Диссертация (Иммуноморфологические ассоциации инфекции helicobacter pylori с показателями воспаления и дисфункции эндотелия в оценке течения и прогноза ишемической болезни сердца), страница 72
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Ardito et all // Lab. Invest. – 1997. – N 77. – P. 257-67.519. Smythies L.E. Helicobacter pylori-induced mucosal inflammation is Th1 mediatedand exacerbated in IL-4, but not IFN-gamma, gene-deficient mice / L.E. Smythies,K.B. Waites, J.R. Lindsey et all // J. Immunol. – 2000. – N 165. – P.
1022-29.520. Solid L.M. Interferon gamma enhances expression of secretory component, theepithelial receptor for polymeric immunoglobulins / L.M. Solid, D. Kvale, P.Brandtzaeg et all // J. Immunol. – 1987. – Vol. 138, N 12. – P. 4303-06.521. Sommer F. Antrum- and corpus mucosa-infiltrating CD4+ lymphocytes inHelicobacter pylori gastritis display a Th1 phenotype / F. Sommer, G. Faller, P.Konturek et all // Infect.
Immun. – 1998. – N 66. – P. 5543-46.522. Stacey L.K. The infections etiology of chronic diseases: defining the relationship,enhancing the research, and mitigation the effects / L.K. Stacey, S.M. Lemon, M.Najafi. – Washington, D.S.: The National Academic Press, 2004. – 215.407523.
Stary H.C. Changes in components and structure of atherosclerotic lesions developing from childhood to middle age in coronary arteries / H.C. Stary // Basic Res.Cardiol. – 1994. – N 89 (suppl. 1). – P. 17-32.524. Stary H.C. A definition of initial, fatty streak, and intermediate lesions of atherosclerosis: a report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association. Special report / H.C. Stary, A.B. Chandler, S. Glagov et all // Arterioscler. Thromb. – 1994. – N 14. – P. 840-56.525.
Stead R.H. The ovulatory cycle / R.H. Stead // Ibid. – 1992. – N 4. – P. 91-99.526. Steinberg D. Conner Memorial Lecture. Oxidative modification of LDL andatherogenesis / D. Steinberg, A. Lewis // Circulation. – 1997. – Vol. 95, N 4. – P.1062-71.527. Stone A.F. Helicobacter pylori is an ethiological factor for ischemic heart disease:the case in favor / A.F. Stone, M.A. Mendall // Digest. Liver Dis. – 2000. – N 32. –P. 62-64.528. Straface E. Gender-specific features of plasmatic and circulating cell alterations asrisk factors in cardiovascular disease / E. Straface, P.
Lista, L. Gambardella et all //Fundam. Clin. Pharmacol. – 2010. – Vol. 24, N 6. – P. 665-74.529. Strandberg T.E. Prospective study of Helicobacter pylori seropositivity and cardiovascular diseases in a general elderly population / T.E. Strandberg, R.S. Tilvis, M.Vuoristo et all // Br. Med. J. – 1997. – N 314. – P. 1317-18.530. Strober W. Reciprocal IFN-gamma and TGF-beta responses regulate theoccurrence of mucosal inflammation / W. Strober, B. Kelsall, I. Fuss et all //Immunol. Today.
– 1997. – N 18. – P. 61-64.531. Stromberg E. Decreased epithelial cytokine responses in the duodenal mucosa ofHelicobacter pylori infected duodenal ulcer patients / E. Stromberg, A. Edebo, A.M.Svennerholm, C. Lindholm // Clin. Diagn. Lab. Immunol. – 2003.
– Vol 1, N 10. –P. 116-24.532. Stuller K.A. CD25+ T cells induce Helicobacter pylori-specific CD25-T cell anergybut are not required to maintain persistent hyporesponsiveness / K.A. Stuller, H.408Dingh, R.W. Redline, S.J. Czinn, T.G. Blanchard // Eur. J. Immunol. – 2008. – N38. – P. 3426-35.533. Sukhove G.K. Evidence for increased collagenolysis by interstitial collagenases-1and -3 in vulnerable human atheromatous plaques / G.K. Sukhove, U. Schonbeck, E.Rabkin et all // Circulation. – 1999.
– N 99. – P. 2503-9.534. Sun J. Mast cells promote atherosclerosis by releasing proinflammatory cytokines /J. Sun, G.K. Sukhova, P.J. Wolters et all // Nat. Med. – 2007. – N 13. – P. 719-24.535. Suzuki T. Effect of Helicobacter pylori eradication in patients with chronicidiopathic thrombocytopenic purpura – A randomised controlled trial / T. Suzuki, M.Matsushima, A. Masui et all // Am. Journal of Gastroenterology. – 2005. – Vol. 100,N 6.
– P. 1265-70.536. Swain S.L. Helper T-cell subsets: phenotype, function and the role of lymphokinesin regulating their development / S.L. Swain, L.М. Bradley, М. Croft et all //Immunol. Rev. – 1991. – N 123. – P. 115-144.537. Swirski F.K. Ly-6Chi monocytes dominate hypercholesterolemia-associatedmonocytosis and give rise to macrophages in atheromata / F.K.
Swirski, P. Libby, E.Aikawa et all // J. Clin. Invest. – 2007. – N 117. – P. 195-205.538. Szabo S.J. Molecular mechanism regulating Th1 response / S.J. Szabo, B.M. Sullivan, S.L. Peng, L.H. Glimcher // Annu. Rev. Immunol. – 2003. – N 21. – P. 713-58.539. Szczepański M. The influence of erythropoietin (Epo) on intercellular adhesionmolecule-1 (ICAM-1, CD54) and platelet-endothelial cell adhesion molecule-1(PECAM-1, CD31) expression on human umbilical vein endothelial cells (HUVEC)induced by tumor necrosis factor-alpha (TNF-alpha) / M.
Szczepański, M.Kamianowska, K. Wnuczko et all // Wiad Lek. – 2007. – Vol. 60, N 1-2. – P. 22-27.540. Szlachcic A. The link between Helicobacter pylori infection and rosacea / A.Szlachcic // Journal Eur. Acad. Dermatol. Venereol. – 2002. – Vol. 15, N 5. – P.551-54.409541. Tacke F. Monocyte subsets differentially employ CCR2, CCR5, and CX3CR1 toaccumulate within atherosclerotic plaques / F.
Tacke, D. Alvarez, T.J. Kaplan et all// J. Clin. Invest. – 2007. – N 117. – P. 185-94.542. Taguchi Т. Analysis of Th1 and Th2 cells in murine gut‐associated tissues Frequencies of CD4+ and CD8+ T cells that secrete INFγ and IL‐5 / Т. Taguchi, J.R.McGhee, R.L. Coffman et all // J. Immunol. – 1990. – N 145. – P. 68-77.543. Takeshima E. Helicobacter pylori induced interleukin-12 p40 expression / E.Takeshima, K.
Tomimori, H. Teruya et all // Infect. Immun. – 2009. – N 77. – P.1337-48.544. Takeuchi J. Discrimination of bacterial lipopolyproteins by Toll-like receptor 6 / J.Takeuchi, T. Kawai, P.F. Muhlradi et all // Int. Immunol. – 2001. – N 13. – P. 93340.545. Tamura A. Relation of Helicobacter pylori infection to plasma vitamin B12, folicacid, and homocysteine levels in patients who underwent diagnostic coronary arteriography / A. Tamura, T.
Fujioka, M. Nasu // Am. J. Gastroenterol. – 2002. – N 97. –P. 861-66.546. Tang P.C. Transmural inflammation by interferon-gammaproducing T cells correlates with outward vascular remodeling and intimal expansion of ascending thoracicaortic aneurysms / P.C. Tang, A.O. Yakimov, M.A. Teesdale et all // Faseb. J. –2005. – N 19. – P. 1528-30.547. Taylor B.V. Homocysteine, vitamins, and coronary artery disease. Comprehensivereview of the literature / B.V. Taylor, G.Y. Oudit, M. Evans // Can. Fam. Physician.– 2000.
– N 46. – P. 2236-45.548. Tedgui А. Cytokines in atherosclerosis: pathogenic and regulatory pathways / А.Tedgui, Z. Mallat // Physiol. Rev. – 2006. – Vol. 86, N 2. – P. 515-81.549. Teerlink J.R. Endothelins: pathophysiology and treatment implication in chronicheart failure / J.R. Teerlink // Current Heart Failure Reports. – 2005.
– N 2. – P. 19197.410550. Terano А. Chemokine expression in Helicobacter pylori-infected gastric mucosa /А. Terano, Т. Shimada // Gastroenterol. – 1998. – N 33. – P. 613-17.551. Tiirola T. Chlamydial lipopolysaccharide is present in serum during acute coronarysyndrome and correlates with CRP levels / T. Tiirola, J. Sinisalo, M.S. Nieminen etall // Atherosclerosis. – 2007.
– Vol. 194, N 2. – P. 403-407.552. Tobin N.P. Helicobacter pylori-induced inhibition of vascular endothelial cellfunctions: a role for VacA-dependent nitric oxide reduction / N.P. Tobin, G.T.Henehan, R.P. Murphy et all // Am. J. Physiol. Heart Circ. Physiol. – 2008.
– Vol.295, N 4. – P. H1403-13.553. Tousoulis D. Inflammatory and thrombotic mechanisms in coronary atherosclerosis / D. Tousoulis, G. Davies, С. Stefanadis et all // Heart. – 2003. – N 89. – P. 99397.554. Tupin E. CD1d-dependent activation of NKT cells aggravates atherosclerosis / E.Tupin, A. Nicoletti, R. Elhage et all // J. Experi. Med. – 2004. – N 199. – P. 417-22.555.
Tzianabos А.О. Bacterial pathogens induce abscess formation by CD4 + Т-cell activation на the CD28-B7-2 costimulatory pathway / А.О. Tzianabos, А. Chandraker,W. Kalka-Моll et all // Infect. and Immun. – 2000. – N 68. – P. 6650-55.556. Uematsu S. Detection of pathogenic intestinal bacteria by Toll-like receptor 5 onintestinal CD11c+ lamina propria cells / S. Uematsu, M.H. Jang, N.