Диссертация (Влияние плазменных факторов микроциркуляции на состояние суммарного поверхностного заряда мембраны эритроцитов у больных метаболическим синдромом), страница 16

/ Broderick T.L. et al. // Drugs RD. —2006. — Vol. 7. № 2. — P. 119-125.84.Effect of erythrocyte aggregation on velocity profiles in venules / J.J. Bishopet al. // Am J Physiol Heart Circ Physiol. — 2001. —Vol.280 —P.222–236.85.Electrochemical sensors for the detection of lead and other toxic heavymetals: the next generation of personal exposure biomonitors / Yantasee W.

[et al.]// Environ. Health Perspect. — 2007. — Vol. 115, № 12. — P. 1683-1690.86.Erythrocyte aggregation and metabolic syndrome / Gyawali P. et al. // ClinHemorheol Microcirc. — 2014—Vol. 57 (1). — P.73-83.87.Estimation of cell membrane properties and erythrocyte red-ox balance inpatients with metabolic syndrome /Kowalczyk E et al.// Mol Biol Rep.

— 2012Dec. — vol.39. — P. 11113–11118.88.Exposure to cobalt in the production of cobalt and cobalt compounds and itseffect on the heart / Linna A. et al. // Occup. Environ. Med. — 2004. — Vol. 61.№ 11. — P. 877-885.89.Fernandez-Real JM. Cross-talk between iron metabolism and diabetes /Fernandez-Real JM, Lopez-Bermejo A, Ricart W. // Diabetes — 2002. — Vol.51.№8.— P.2348–2354.10390.Fibrinogen in Type 2 Diabetic Patients with Metabolic Syndrome and itsRelation with Ischemic Heart Disease (IHD) and Retinopathy / Mahendra JV [etal.] // J Clin Diagn Res.

— 2015. — Vol. 9(1). — P.18-21.91.Fleming D.J. Iron status of the free-living, elderly Framingham Heart Studycohort: an iron-replete population with a high prevalence of elevated iron stores /D.J. Fleming, P.F. Jacques, K.L. Tucker // Am.

J. Clin. Nutr. – 2001. – Vol. 73. –P. 638–646.92.Gestational diabetes mellitus increases the risk of cardiovascular disease inwomen with a family history of type 2 diabetes / Carr D.B. et al.// Diabetes Care.—2006 Sep. — Vol.29(9). — P.2078-2083.93.Glasdam S.M. The importance of magnesium in the human body: Asystematic literature review /Glasdam S.M., Glasdam S., Peters, G.H. // Advancesin Clinical Chemistry, — 2016; — Vol.73.

— P.169–193.94.Grundy SM. Pre-diabetes, metabolic syndrome, and cardiovascular risk. JAm Coll Cardiol. — 2012. — Vol.59. — P.635-643.95.Guerrero-Romero F. Severe hypomagnesemia and low-grade inflammationin metabolic syndrome / Guerrero-Romero F, Bermudez-Peña C, Rodríguez-MoránM. // Magnes Res. — 2011. — Vol.24(2). — P.45-53.96.Gyawali P. Erythrocyte morphology in metabolic syndrome / Gyawali P.,Richards R.S., Nwose E.U.// Expert Rev Hematol. — 2012. — Vol.5. — P.523–531.97.Hemorheological parameters better classify metabolic syndrome than novelcardiovascular risk factors and peripheral vascular disease marker / Gyawali P.

etal. // Clinical Hemorheology and Microcirculation— 2016 — vol. 64. — P. 1-5.98.High levels of iron status and oxidative stress in patients with metabolicsyndrome / Leiva E. et al. // Biol.Trace Elem.Res., — 2013 Jan., — Vol.151 (1).— P.1-8.99.Hoenig M.R. Hypoxia inducible factor-1 alpha, endothelial progenitor cells,monocytes, cardiovascular risk, wound healing, cobalt and hydralazine: a unifying104hypothesis / Hoenig M.R., Bianchi C., Sellke F.W. // Curr. Drug.

Targets. — 2008.— Vol. 9. —P.422-435.100. Hypomagnesemia and its relation with chronic low-grade inflammation inobesity / Oliveira AR et al. // Rev. Assoc. Med. Bras. — 2017. — Vol.63. —P.156-163.101. Impact of the metabolic syndrome on mortality from coronary heart disease,cardiovascular disease, and all cause in United States adults / Malik S [et al.] //Circulation— 2004.— Vol.110. — P.1245–1250.102. Increased ability of erythrocytes to aggregate in spontaneously hypertensiverats / D.

Lominadze D.A. et al. //Clin Exp Hypertens — 2002, P.397–406.103. Increased oxidative stress and decreased membrane fluidity in erythrocytesof CAD patients / Pytel E et al. // Biochem Cell Biol. — 2013 Oct— Vol.91(5). —P.315-318.104. Isomaa B. A major health hazard: the metabolic syndrome. Life Sci. — 2003— Vol.73(19). — P.2395–2411.105. Jewell S. A. The effect of oxidative stress on the membrane dipole potentialof human red blood cells / Jewell S. A., Petrov P. G., Winlove C.

P. // Biochimicaet Biophysica Acta—Biomembranes — 2013. —Vol. 1828. —P.1250–1258.106. Jomova K. Advances in metal-induced oxidative stress and human disease /Jomova K., Valko M.// Toxicology. — 2011. — Vol. 283. —P. 65-87.107. K.R. Kensey. The mechanistic relationships between hemorheologicalcharacteristics and cardiovascular disease // Curr Med Res Opin. —2003. —Vol.19. — P.587–596.108. Liu B. Deficient zinc levels and myocardial infarction: association betweendeficient zinc levels and myocardial infarction: a meta-analysis / Liu B., Cai Z.Q.,Zhou Y.M. // Biol Trace Elem Res. — 2015.— Vol.165, — P.41–50.109. Lominadze, D. Involvement of fibrinogen specific binding in erythrocyteaggregation / D. Lominadze, W.

Dean // FEBS Lett. — 2002. —Vol. 517. — Р.41–44.105r=0,41;p<0,05765Al432101,902,402,683,003,263,50 3,70 3,90 4,10 4,30Фибриноген(г/л)Рисунок 32. Зависимость уровня Al от концентрации фибриногена.r=0,46;p<0,050,320,300,280,260,240,220,20Pb0,180,160,140,120,100,080,060,040,020,001,902,682,403,263,003,703,504,103,904,30Фибриноген(г/л)Рисунок 33. Зависимость уровня Pb от концентрации фибриногена.3.3. Взаимосвязь МЭ и металлов крови с СПЗМЭрОднимизнаименееизученныхплазменныхфактороввнутрисосудистой микроциркуляции является содержание МЭ и металловкрови при подавляющем большинстве ССЗ, в том числе и при МС.

При этомнам не встретилось литературных сведений по исследованию СПЗМЭр при79119. O' Neill S. Metabolic syndrome: a closer look at the growing epidemic andits associated pathologies / O' Neill S, O'Driscoll L. // Obes Rev. — 2015. — Vol.16. — P.1-12.120.

Obesity is a major determinant of the association of C_reactive proteinlevelsand the metabolic syndrome in type 2 diabetes / Kаhn S.E. et al. // Diabetes—2006 — Vol.55. №8. — P.2357–2364.121. Oss van C. J. Zeta potentials, van der Waals forces and hemagglutination//Vox Sang. — 1983. — № 44 (3). — Р. 183–190.122. Oxidative damages in erythrocytes of patients with metabolic syndrome /Ziobro A. et al.

// Mol. Cell Biochem. — 2013. — Vol.378. — P.267-273.123. Oxidative damages in erythrocytes of patients with metabolic syndrome / A.Ziobro [et al.] // Molecular and cellular biochemistry, — 2013. —Vol.378. — P.267-273.124. Periasamy M., Janssen P.M. Molecular basis of diastolic dysfunction //Heart Fail. Clin. — 2008. — Vol. 4.

№ 1. — P. 13-21.125. Plasma mineral content in type-2 diabetic patients and their association withthe metabolic syndrome / Aguilar MV [et al.] // Ann Nutr Metab. —2007. —Vol.51. — P.402-406.126. Prasad A.S. Discovery of human zinc deficiency: its impact on human healthand disease // Advances in Nutrition. — 2013. — Vol.4. — P.176–190.127. Prozialeck W.C. The vascular endothelium as a target of cadmium toxicity /Prozialeck W.C., Edwards J.R., Woods J.M. // Life Sci. — 2006. — Vol. 79. — P.1493-1506.128. Rani V. Oxidative stress and metabolic disorders: Pathogenesis andtherapeutic strategies / Rani V. Deep G, Singh RK, Palle K // Life Sci. — 2016.

—Vol.148. — P.183-193.129. Reaven G. Metabolic syndrome: pathophysiology and implications formanagement of cardiovascular disease // Circulation. — 2002. — Vol.106. — P.286–288.107130. Reaven GM. Banting lecture 1988. Role of insulin resistance in humandisease. Diabetes. — 1988. — Vol.37.

— P.1595-1607.131. Reaven GM. Insulin resistance: the link between obesity and cardiovasculardisease. Med Clin North Am. — 2011. — Vol.95. — P.875-892.132. Relationship between insulin resistance and some coagulation andfibrinolytic parameters it patients with metabolic syndrome / Ragab A. [et al.] //Lab.Hematol — 2008. — Vol. 14. — P. 1–6.133. Romeo G.R., Lee J. and Shoelson S.E.

Metabolic syndrome, insulinresistance, and roles of inflammation – mechanisms and therapeutic targets /Romeo G.R., Lee J. and Shoelson S.E. // Arterioscler Thromb Vasc Biol. — 2012.— Vol.32 — P. 1771–1776.134. Study of effects of copper deficiency on internal organ through a copperdeficiency model in rat / Liu X.Z. et al.

// — 2007. — Vol. 41, — P. 127-130.135. Study of Erythrocyte Indices, Erythrocyte Morphometric Indicators, andOxygen-Binding Properties of Hemoglobin Hematoporphyrin Patients withCardiovascular Diseases / Victor V.Revin [et al.] // Advances at Hemotology. —2017. — Vol. 2017. — P.1-9.136. Sullivan J.L. Stored Iron and Vascular Reactivity // Arteriosclerosis,Thrombosis, and Vascular Biology.

— 2005. — Vol. 25. — P. 1532.137. Sura P. Cadmium toxielty related to cysteine metabolism and glutationlevels in frog Rana ridibunda tissues / P.Sura, N.Ristic, P.Bronowicka //Comp.Biochem.Physiol.Toxicol.Pharmacol. — 2006. — Vol.142. №1. —P.128135.138. Systematic review of potential health risks posed by pharmaceutical,occupational and consumer exposures to metallic and nanoscale aluminum,aluminum oxides, aluminum hydroxide and its soluble salts / Willhite C.C.

[et al.]// Crit. Rev. Toxicol. — 2014. — Vol. 44. — P.1-80.139. The association of dyslipidemia with erythrocyte aggregation / Richards R.S.[at al.] // Clinical lipidology. — 2012. №2. — P.129-135.108140. The association of OSA with insulin resistance, inflammation and metabolicsyndrome / N. Peled [et al.] // Respir. Med. — 2007.

— Vol.101, — P.1696–1701.141. The Human Microcirculation: Regulation of Flow and Beyond / GuttermanDD. [et al.] // Circ. Res. — 2016. — Vol.118. — P.157-172.142. The metabolic syndrome and cardiovascular risk a systematic review andmeta-analysis / Mottillo S. [ et al.] // J.Am.Coll.Cardiol. — 2010. —Vol.56. —P.1113-1132.143. The metabolic syndrome and insulin resistance: relationship to haemostaticand inflammatory markers in older nondiabeticmen / Wannamethee S.G. [et al.] //Atherosclerosis.