
№ 4 (126). C.451–461.168. Song P., Spindel E.R. Basic and clinical aspects of non-neuronal acetylcholine:Expression of non-neuronal acetylcholine in Lung cancer provides a new target forcancer therapy // Journal of pharmacological science. 2008. (106). C.
180–185.169. Sousa E., Melo F. De [et al.]. Poor-prognosis colon cancer is defined by amolecularly distinct subtype and develops from serrated precursor lesions // NatureMedicine. 2013. № 5 (19). C. 614–618.170. Sun X. [et al.]. Rosiglitazone inhibits 4 nicotinic acetylcholine receptorexpression in human lung carcinoma cells through peroxisome proliferator-activated144receptor -independent signals // Molecular Cancer Therapeutics.
2009. № 1 (8). C.110–118.171. Suneja M. [et al.]. Hormonal regulation of energy-protein homeostasis inhemodialysis patients: an anorexigenic profile that may predispose to adversecardiovascular outcomes. // American journal of physiology. Endocrinology andmetabolism. 2011. № 1 (300). C. E55-64.172. Syed M. [et al.].
Acetylcholinesterase supports anchorage independence incolon cancer // Clinical & Experimental Metastasis. 2008. № 7 (25). C. 787–798.173. Taketo M.M., Edelmann W. Mouse Models of Colon Cancer // YGAST. 2009.№ 3 (136). C. 780–798.174. Tanaka T. [et al.]. A novel inflammation-related mouse colon carcinogenesismodel induced by azoxymethane and dextran sodium sulfate // Cancer Science.2003.
№ 11 (94). C. 965–973.175. Tanaka T. [et al.]. Melatonin suppresses AOM/DSS-induced large boweloncogenesis in rats // Chemico-Biological Interactions. 2009. № 2 (177). C. 128–136.176. Thomas G.A., Rhodes J., Ingram J.R. Mechanisms of Disease: nicotine—areview of its actions in the context of gastrointestinal disease // Nature ClinicalPractice Gastroenterology & Hepatology.
2005. № 11 (2). C. 536–544.177. Tracey K.J. The inflammatory reflex 2002. № December (420). C. 853–859.178. Uysal M., Sava S. Caecum location in laboratory rats andmice : an anatomicaland radiological study 2016. (4). C. 1–10.179. Vadlamudi H.C. [et al.]. Receptors and ligands role in colon physiology andpathology // J Recept Signal Transduct Res. 2013.
№ 1 (33). C. 1–9.180. Wagenaar-Miller R. a, Gorden L., Matrisian L.M. Matrix metalloproteinases incolorectal cancer: is it worth talking about? // Cancer metastasis reviews. 2004. №1–2 (23). C. 119–135.181. Wang H. [et al.]. Nicotinic acetylcholine receptor a 7 subunit is an essentialregulator of inflammation 2003.
№ January (421). C. 1–5.182. Wang J. [et al.]. Regulation of acetylcholine receptor clustering by the tumor145suppressor APC // Nature Neuroscience. 2003. № 10 (6). C. 1017–1018.183. Wang K., Grivennikov S.I., Karin M. Implications of anti-cytokine therapy incolorectal cancer and autoimmune diseases. // Annals of the rheumatic diseases.2013. (72 Suppl 2). C. ii100-3.184. Weber R. V. [et al.]. Obesity potentiates AOM-induced colon cancer //Digestive Diseases and Sciences. 2000. № 5 (45). C. 890–895.185.
Wessler I. [et al.]. Release of non-neuronal acetylcholine from the humanplacenta: difference to neuronal acetylcholine // Naunyn-Schmiedeberg’s Archivesof Pharmacology. 2001. № 3 (364). C. 205–212.186. Wessler I. [et al.]. The non-neuronal cholinergic system in humans:Expression, function and pathophysiology // Life Sciences. 2003. № 18–19 (72). C.2055–2061.187. Wessler I., Kirkpatrick C.J. Acetylcholine beyond neurons: the non-neuronalcholinergic system in humans // British Journal of Pharmacology.
2009. № 8 (154).C. 1558–1571.188. Whiteside T.L. The tumor microenvironment and its role in promoting tumorgrowth // Oncogene. 2008. № 45 (27). C. 5904–5912.189. Wieczorek E. [et al.]. Matrix metalloproteinases and genetic mouse models incancer research: a mini-review // Tumor Biology. 2015. № 1 (36). C. 163–175.190. Winder T., Lenz H.-J. Molecular predictive and prognostic markers in coloncancer // Cancer Treatment Reviews. 2010. № 7 (36). C. 550–556.191.
Winter B.Y. De Interplay between inflammation, immune system and neuronalpathways: Effect on gastrointestinal motility // World Journal of Gastroenterology.2010. № 44 (16). C. 5523.192. Witty J.P. [et al.]. Modulation of Matrilysin Levels in Colon-Carcinoma CellLines Affects Tumorigenicity in-Vivo // Cancer Research. 1994. № Iss 17 (Vol 54).C. 4805–4812.193. Wong H.P.S.
[et al.]. Nicotine promotes cell proliferation via α7-nicotinicacetylcholine receptor and catecholamine-synthesizing enzymes-mediated pathwayin human colon adenocarcinoma HT-29 cells // Toxicology and Applied146Pharmacology. 2007. № 3 (221). C. 261–267.194. Wu C.-H., Lee C.-H., Ho Y.-S. Nicotinic Acetylcholine Receptor-BasedBlockade: Applications of Molecular Targets for Cancer Therapy // Clinical CancerResearch. 2011. № 11 (17).
C. 3533–3541.195. Xu W. [et al.]. The role of nitric oxide in cancer. // Cell research. 2002. № 5–6(12). C. 311–320.196. Yang K. [et al.]. Interaction of Muc2 and Apc on Wnt signaling and inintestinal tumorigenesis: Potential role of chronic inflammation // Cancer Research.2008. № 18 (68). C. 7313–7322.197. Zlobec I. [et al.]. Clinicopathological and protein characterization of BRAF and K-RAS -mutated colorectal cancer and implications for prognosis // InternationalJournal of Cancer. 2010.
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