Диссертация (1140524), страница 24
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Tremmel, U. Gerdtham, P. Nilsson, S. Saha // Int. J. Environ. Res. Public. Health.– 2017. - Vol.14(4). - P.254-258.278. Tzanavari, T. TNF-alpha and obesity /T. Tzanavari, P. Giannogonas, K. Karalis// Curr Dir Autoimmun. – 2010. – Vol.11. – P.145-156.279. Vahratian, A. Prevalence of overweight and obesity among women ofchildbearing age: results from the 2002 National Survey of Family Growth /A.Vahratian // Matern. Child Health J. – 2009. - Vol.13. – Р.268–273.280.
Valensi, P. Atherogenic dyslipidemia and risk of silent coronary artery diseasein asymptomatic patients with type 2 diabetes: a cross-sectional study /P.Valensi, A.Avignon, A. Sultan et al.// Cardiovasc Diabetol. – 2016. - Vol.15(1). – P.104.147281. van der Steeg, J.W. Obesity affects spontaneous pregnancy chances insubfertile, ovulatory women /J.W.van der Steeg, P. Steures, M.J.
Eijkemans et al. //Hum. Reprod. – 2008. - Vol.23(2). – Р.324-328.282. Van Doorn, C. Leptin resistance and hippocampal behavioral deficits /C. VanDoorn, V.A. Macht, C.A. Grillo, L.P. Reagan // Physiol. Behav. - 2017. - Vol.176. –Р.207-213.283. Viani, I. Ghrelin in hibits steroid biosynthesis by cultured granulosa-lutein cells/I. Viani, A. Vottero, F. Tassi et al. // J. Clin.
Endocrinol. Metab. – 2008. - Vol.93(4).– Р.1476-1481.284. Vieira-Potter, V. J. Inflammation and macrophage modulation in adipose tissues/V.J. Vieira-Potter // Cell. Microbiol. – 2014. – Vol.16. – P.1484–1492.285. Vink, J.M. Heritability of polycystic ovary syndrome in a Dutch twin-familystudy /J.M.Vink, S. Sadrzadeh, C. Lambalk, D. Boomsma // J. Clin. Endocrinol.Metab. – 2006. - Vol.91. – Р.2100–2104.286. Virtue, S.Adipose tissue expandability, lipotoxicity and the metabolicsyndrome--an allostatic perspective /S.
Virtue, A. Vidal-Puig // Biochim. BiophysActa. – 2010. - Vol.1801(3). – Р.338–349.287. Wajchenberg, B.L. Subcutaneous and visceral adipose tissue: their relation tothe metabolic syndrome /B.L. Wajchenberg // Endocr. Rev. – 2000. - Vol.21(6). –Р.697–738.288. Warnick, G.R. Evolution of methods for measurement of HDL-cholesterol.From ultracentrifugation to homogenous assays /G.R. Warnick, M. Nauck, N.
Rifai //Clin. Chem. -2001. - Vol.47(9). – Р.1579-1596.289. Webb, P.M. Obesity and gynecologic cancer etiology and survival /P.M. Webb// AmSoc Clin Oncol Educ Book. – 2013. – Vol.33. – P.222–228.148290. Wei, S. Obesity and menstrual irregularity: associations with SHBG,testosterone, and insulin /S. Wei, M.D. Schmidt, T. Dwyer et al.
// Obesity. – 2009. Vol.17. – Р.1070–1076.291. Wise, L.A. An internet-based prospective study of body size and time-topregnancy /L.A. Wise, K.J. Rothman, E.M. Mikkelsen et al. // Hum. Reprod. – 2010.- Vol.25. – Р.253–264.292. Wozniak, S.E. Adipose tissue: the new endocrine organ? /S.E. Wozniak, L.I.Gee, M.S. Wachtel // Dig. Dis. Sci. – 2009. – Vol.54. – P.1847–1856.293.
Xita, N. A polymorphism in the resistin gene promoter is associated with bodymass index in women with polycystic ovary syndrome /N. Xita, I. Georgiou, A.Tsatsoulis et al. // Fertil. Steril. – 2004. - Vol.82(5). – Р.1466–1467.294. Yamauchi, J. Serum resistin is reduced by glucose and meal loading in healthyhuman subjects /J. Yamauchi, H.
Osawa, T. Takasuka et al.// Metabolism. – 2008. –Vol.57(2). – P.149–56.295. Yaylali, G.F. Sexual dysfunction in obese and overweight women /G.F. Yaylali,S. Tekekoglu, F. Akin // Int. J. Impot. Res. – 2010. – Vol.22(4). – P.220–226.296. Yildiz, B.O. Prevalence, phenotype and cardiometabolic risk of polycysticovary syndrome under different diagnostic criteria /B.O. Yildiz, G. Bozdag, Z.Yapici et al. // Hum. Reprod. – 2012. - Vol.27. – Р.3067–3073.297. Yildiz, B.O.
Impact of obesity on the risk for polycystic ovary syndrome /B.O.Yildiz, E.S. Knochenhauer// J.Clin. Endocrinol. Metab. – 2008. - Vol.93. – Р.162–168.298. Yilmaz, M. Levels of lipoprotein and homocysteine in non-obese and obesepatients with polycystic ovary syndrome /M. Yilmaz, A. Biri, N. Bukan et al.Gynecol. Endocrinol. - 2005.
- Vol.20(5). – Р.258-263.299. Yura, S. Resistin is expressed in the human placenta /S. Yura, N. Sagawa , H.Itoh et al.// Journal of Clinical Endocrinology and Metabolism. – 2003. – Vol.88(3). –P.1394–1397.149300. Zaidi, S.I. Relationship of serum resistin with insulin resistance and obesity/S.I. Zaidi, T.A. Shirwany // J Ayub Med Coll Abbottabad. – 2015. - Vol.27(3). –P.552-555.301. Zhang, J. The plasma level and gene expression of resistin in polycystic ovarysyndrome /J.
Zhang, L. Zhou, L. Tang et al. // Gynecol Endocrinol. – 2011. –Vol.27(12). – P.982–7.302. Zheng, S.H. Leptin Levels in Women With Polycystic Ovary Syndrome: ASystematic Review and a Meta-Analysis /S.H. Zheng, D.F. Du, X.L. Li // Reprod. Sci.– 2017. - Vol.24(5). – P.656-670.303. Zwirska-Korczala, K. Postprandial response of ghrelin and PYY and indices oflow grade inflammation in lean young women with polycystic ovary syndrome /K.Zwirska-Korczala, K. Sodowski, S. Konturek et al. // PJ Physiol. Pharmacol. – 2008.
Vol.2. – Р.161-178..
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